Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse. - sprookjes - yvandenbroek - TriplyDB

Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse.

Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse.

http://www.wikidata.org/entity/Q33735687

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Epigenetic Mechanisms in Bone Biology and Osteoporosis: Can They Drive Therapeutic Choices?Vitamin D and microRNAs in bone MicroRNAs regulate bone development and regeneration Bone marrow stroma-derived miRNAs as regulators, biomarkers and therapeutic targets of bone metastasis miRNA-34c regulates Notch signaling during bone development A program of microRNAs controls osteogenic lineage progression by targeting transcription factor Runx2 Identification of genes for complex diseases using integrated analysis of multiple types of genomic data.Identification and characterization of microRNAs by high through-put sequencing in mesenchymal stem cells and bone tissue from mice of age-related osteoporosis.MicroRNA miR-23a cluster promotes osteocyte differentiation by regulating TGF-β signalling in osteoblasts.miRNAs in circulation: mirroring bone conditions?miR-214 targets ATF4 to inhibit bone formation.A network connecting Runx2, SATB2, and the miR-23a~27a~24-2 cluster regulates the osteoblast differentiation program.Epigenetic pathways regulating bone homeostasis: potential targeting for intervention of skeletal disorders.MicroRNAs involved in bone formation.Identification and characterization of microRNAs controlled by the osteoblast-specific transcription factor Osterix.Regulation of microRNA function in somatic stem cell proliferation and differentiation A microRNA expression signature of osteoclastogenesis Conditional disruption of miR17-92 cluster in collagen type I-producing osteoblasts results in reduced periosteal bone formation and bone anabolic response to exercise.The role of microRNAs in cellular senescence and age-related conditions of cartilage and bone.GATA4 is essential for bone mineralization via ERα and TGFβ/BMP pathways.MicroRNA delivery for regenerative medicine.MicroRNA-103a-3p controls proliferation and osteogenic differentiation of human adipose tissue-derived stromal cells.miR-34s inhibit osteoblast proliferation and differentiation in the mouse by targeting SATB2.MicroRNA 665 Regulates Dentinogenesis through MicroRNA-Mediated Silencing and Epigenetic Mechanisms Control of mesenchymal lineage progression by microRNAs targeting skeletal gene regulators Trps1 and Runx2.Impaired expression of DICER, DROSHA, SBDS and some microRNAs in mesenchymal stromal cells from myelodysplastic syndrome patients miR-218 directs a Wnt signaling circuit to promote differentiation of osteoblasts and osteomimicry of metastatic cancer cells MicroRNA control of bone formation and homeostasis.MicroRNA functions in osteogenesis and dysfunctions in osteoporosis Effects of miR-335-5p in modulating osteogenic differentiation by specifically downregulating Wnt antagonist DKK1 Composite cell sheet for periodontal regeneration: crosstalk between different types of MSCs in cell sheet facilitates complex periodontal-like tissue regeneration.MicroRNA biogenesis and regulation of bone remodeling.MicroRNAs regulate bone metabolism.Overview of skeletal development.MicroRNAs as regulators of bone homeostasis and bone metastasis.Synovium-Derived MicroRNAs Regulate Bone Pathways in Rheumatoid Arthritis.Non-coding RNAs: Epigenetic regulators of bone development and homeostasis.Bone as a Target Organ in Rheumatic Disease: Impact on Osteoclasts and Osteoblasts.miRNAs in Bone Development.Epigenetics and Bone Remodeling.

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P2860

Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse.

http://www.wikidata.org/entity/Q33735687

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Dicer inactivation in osteopro ...... bone mass in the adult mouse.

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Dicer inactivation in osteopro ...... bone mass in the adult mouse.

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type

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Dicer inactivation in osteopro ...... bone mass in the adult mouse.

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Dicer inactivation in osteopro ...... bone mass in the adult mouse.

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Dicer inactivation in osteopro ...... bone mass in the adult mouse.

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Dicer inactivation in osteopro ...... bone mass in the adult mouse.

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J.YDBIO.2010.01.008

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Developmental Biology

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Dicer inactivation in osteopro ...... bone mass in the adult mouse.

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Andre J van Wijnen

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Barbara E Kream

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Dana Frederick

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Gary S Stein

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Isha Parulkar

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Jane B Lian

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Janet L Stein

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Jennifer L Colby

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Rajini Mudhasani

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Sadiq Hussain

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P2860

The RNaseIII enzyme Dicer is required for morphogenesis but not patterning of the vertebrate limb.

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

A microRNA signature for a BMP2-induced osteoblast lineage commitment program

Targeted deletion of Dicer in the heart leads to dilated cardiomyopathy and heart failure

Cerebellar neurodegeneration in the absence of microRNAs

High bone mass in mice expressing a mutant LRP5 gene

Osteogenic differentiation of human adipose tissue-derived stem cells is modulated by the miR-26a targeting of the SMAD1 transcription factor

Dicer function is essential for lung epithelium morphogenesis.

Dicer is essential for mouse development

Morphogenesis in skin is governed by discrete sets of differentially expressed microRNAs

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10-21

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10.1016/J.YDBIO.2010.01.008

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20079730

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