Mitochondrial dysfunction induced by nuclear poly(ADP-ribose) polymerase-1: a treatable cause of cell death in stroke.
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Epigenetic Regulation of Oxidative Stress in Ischemic StrokeProtection against Experimental Stroke by Ganglioside GM1 Is Associated with the Inhibition of AutophagyLactate shuttling and lactate use as fuel after traumatic brain injury: metabolic considerationsDemyelination as a rational therapeutic target for ischemic or traumatic brain injury.PARP1 Inhibitors: antitumor drug design.Paradigms and mechanisms of inhalational anesthetics mediated neuroprotection against cerebral ischemic stroke.NaHS Protects against the Impairments Induced by Oxygen-Glucose Deprivation in Different Ages of Primary Hippocampal Neurons.The NAD+-Dependent Family of Sirtuins in Cerebral Ischemia and Preconditioning.Opportunities for the repurposing of PARP inhibitors for the therapy of non-oncological diseases.Anti-inflammatory signaling: the point of convergence for medical gases in neuroprotection against ischemic stroke.Minocycline targets multiple secondary injury mechanisms in traumatic spinal cord injury.Bioenergetic Impairment in Animal and Cellular Models of Alzheimer's Disease: PARP-1 Inhibition Rescues Metabolic Dysfunctions.Assessment at the single-cell level identifies neuronal glutathione depletion as both a cause and effect of ischemia-reperfusion oxidative stress.CD38 Knockout Mice Show Significant Protection Against Ischemic Brain Damage Despite High Level Poly-ADP-Ribosylation.TSPO regulation in reactive gliotic diseases.Mfn2-Mediated Preservation of Mitochondrial Function Contributes to the Protective Effects of BHAPI in Response to Ischemia.YiQiFuMai Powder Injection Protects against Ischemic Stroke via Inhibiting Neuronal Apoptosis and PKCδ/Drp1-Mediated Excessive Mitochondrial Fission.Neural Stem Cell-Conditioned Medium Ameliorated Cerebral Ischemia-Reperfusion Injury in Rats.Mild therapeutic hypothermia protects against cerebral ischemia/reperfusion injury by inhibiting miR-15b expression in ratsSIRT3 Regulation Under Cellular Stress: Making Sense of the Ups and Downs
P2860
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P2860
Mitochondrial dysfunction induced by nuclear poly(ADP-ribose) polymerase-1: a treatable cause of cell death in stroke.
description
2013 nî lūn-bûn
@nan
2013 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Mitochondrial dysfunction indu ...... cause of cell death in stroke.
@ast
Mitochondrial dysfunction indu ...... cause of cell death in stroke.
@en
type
label
Mitochondrial dysfunction indu ...... cause of cell death in stroke.
@ast
Mitochondrial dysfunction indu ...... cause of cell death in stroke.
@en
prefLabel
Mitochondrial dysfunction indu ...... cause of cell death in stroke.
@ast
Mitochondrial dysfunction indu ...... cause of cell death in stroke.
@en
P2093
P2860
P1476
Mitochondrial dysfunction indu ...... cause of cell death in stroke.
@en
P2093
P2860
P2888
P304
P356
10.1007/S12975-013-0283-0
P577
2013-09-07T00:00:00Z
P6179
1014612695