Inflammatory skin disease in transgenic mice that express high levels of interleukin 1 alpha in basal epidermis - sprookjes - yvandenbroek - TriplyDB

Inflammatory skin disease in transgenic mice that express high levels of interleukin 1 alpha in basal epidermis

Inflammatory skin disease in transgenic mice that express high levels of interleukin 1 alpha in basal epidermis

http://www.wikidata.org/entity/Q33730207

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Transgenic expression of the human amphiregulin gene induces a psoriasis-like phenotype Keratinocyte expression of the type 2 interleukin 1 receptor mediates local and specific inhibition of interleukin 1-mediated inflammation Opposing activities of two novel members of the IL-1 ligand family regulate skin inflammation A nuclear target for interleukin-1alpha: interaction with the growth suppressor necdin modulates proliferation and collagen expression Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk Development of transgenic cloned pig models of skin inflammation by DNA transposon-directed ectopic expression of human β1 and α2 integrin Dermatitis due to epiregulin deficiency and a critical role of epiregulin in immune-related responses of keratinocyte and macrophage.Cytokines in psoriasis.Role of FGFR2-signaling in the pathogenesis of acne.Androgenetic alopecia and microinflammation.Malnutrition-associated rash of cystic fibrosis.Skin immune sentinels in health and disease.Severe combined immunodeficiency mouse-human skin chimeras: a unique animal model for the study of psoriasis and cutaneous inflammation.Psoriasis-like skin disease and arthritis caused by inducible epidermal deletion of Jun proteins.Mutant laboratory mice with abnormalities in hair follicle morphogenesis, cycling, and/or structure: annotated tables.Sleep loss and cytokines levels in an experimental model of psoriasis.Lymphocyte deficiency limits Epstein-Barr virus latent membrane protein 1 induced chronic inflammation and carcinogenic pathology in vivo Depletion of antigen-presenting cells by clodronate liposomes reverses the psoriatic skin phenotype in KC-Tie2 mice.Genetic background of psoriasis The antipsoriatic agent monomethylfumarate has antiproliferative, prodifferentiative, and anti-inflammatory effects on keratinocytes.Animal models of psoriasis and pustular psoriasis.Inhibition of keratinocyte differentiation by the synergistic effect of IL-17A, IL-22, IL-1α, TNFα and oncostatin M.The psoriatic transcriptome closely resembles that induced by interleukin-1 in cultured keratinocytes: dominance of innate immune responses in psoriasis Genetically modified mice as a tool to study inflammatory skin diseases.Squamous metaplasia amplifies pathologic epithelial-mesenchymal interactions in COPD patients.Macrophage- and neutrophil-dominant arthritis in human IL-1 alpha transgenic mice A role for mitogen-activated protein kinase activation by integrins in the pathogenesis of psoriasis.Animal models of psoriasis.IkappaBalpha deficiency results in a sustained NF-kappaB response and severe widespread dermatitis in mice.Inducible deletion of the Blimp-1 gene in adult epidermis causes granulocyte-dominated chronic skin inflammation in mice The evolution of the psoriatic lesion.Autoinflammatory Skin Disorders: The Inflammasomme in Focus.Inflammatory doses of UV may not be necessary for skin carcinogenesis.Dynamic expression of epidermal caspase 8 simulates a wound healing response.Hair biology: an update.Autoinflammatory genes and susceptibility to psoriatic juvenile idiopathic arthritis.Inflammatory and hyperproliferative skin disease in mice that express elevated levels of the IL-1 receptor (type I) on epidermal keratinocytes. Evidence that IL-1-inducible secondary cytokines produced by keratinocytes in vivo can cause skin disease Epithelial decision makers: in search of the 'epimmunome'.Cytokines and the skin barrier.Junctions and inflammation in the skin.

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P2860

Inflammatory skin disease in transgenic mice that express high levels of interleukin 1 alpha in basal epidermis

http://www.wikidata.org/entity/Q33730207

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1995 nî lūn-bûn

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1995 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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Inflammatory skin disease in t ...... kin 1 alpha in basal epidermis

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Proceedings of the National Academy of Sciences of the United States of America

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Inflammatory skin disease in t ...... kin 1 alpha in basal epidermis

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H Mizutani

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J D Kieffer

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R W Groves

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T S Kupper

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P2860

Interleukin 1 is present in normal human epidermis

Tissue-specific and differentiation-specific expression of a human K14 keratin gene in transgenic mice

Effect of in vivo interleukin-1 on adhesion molecule expression in normal human skin.

Marked synergism between tumor necrosis factor-alpha and interferon-gamma in regulation of keratinocyte-derived adhesion molecules and chemotactic factors

Secretion of peptides and proteins lacking hydrophobic signal sequences: the role of adenosine triphosphate-driven membrane translocators.

Interleukin-1 receptor antagonist in normal and psoriatic epidermis.

Epidermal expression of intercellular adhesion molecule 1 is not a primary inducer of cutaneous inflammation in transgenic mice.

Immune and inflammatory processes in cutaneous tissues. Mechanisms and speculations.

Detection of interleukin-1 receptors in human epidermis. Induction of the type II receptor after organ culture and in psoriasis.

Human keratinocytes contain mRNA indistinguishable from monocyte interleukin 1 alpha and beta mRNA. Keratinocyte epidermal cell-derived thymocyte-activating factor is identical to interleukin 1.

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