Antibiotics induce redox-related physiological alterations as part of their lethality. - sprookjes - yvandenbroek - TriplyDB

Antibiotics induce redox-related physiological alterations as part of their lethality.

Antibiotics induce redox-related physiological alterations as part of their lethality.

http://www.wikidata.org/entity/Q33665248

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The effects of high perioperative inspiratory oxygen fraction for adult surgical patients Moving forward with reactive oxygen species involvement in antimicrobial lethality A role for the bacterial GATC methylome in antibiotic stress survival Microscale insights into pneumococcal antibiotic mutant selection windows Antifungal Activity and Action Mechanism of Histatin 5-Halocidin Hybrid Peptides against Candida ssp Antimicrobial-induced DNA damage and genomic instability in microbial pathogens Reactive Oxygen Species Play an Important Role in the Bactericidal Activity of Quinolone Antibiotics Dysregulated mitochondrial and chloroplast bioenergetics from a translational medical perspective (Review)Review of Antimicrobial Resistance in the Environment and Its Relevance to Environmental Regulators Antibiotic treatment enhances the genome-wide mutation rate of target cells Bactericidal antibiotics promote reactive oxygen species formation and inflammation in human sinonasal epithelial cells New insights into TB physiology suggest untapped therapeutic opportunities Bioactivity Studies of β-Lactam Derived Polycyclic Fused Pyrroli-Dine/Pyrrolizidine Derivatives in Dentistry: In Vitro, In Vivo and In Silico Studies The Role of Reactive Oxygen Species in Antibiotic-Induced Cell Death in Burkholderia cepacia Complex Bacteria Antibiotics May Trigger Mitochondrial Dysfunction Inducing Psychiatric Disorders Chemogenomics and orthology-based design of antibiotic combination therapies Endless Resistance. Endless Antibiotics?Reactive oxygen species do not contribute to ObgE*-mediated programmed cell death Gentamicin differentially alters cellular metabolism of cochlear hair cells as revealed by NAD(P)H fluorescence lifetime imaging.Single-cell, real-time detection of oxidative stress induced in Escherichia coli by the antimicrobial peptide CM15.Physiological levels of nitrate support anoxic growth by denitrification of Pseudomonas aeruginosa at growth rates reported in cystic fibrosis lungs and sputum.PheNetic: network-based interpretation of molecular profiling data Spatial Dependence of DNA Damage in Bacteria due to Low-Temperature Plasma Application as Assessed at the Single Cell Level.A scalable metabolite supplementation strategy against antibiotic resistant pathogen Chromobacterium violaceum induced by NAD+/NADH+ imbalance.Predicting disease onset in clinically healthy people DNA Backbone Sulfur-Modification Expands Microbial Growth Range under Multiple Stresses by its anti-oxidation function Metabolic fingerprinting of bacteria by fluorescence lifetime imaging microscopy.The impact of horizontal gene transfer on the adaptive ability of the human oral microbiome.Sigma S-dependent antioxidant defense protects stationary-phase Escherichia coli against the bactericidal antibiotic gentamicin.DNA mismatch repair and oxidative DNA damage: implications for cancer biology and treatment.Induction of DNA damage and erroneous repair can explain genomic instability caused by endosulfan.Reactive oxygen species and the bacterial response to lethal stress Oxidative stress enhances cephalosporin resistance of Enterococcus faecalis through activation of a two-component signaling system.Ribosomal elongation factor 4 promotes cell death associated with lethal stress Generation of reactive oxygen species by lethal attacks from competing microbes.Diagnosing oxidative stress in bacteria: not as easy as you might think The induction of two biosynthetic enzymes helps Escherichia coli sustain heme synthesis and activate catalase during hydrogen peroxide stress.Deficiency of the novel exopolyphosphatase Rv1026/PPX2 leads to metabolic downshift and altered cell wall permeability in Mycobacterium tuberculosis Biofilm Formation As a Response to Ecological Competition Exposure to sub-inhibitory concentrations of cefotaxime enhances the systemic colonization of Salmonella Typhimurium in BALB/c mice.

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Antibiotics induce redox-related physiological alterations as part of their lethality.

http://www.wikidata.org/entity/Q33665248

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年學術文章

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Antibiotics induce redox-related physiological alterations as part of their lethality.

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Antibiotics induce redox-related physiological alterations as part of their lethality.

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Antibiotics induce redox-related physiological alterations as part of their lethality.

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Antibiotics induce redox-related physiological alterations as part of their lethality.

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Antibiotics induce redox-related physiological alterations as part of their lethality.

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Antibiotics induce redox-related physiological alterations as part of their lethality.

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PNAS.1401876111

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Proceedings of the National Academy of Sciences of the United States of America

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Antibiotics induce redox-related physiological alterations as part of their lethality

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Ahmad S Khalil

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Alice Y Ting

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Dana Braff

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Eric G Schwarz

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I Cody MacDonald

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James J Collins

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Jeffrey D Martell

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Jonathan D Ye

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Kyle R Allison

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Maarten Vercruysse

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P2860

Endogenous nitric oxide protects bacteria against a wide spectrum of antibiotics

Sublethal antibiotic treatment leads to multidrug resistance via radical-induced mutagenesis

Metabolite-enabled eradication of bacterial persisters by aminoglycosides

Statistical significance for genomewide studies

A comparison of normalization methods for high density oligonucleotide array data based on variance and bias

A common mechanism of cellular death induced by bactericidal antibiotics

Oxidation of the guanine nucleotide pool underlies cell death by bactericidal antibiotics

Antimicrobial reactive oxygen and nitrogen species: concepts and controversies

Analysis of ahpC gene mutations in isoniazid-resistant clinical isolates of Mycobacterium tuberculosis

Genetic architecture of intrinsic antibiotic susceptibility

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2014-05-06T00:00:00Z

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