Diabetes mellitus: channeling care through cellular discovery
about
Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTORFoxO Transcription Factors and Regenerative Pathways in Diabetes MellitusErythropoietin: new directions for the nervous systemNovel applications of trophic factors, Wnt and WISP for neuronal repair and regeneration in metabolic diseaseEarly apoptotic vascular signaling is determined by Sirt1 through nuclear shuttling, forkhead trafficking, bad, and mitochondrial caspase activation.Wnt1 neuroprotection translates into improved neurological function during oxidant stress and cerebral ischemia through AKT1 and mitochondrial apoptotic pathways.EPO relies upon novel signaling of Wnt1 that requires Akt1, FoxO3a, GSK-3β, and β-catenin to foster vascular integrity during experimental diabetes.Erythropoietin employs cell longevity pathways of SIRT1 to foster endothelial vascular integrity during oxidant stress.Mammalian target of rapamycin: hitting the bull's-eye for neurological disorders.Translating cell survival and cell longevity into treatment strategies with SIRT1Targeting cardiovascular disease with novel SIRT1 pathways.Erythropoietin and Wnt1 govern pathways of mTOR, Apaf-1, and XIAP in inflammatory microglia.SIRT1: new avenues of discovery for disorders of oxidative stressWnt1 Participates in Inflammation Induced by Lipopolysaccharide Through Upregulating Scavenger Receptor A and NF-kBMammalian target of rapamycin signaling in diabetic cardiovascular diseaseOxidant stress and signal transduction in the nervous system with the PI 3-K, Akt, and mTOR cascadeNovel directions for diabetes mellitus drug discoveryInflammatory markers and haptoglobin polymorphism in Saudi with non-insulin-dependent diabetes mellitusProgramming apoptosis and autophagy with novel approaches for diabetes mellitus.mTOR: Driving apoptosis and autophagy for neurocardiac complications of diabetes mellitus.The possible role of interleukin-33 as a new player in the pathogenesis of contrast-induced nephropathy in diabetic rats.The dual targeting of EGFR and ErbB2 with the inhibitor Lapatinib corrects high glucose-induced apoptosis and vascular dysfunction by opposing multiple diabetes-induced signaling changes.Bacille Calmette-Guérin/DNAhsp65 prime-boost is protective against diabetes in non-obese diabetic mice but not in the streptozotocin model of type 1 diabetes.
P2860
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P2860
Diabetes mellitus: channeling care through cellular discovery
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Diabetes mellitus: channeling care through cellular discovery
@ast
Diabetes mellitus: channeling care through cellular discovery
@en
type
label
Diabetes mellitus: channeling care through cellular discovery
@ast
Diabetes mellitus: channeling care through cellular discovery
@en
prefLabel
Diabetes mellitus: channeling care through cellular discovery
@ast
Diabetes mellitus: channeling care through cellular discovery
@en
P2093
P2860
P1476
Diabetes mellitus: channeling care through cellular discovery
@en
P2093
Jinling Hou
Kenneth Maiese
Yan Chen Shang
Zhao Zhong Chong
P2860
P356
10.2174/156720210790820217
P577
2010-02-01T00:00:00Z