Coordinate regulation of two opaque-phase-specific genes during white-opaque switching in Candida albicans.
about
EFG1 null mutants of Candida albicans switch but cannot express the complete phenotype of white-phase budding cellsRegulation of phenotypic transitions in the fungal pathogen Candida albicansThe role of phenotypic switching in the basic biology and pathogenesis of Candida albicansThe ins and outs of DNA fingerprinting the infectious fungiPhenotypic switch: The enigmatic white-gray-opaque transition system of Candida albicans.Lack of consistent short sequence repeat polymorphisms in genetically homologous colonizing and invasive Candida albicans strainsA MADS box protein consensus binding site is necessary and sufficient for activation of the opaque-phase-specific gene OP4 of Candida albicans.Elevated phenotypic switching and drug resistance of Candida albicans from human immunodeficiency virus-positive individuals prior to first thrush episodeThe histone deacetylase genes HDA1 and RPD3 play distinct roles in regulation of high-frequency phenotypic switching in Candida albicans.Misexpression of the opaque-phase-specific gene PEP1 (SAP1) in the white phase of Candida albicans confers increased virulence in a mouse model of cutaneous infection.Hemoglobin regulates expression of an activator of mating-type locus alpha genes in Candida albicans.Metabolic specialization associated with phenotypic switching in Candidaalbicans.In Candida albicans, white-opaque switchers are homozygous for mating typeHIV aspartyl protease inhibitors as promising compounds against Candida albicans André Luis Souza dos SantosRelationship between switching and mating in Candida albicansCandida albicans secreted aspartyl proteinases in virulence and pathogenesisMisexpression of the white-phase-specific gene WH11 in the opaque phase of Candida albicans affects switching and virulenceCandida albicans secreted aspartyl proteinases: isoenzyme pattern is determined by cell type, and levels are determined by environmental factors.The sea pansy Renilla reniformis luciferase serves as a sensitive bioluminescent reporter for differential gene expression in Candida albicans.In vivo and in vitro anaerobic mating in Candida albicans.Increased expression of Candida albicans secretory proteinase, a putative virulence factor, in isolates from human immunodeficiency virus-positive patientsFunctional analysis of the promoter of the phase-specific WH11 gene of Candida albicansShared themes of antigenic variation and virulence in bacterial, protozoal, and fungal infections.Candida albicans forms a specialized "sexual" as well as "pathogenic" biofilm.Bcr1 plays a central role in the regulation of opaque cell filamentation in Candida albicansAnalysis of phase-specific gene expression at the single-cell level in the white-opaque switching system of Candida albicans.Phenotypic switching in Candida glabrata involves phase-specific regulation of the metallothionein gene MT-II and the newly discovered hemolysin gene HLP.Large-scale identification of putative exported proteins in Candida albicans by genetic selection.Roles of TUP1 in switching, phase maintenance, and phase-specific gene expression in Candida albicans.The "universal" leucine codon CTG in the secreted aspartyl proteinase 1 (SAP1) gene of Candida albicans encodes a serine in vivo.The WH11 gene of Candida albicans is regulated in two distinct developmental programs through the same transcription activation sequences.The Candida albicans CDR3 gene codes for an opaque-phase ABC transporterA fourth secreted aspartyl proteinase gene (SAP4) and a CARE2 repetitive element are located upstream of the SAP1 gene in Candida albicans.Alpha-pheromone-induced "shmooing" and gene regulation require white-opaque switching during Candida albicans matingRelease of a potent polymorphonuclear leukocyte chemoattractant is regulated by white-opaque switching in Candida albicansTranscriptional loops meet chromatin: a dual-layer network controls white-opaque switching in Candida albicans.Unique aspects of gene expression during Candida albicans mating and possible G(1) dependency.Carbon source-induced reprogramming of the cell wall proteome and secretome modulates the adherence and drug resistance of the fungal pathogen Candida albicans.CO(2) regulates white-to-opaque switching in Candida albicans.Sac7 and Rho1 regulate the white-to-opaque switching in Candida albicans.
P2860
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P2860
Coordinate regulation of two opaque-phase-specific genes during white-opaque switching in Candida albicans.
description
1993 nî lūn-bûn
@nan
1993 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
Coordinate regulation of two o ...... switching in Candida albicans.
@ast
Coordinate regulation of two o ...... switching in Candida albicans.
@en
type
label
Coordinate regulation of two o ...... switching in Candida albicans.
@ast
Coordinate regulation of two o ...... switching in Candida albicans.
@en
prefLabel
Coordinate regulation of two o ...... switching in Candida albicans.
@ast
Coordinate regulation of two o ...... switching in Candida albicans.
@en
P2093
P2860
P1476
Coordinate regulation of two o ...... switching in Candida albicans.
@en
P2093
P2860
P304
P407
P577
1993-05-01T00:00:00Z