Blood pressure, blood flow, and oxygenation in the clipped kidney of chronic 2-kidney, 1-clip rats: effects of tempol and Angiotensin blockade. - sprookjes - yvandenbroek - TriplyDB

Blood pressure, blood flow, and oxygenation in the clipped kidney of chronic 2-kidney, 1-clip rats: effects of tempol and Angiotensin blockade.

Blood pressure, blood flow, and oxygenation in the clipped kidney of chronic 2-kidney, 1-clip rats: effects of tempol and Angiotensin blockade.

http://www.wikidata.org/entity/Q33677156

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Asymmetric dimethylarginine and reactive oxygen species: unwelcome twin visitors to the cardiovascular and kidney disease tables Renal oxygenation and haemodynamics in acute kidney injury and chronic kidney disease Gained in translation: protective paradigms for the poststenotic kidney Darkness at the end of the tunnel: poststenotic kidney injury Antioxidant-based therapies for angiotensin II-associated cardiovascular diseases Silencing of hypoxia-inducible factor-1α gene attenuates chronic ischemic renal injury in two-kidney, one-clip rats.Syncytial communication in descending vasa recta includes myoendothelial coupling Neuronal uptake of nanoformulated superoxide dismutase and attenuation of angiotensin II-dependent hypertension after central administration.Role of the rostral ventrolateral medulla in the arterial hypertension in chronic renal failure Is the serum level of salusin-β associated with hypertension and atherosclerosis in the pediatric population?Persistent kidney dysfunction in swine renal artery stenosis correlates with outer cortical microvascular remodeling.Atherosclerotic renal artery stenosis: current status Renal oxidative stress, oxygenation, and hypertension Impacts of nitric oxide and superoxide on renal medullary oxygen transport and urine concentration.Kidney hypoxia, attributable to increased oxygen consumption, induces nephropathy independently of hyperglycemia and oxidative stress.Oxidative stress in hypertension: role of the kidney.Renal disease pathophysiology and treatment: contributions from the rat.Determinants of kidney oxygen consumption and their relationship to tissue oxygen tension in diabetes and hypertension.The role of oxidative stress in renovascular hypertension.Renal tubulointerstitial hypoxia: cause and consequence of kidney dysfunction.Angeli's Salt, a nitroxyl anion donor, reverses endothelin-1 mediated vascular dysfunction in murine aorta.Vascular Endothelial Growth Factor Therapy for the Kidney: Are We There Yet?Losartan reduces oxidative stress within the rostral ventrolateral medulla of rats with renovascular hypertension.

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P2860

Blood pressure, blood flow, and oxygenation in the clipped kidney of chronic 2-kidney, 1-clip rats: effects of tempol and Angiotensin blockade.

http://www.wikidata.org/entity/Q33677156

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Blood pressure, blood flow, an ...... mpol and Angiotensin blockade.

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HYPERTENSIONAHA.109.135426

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Christopher S Wilcox

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Fredrik Palm

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Maristela Onozato

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William J Welch

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P2860

Evaluation of methods for estimating renal medullary blood flow.

Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension.

Reduced nitric oxide concentration in the renal cortex of streptozotocin-induced diabetic rats: effects on renal oxygenation and microcirculation.

Nitric oxide-angiotensin II interactions in angiotensin II-dependent hypertension.

Is there a common mechanism for the progression of different types of renal diseases other than proteinuria? Towards the unifying theme of chronic hypoxia.

Enhanced intrarenal angiotensinogen contributes to early renal injury in spontaneously hypertensive rats.

STUDIES ON EXPERIMENTAL HYPERTENSION : I. THE PRODUCTION OF PERSISTENT ELEVATION OF SYSTOLIC BLOOD PRESSURE BY MEANS OF RENAL ISCHEMIA.

Oxidative stress and nitric oxide deficiency in the kidney: a critical link to hypertension?

NADPH oxidases in the kidney.

Angiotensin-converting enzyme inhibition in renal disease; contrasting effects on renal function in renal artery stenosis and progressive renal injury.

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