Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
about
Using Drosophila to discover mechanisms underlying type 2 diabetesCyclin G Functions as a Positive Regulator of Growth and Metabolism in DrosophilaLoss of the tumor suppressor Pten promotes proliferation of Drosophila melanogaster cells in vitro and gives rise to continuous cell linesAltered regulation of sleep and feeding contributes to starvation resistance in Drosophila melanogasterDiet and energy-sensing inputs affect TorC1-mediated axon misrouting but not TorC2-directed synapse growth in a Drosophila model of tuberous sclerosis.Unkempt is negatively regulated by mTOR and uncouples neuronal differentiation from growth control.TOR and ageing: a complex pathway for a complex process.Using answer set programming to integrate RNA expression with signalling pathway information to infer how mutations affect ageing.Genome-scale RNAi on living-cell microarrays identifies novel regulators of Drosophila melanogaster TORC1-S6K pathway signaling.A systems-level interrogation identifies regulators of Drosophila blood cell number and survival.Drosophila Longevity Assurance Conferred by Reduced Insulin Receptor Substrate Chico Partially Requires d4eBP.Drosophila Spidey/Kar Regulates Oenocyte Growth via PI3-Kinase Signaling.Proteomic and functional genomic landscape of receptor tyrosine kinase and ras to extracellular signal-regulated kinase signaling.Multifaceted roles of PTEN and TSC orchestrate growth and differentiation of Drosophila blood progenitors.Noncanonical control of C. elegans germline apoptosis by the insulin/IGF-1 and Ras/MAPK signaling pathwaysNutrient restriction enhances the proliferative potential of cells lacking the tumor suppressor PTEN in mitotic tissuesAn Integrative Analysis of the InR/PI3K/Akt Network Identifies the Dynamic Response to Insulin SignalingDrosophila RNAi screening in a postgenomic world.Evolution of the TOR pathway.The TSC1/2 complex controls Drosophila pigmentation through TORC1-dependent regulation of catecholamine biosynthesis.Nutrition-responsive glia control exit of neural stem cells from quiescence.Signaling pathways involved in 1-octen-3-ol-mediated neurotoxicity in Drosophila melanogaster: implication in Parkinson’s disease.Detrimental effects of proteasome inhibition activity in Drosophila melanogaster: implication of ER stress, autophagy, and apoptosis.Genetic dissection reveals that Akt is the critical kinase downstream of LRRK2 to phosphorylate and inhibit FOXO1, and promotes neuron survival.A Drosophila model of insulin resistance associated with the human TRIB3 Q/R polymorphism.Regulation of Carbohydrate Energy Metabolism in Drosophila melanogaster.Oocyte destruction is activated during viral infection.FoxO restricts growth and differentiation of cells with elevated TORC1 activity under nutrient restriction.
P2860
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P2860
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
@ast
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
@en
type
label
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
@ast
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
@en
prefLabel
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
@ast
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
@en
P2093
P2860
P1433
P1476
Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.
@en
P2093
Katja Brückner
Kimberly S Kerr
Lutz Kockel
Matthias Hebrok
Michael Melnick
P2860
P304
P356
10.1371/JOURNAL.PGEN.1000990
P577
2010-06-17T00:00:00Z