The innate immune system is activated by stimulation of vaginal epithelial cells with Staphylococcus aureus and toxic shock syndrome toxin 1.
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P1343
Gram-positive bacterial superantigen outside-in signaling causes toxic shock syndromeStaphylococcal Superantigens Spark Host-Mediated Danger SignalsRole of Staphylococcus aureus Virulence Factors in Inducing Inflammation and Vascular Permeability in a Mouse Model of Bacterial EndophthalmitisCytolysins augment superantigen penetration of stratified mucosaSecreted virulence factor comparison between methicillin-resistant and methicillin-sensitive Staphylococcus aureus, and its relevance to atopic dermatitis.Staphylococcus aureus exotoxins are present in vivo in tamponsDermal wound transcriptomic responses to Infection with Pseudomonas aeruginosa versus Klebsiella pneumoniae in a rabbit ear wound model.Staphylococcus aureus Biofilm and Planktonic cultures differentially impact gene expression, mapk phosphorylation, and cytokine production in human keratinocytes.Proinflammatory exoprotein characterization of toxic shock syndrome Staphylococcus aureus.Staphylococcal superantigens interact with multiple host receptors to cause serious diseases.Epithelial proinflammatory response and curcumin-mediated protection from staphylococcal toxic shock syndrome toxin-1Staphylococcal and streptococcal superantigen exotoxinsThe contribution of group A streptococcal virulence determinants to the pathogenesis of sepsis.Enterococcus faecalis inhibits superantigen toxic shock syndrome toxin-1-induced interleukin-8 from human vaginal epithelial cells through tetramic acidsChecks and balances: the ocular response to infection.Response of corneal epithelial cells to Staphylococcus aureus.Glycerol monolaurate prevents mucosal SIV transmissionSuperantigens of Staphylococcus aureus from patients with diabetic foot ulcers.New insights into the prevention of staphylococcal infections and toxic shock syndrome.Staphylococcal superantigens stimulate immortalized human adipocytes to produce chemokines.Staphylococcus aureus enterotoxins induce IL-8 secretion by human nasal epithelial cells.Adherence and blocking of Candida albicans to cultured vaginal epithelial cells: treatments to decrease adherence.Novel antimicrobial peptides that inhibit gram positive bacterial exotoxin synthesis.Exosomes in human semen restrict HIV-1 transmission by vaginal cells and block intravaginal replication of LP-BM5 murine AIDS virus complex.A Multiplex Assay for Detection of Staphylococcal and Streptococcal Exotoxins.Lactobacillus crispatus inhibits growth of Gardnerella vaginalis and Neisseria gonorrhoeae on a porcine vaginal mucosa model.Lactobacilli require physical contact to reduce staphylococcal TSST-1 secretion and vaginal epithelial inflammatory response.Novel Tissue Level Effects of the Staphylococcus aureus Enterotoxin Gene Cluster Are Essential for Infective Endocarditis.Local Epidermal Growth Factor Receptor Signaling Mediates the Systemic Pathogenic Effects of Staphylococcus aureus Toxic Shock SyndromeImmunity to Staphylococcus aureus secreted proteins protects rabbits from serious illnesses.Probiotic lactobacillus and estrogen effects on vaginal epithelial gene expression responses to Candida albicansAlpha-toxin promotes Staphylococcus aureus mucosal biofilm formation.A disintegrin and metalloproteinase 17 (ADAM17) and epidermal growth factor receptor (EGFR) signaling drive the epithelial response to Staphylococcus aureus toxic shock syndrome toxin-1 (TSST-1)Vaginal toxic shock reaction triggering desquamative inflammatory vaginitisStaphylococcal Enterotoxin O Exhibits Cell Cycle Modulating Activity.Norepinephrine potentiates proinflammatory responses of human vaginal epithelial cells.Glycerol monolaurate inhibits the effects of Gram-positive select agents on eukaryotic cells.Glycerol monolaurate does not alter rhesus macaque (Macaca mulatta) vaginal lactobacilli and is safe for chronic useNovel toxic shock syndrome toxin-1 amino acids required for biological activity.Temporal and Racial Differences Associated with Atopic Dermatitis Staphylococcusaureus and Encoded Virulence Factors
P2860
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P2860
The innate immune system is activated by stimulation of vaginal epithelial cells with Staphylococcus aureus and toxic shock syndrome toxin 1.
description
2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
The innate immune system is ac ...... toxic shock syndrome toxin 1.
@ast
The innate immune system is ac ...... toxic shock syndrome toxin 1.
@en
type
label
The innate immune system is ac ...... toxic shock syndrome toxin 1.
@ast
The innate immune system is ac ...... toxic shock syndrome toxin 1.
@en
prefLabel
The innate immune system is ac ...... toxic shock syndrome toxin 1.
@ast
The innate immune system is ac ...... toxic shock syndrome toxin 1.
@en
P2093
P2860
P1476
The innate immune system is ac ...... toxic shock syndrome toxin 1.
@en
P2093
Catherine C Davis
Christopher A Squier
Kevin Ault
Marnie L Peterson
Mary J Kremer
P2860
P304
P356
10.1128/IAI.73.4.2164-2174.2005
P407
P577
2005-04-01T00:00:00Z