Increased tissue factor expression on circulating monocytes in chronic HIV infection: relationship to in vivo coagulation and immune activation - sprookjes - yvandenbroek - TriplyDB

Increased tissue factor expression on circulating monocytes in chronic HIV infection: relationship to in vivo coagulation and immune activation

Increased tissue factor expression on circulating monocytes in chronic HIV infection: relationship to in vivo coagulation and immune activation

http://www.wikidata.org/entity/Q33597485

about

Is the neutrophil a 'prima donna' in the procoagulant process during sepsis?Monocytes as regulators of inflammation and HIV-related comorbidities during cART HIV and aging: emerging research issues Markers of coagulation and inflammation often remain elevated in ART-treated HIV-infected patients Immune restoration after antiretroviral therapy: the pitfalls of hasty or incomplete repairs.The end of AIDS: HIV infection as a chronic disease Antibiotic and Antiinflammatory Therapy Transiently Reduces Inflammation and Hypercoagulation in Acutely SIV-Infected Pigtailed Macaques Immunologic Biomarkers, Morbidity, and Mortality in Treated HIV Infection Enhanced effector function of CD8(+) T cells from healthy controls and HIV-infected patients occurs through thrombin activation of protease-activated receptor 1.Dynamics of immune reconstitution and activation markers in HIV+ treatment-naïve patients treated with raltegravir, tenofovir disoproxil fumarate and emtricitabine Platelet count kinetics following interruption of antiretroviral treatment.Systemic effects of inflammation on health during chronic HIV infection Coagulation and morbidity in treated HIV infection.HIV-infected individuals with low CD4/CD8 ratio despite effective antiretroviral therapy exhibit altered T cell subsets, heightened CD8+ T cell activation, and increased risk of non-AIDS morbidity and mortality.Similarity in viral and host promoters couples viral reactivation with host cell migration Immune activation and HIV persistence: considerations for novel therapeutic interventions.Microbial translocation and cardiometabolic risk factors in HIV infection.Soluble CD14 is independently associated with coronary calcification and extent of subclinical vascular disease in treated HIV infection.Traditional risk factors and D-dimer predict incident cardiovascular disease events in chronic HIV infection Microbial translocation induces persistent macrophage activation unrelated to HIV-1 levels or T-cell activation following therapy.Nonprogressive and progressive primate immunodeficiency lentivirus infections.Residual immune dysregulation syndrome in treated HIV infection.Th17, gut, and HIV: therapeutic implications Can early therapy reduce inflammation?Biomarkers and HIV-associated cardiovascular disease.Chronic HIV disease and activation of the coagulation system Immune activation and HIV persistence: implications for curative approaches to HIV infection.Invariant natural killer T (iNKT) cells in HAART-treated, HIV-positive patients with bone and cardiovascular impairment.Changes in inflammatory and coagulation biomarkers: a randomized comparison of immediate versus deferred antiretroviral therapy in patients with HIV infection.T-cell phenotypes, apoptosis and inflammation in HIV+ patients on virologically effective cART with early atherosclerosis Biomarkers of immune dysfunction in HIV.Different plasma markers of inflammation are influenced by immune recovery and cART composition or intensification in treated HIV infected individuals HIV-associated chronic immune activation.Cardiovascular implications from untreated human immunodeficiency virus infection.The role of extended-release niacin on immune activation and neurocognition in HIV-infected patients treated with antiretroviral therapy - CTN PT006: study protocol for a randomized controlled trial.IL-1Β enriched monocytes mount massive IL-6 responses to common inflammatory triggers among chronically HIV-1 infected adults on stable anti-retroviral therapy at risk for cardiovascular disease The CD14 C-260T single nucleotide polymorphism (SNP) modulates monocyte/macrophage activation in treated HIV-infected individuals.Rosuvastatin reduces vascular inflammation and T-cell and monocyte activation in HIV-infected subjects on antiretroviral therapy.Factors associated with D-dimer levels in HIV-infected individuals.HIV-Associated Venous Thromboembolism

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P2860

Increased tissue factor expression on circulating monocytes in chronic HIV infection: relationship to in vivo coagulation and immune activation

http://www.wikidata.org/entity/Q33597485

description

2009 nî lūn-bûn

@nan

2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

@zh-tw

2009年论文

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name

Increased tissue factor expres ...... gulation and immune activation

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Increased tissue factor expres ...... gulation and immune activation

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type

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Increased tissue factor expres ...... gulation and immune activation

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Increased tissue factor expres ...... gulation and immune activation

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BLOOD-2009-03-210179

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Increased tissue factor expres ...... gulation and immune activation

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Angel A Luciano

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Daniel C Douek

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Elizabeth Mayne

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Jason M Brenchley

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Magdalena Kalinowska

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Michael M Lederman

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Robert Asaad

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Scott F Sieg

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Wei Jiang

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Wendy Stevens

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P2860

Microbial translocation is a cause of systemic immune activation in chronic HIV infection

Inflammatory and coagulation biomarkers and mortality in patients with HIV infection

High prevalence of peripheral arterial disease in HIV-infected persons.

Toll-like receptor ligands induce human T cell activation and death, a model for HIV pathogenesis.

Mechanisms of thrombus formation

Atherosclerosis and HIV in the highly active antiretroviral therapy era: towards an epidemic of cardiovascular disease?

Tissue factor pathway inhibitor reduces mortality from Escherichia coli septic shock.

Relevance of tissue factor in cardiovascular disease.

Modulatory effects of sCD14 and LBP on LPS-host cell interactions.

Role of tissue factor in hemostasis and thrombosis.

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161-167

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scholarly article

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10.1182/BLOOD-2009-03-210179

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2

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115

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Benigno Rodriguez

Nicholas Funderburg

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2009-10-14T00:00:00Z

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38011166

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19828697

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2808148

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