Microglial activation and amyloid deposition in mild cognitive impairment: a PET study - sprookjes - yvandenbroek - TriplyDB

Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

http://www.wikidata.org/entity/Q33634089

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18 F-FDG PET for the early diagnosis of Alzheimer’s disease dementia and other dementias in people with mild cognitive impairment (MCI)The accuracy of 18 FDG-PET in the early diagnosis of Alzheimer’s disease dementia and other dementias in people with MCI Targeting innate immunity for neurodegenerative disorders of the central nervous system Repurposing psychiatric medicines to target activated microglia in anxious mild cognitive impairment and early Parkinson's disease The methodology of TSPO imaging with positron emission tomography Alzheimer disease: epidemiology, diagnostic criteria, risk factors and biomarkers Imaging multiple sclerosis and other neurodegenerative diseases Innate immunity in Alzheimer's disease: a complex affair Neuroimaging biomarkers of neurodegenerative diseases and dementia Neuroinflammation in Alzheimer's disease Positron emission tomography imaging and clinical progression in relation to molecular pathology in the first Pittsburgh Compound B positron emission tomography patient with Alzheimer's disease Future Directions in Imaging Neurodegeneration.Translocator Protein-18 kDa (TSPO) Positron Emission Tomography (PET) Imaging and Its Clinical Impact in Neurodegenerative Diseases Inflammation, microglia, and Alzheimer's disease.Conversion of amyloid positive and negative MCI to AD over 3 years: an 11C-PIB PET study.NOX activity is increased in mild cognitive impairment Mapping brain beta-amyloid.Tracking neuroinflammation in Alzheimer's disease: the role of positron emission tomography imaging.Microglia activation and anti-inflammatory regulation in Alzheimer's disease Role of microglia adenosine A(2A) receptors in retinal and brain neurodegenerative diseases.Fluorodeoxyglucose positron emission tomography of mild cognitive impairment with clinical follow-up at 3 years Brain imaging in the study of Alzheimer's disease An evaluation of MSDC-0160, a prototype mTOT modulating insulin sensitizer, in patients with mild Alzheimer's disease.Proteolysis of calcineurin is increased in human hippocampus during mild cognitive impairment and is stimulated by oligomeric Abeta in primary cell culture.Amyloid-beta protein oligomer at low nanomolar concentrations activates microglia and induces microglial neurotoxicity.Peripheral inflammatory markers in amnestic mild cognitive impairment Microglial P2X₇ receptor expression is accompanied by neuronal damage in the cerebral cortex of the APPswe/PS1dE9 mouse model of Alzheimer's disease.Neuroinflammation and neuronal loss precede Aβ plaque deposition in the hAPP-J20 mouse model of Alzheimer's disease.The 21st century epidemic: infections as inductors of neuro-degeneration associated with Alzheimer's Disease.White matter abnormalities and structural hippocampal disconnections in amnestic mild cognitive impairment and Alzheimer's disease.Image-derived input function derived from a supervised clustering algorithm: methodology and validation in a clinical protocol using [11C](R)-rolipram Microglial phagocytosis induced by fibrillar β-amyloid is attenuated by oligomeric β-amyloid: implications for Alzheimer's disease.Reactive glia not only associates with plaques but also parallels tangles in Alzheimer's disease Biomarkers for microglial activation in Alzheimer's disease.Magnitude of [(11)C]PK11195 binding is related to severity of motor deficits in a rabbit model of cerebral palsy induced by intrauterine endotoxin exposure.The classification of microglial activation phenotypes on neurodegeneration and regeneration in Alzheimer's disease brain.Amyloid imaging as a biomarker for cerebral β-amyloidosis and risk prediction for Alzheimer dementia.Neuroinflammation in Alzheimer's disease wanes with age In vivo positron emission tomographic imaging of glial responses to amyloid-beta and tau pathologies in mouse models of Alzheimer's disease and related disorders.Molecular imaging in Alzheimer's disease: new perspectives on biomarkers for early diagnosis and drug development.

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P2860

Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

http://www.wikidata.org/entity/Q33634089

description

2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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name

Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

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Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

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Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

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Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

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Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

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Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

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Microglial activation and amyloid deposition in mild cognitive impairment: a PET study

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Clinical diagnosis of Alzheimer's disease: report of the NINCDS-ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease

Neuropathologic features of amnestic mild cognitive impairment

Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound-B

Estimation of the number of "true" null hypotheses in multivariate analysis of neuroimaging data.

Current concepts in mild cognitive impairment.

Inflammation, autotoxicity and Alzheimer disease.

Demonstration of microglial cells in and around senile (neuritic) plaques in the Alzheimer brain. An immunohistochemical study using a novel monoclonal antibody.

Rapid appearance and local toxicity of amyloid-beta plaques in a mouse model of Alzheimer's disease

Contribution of glial cells to the development of amyloid plaques in Alzheimer's disease.

Diffuse senile plaques occur commonly in the cerebellum in Alzheimer's disease.

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