Steroid metabolome analysis reveals prevalent glucocorticoid excess in primary aldosteronism

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http://www.wikidata.org/entity/Q33579003

description

2017 nî lūn-bûn

@nan

2017 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի ապրիլին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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name

Steroid metabolome analysis re ...... xcess in primary aldosteronism

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Steroid metabolome analysis re ...... xcess in primary aldosteronism

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Steroid metabolome analysis re ...... cess in primary aldosteronism.

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type

Item

label

Steroid metabolome analysis re ...... xcess in primary aldosteronism

@ast

Steroid metabolome analysis re ...... xcess in primary aldosteronism

@en

Steroid metabolome analysis re ...... cess in primary aldosteronism.

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prefLabel

Steroid metabolome analysis re ...... xcess in primary aldosteronism

@ast

Steroid metabolome analysis re ...... xcess in primary aldosteronism

@en

Steroid metabolome analysis re ...... cess in primary aldosteronism.

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P1476

Steroid metabolome analysis re ...... xcess in primary aldosteronism

@en

P2093

Anna Riester

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Anna S Dietz

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Annette Feuchtinger

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Axel K Walch

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Beverly A Hughes

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Brian G Keevil

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Cedric H L Shackleton

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Christian Adolf

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D Aled Rees

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Donna M O'Neil

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P2860

Subclinical hypercortisolism: a state, a syndrome, or a disease?

A Randomized Trial of Intensive versus Standard Blood-Pressure Control

The Management of Primary Aldosteronism: Case Detection, Diagnosis, and Treatment: An Endocrine Society Clinical Practice Guideline.

Management of adrenal incidentalomas: European Society of Endocrinology Clinical Practice Guideline in collaboration with the European Network for the Study of Adrenal Tumors.

Sympercents: symmetric percentage differences on the 100 log(e) scale simplify the presentation of log transformed data.

Gas chromatography/mass spectrometry (GC/MS) remains a pre-eminent discovery tool in clinical steroid investigations even in the era of fast liquid chromatography tandem mass spectrometry (LC/MS/MS).

Somatic mutations in ATP1A1 and ATP2B3 lead to aldosterone-producing adenomas and secondary hypertension.

Somatic and germline CACNA1D calcium channel mutations in aldosterone-producing adenomas and primary aldosteronism

Somatic mutations in ATP1A1 and CACNA1D underlie a common subtype of adrenal hypertension.

Cortisol as a marker for increased mortality in patients with incidental adrenocortical adenomas.

P31

scholarly article

P356

10.1172/JCI.INSIGHT.93136

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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P577

2017-04-20T00:00:00Z

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P698

28422753

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P932

5396526

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