The Epstein-Barr virus major latent promoter Qp is constitutively active, hypomethylated, and methylation sensitive
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Epigenetic Impact on EBV Associated B-Cell LymphomagenesisOPCML is a broad tumor suppressor for multiple carcinomas and lymphomas with frequently epigenetic inactivation.Epigenetic regulation of EBV persistence and oncogenesis.Interferon regulatory factor 7 is induced by Epstein-Barr virus latent membrane protein 1Reactivation of the human cytomegalovirus major immediate-early regulatory region and viral replication in embryonal NTera2 cells: role of trichostatin A, retinoic acid, and deletion of the 21-base-pair repeats and modulator.BZLF1 activation of the methylated form of the BRLF1 immediate-early promoter is regulated by BZLF1 residue 186.Protein-DNA binding and CpG methylation at nucleotide resolution of latency-associated promoters Qp, Cp, and LMP1p of Epstein-Barr virusMethylation profiling of Epstein-Barr virus immediate-early gene promoters, BZLF1 and BRLF1 in tumors of epithelial, NK- and B-cell origins.BZLF1 governs CpG-methylated chromatin of Epstein-Barr Virus reversing epigenetic repression.The importance of epigenetic alterations in the development of epstein-barr virus-related lymphomas.Tonsillar memory B cells, latently infected with Epstein-Barr virus, express the restricted pattern of latent genes previously found only in Epstein-Barr virus-associated tumors.The Epstein-Barr virus latency BamHI-Q promoter is positively regulated by STATs and Zta interference with JAK/STAT activation leads to loss of BamHI-Q promoter activityMethylation status of the Epstein-Barr virus major latent promoter C in iatrogenic B cell lymphoproliferative disease. Application of PCR-based analysis.Epigenetic silencing of a Ca(2+)-regulated Ras GTPase-activating protein RASAL defines a new mechanism of Ras activation in human cancers.Epigenetic mechanisms in virus-induced tumorigenesis.Epigenetic silencing of the 3p22 tumor suppressor DLEC1 by promoter CpG methylation in non-Hodgkin and Hodgkin lymphomas.Occupancy of chromatin organizers in the Epstein-Barr virus genome.Epstein-Barr virus transcription activator R upregulates BARF1 expression by direct binding to its promoter, independent of methylation.Methylation of transcription factor binding sites in the Epstein-Barr virus latent cycle promoter Wp coincides with promoter down-regulation during virus-induced B-cell transformation.Differential methylation of Epstein-Barr virus latency promoters facilitates viral persistence in healthy seropositive individuals.NF-κB Signaling Regulates Epstein-Barr Virus BamHI-Q-Driven EBNA1 Expression.
P2860
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P2860
The Epstein-Barr virus major latent promoter Qp is constitutively active, hypomethylated, and methylation sensitive
description
1998 nî lūn-bûn
@nan
1998 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
The Epstein-Barr virus major l ...... ted, and methylation sensitive
@ast
The Epstein-Barr virus major l ...... ted, and methylation sensitive
@en
type
label
The Epstein-Barr virus major l ...... ted, and methylation sensitive
@ast
The Epstein-Barr virus major l ...... ted, and methylation sensitive
@en
prefLabel
The Epstein-Barr virus major l ...... ted, and methylation sensitive
@ast
The Epstein-Barr virus major l ...... ted, and methylation sensitive
@en
P2093
P2860
P1433
P1476
The Epstein-Barr virus major l ...... ted, and methylation sensitive
@en
P2093
A Hildesheim
K D Robertson
R F Ambinder
P2860
P304
P577
1998-09-01T00:00:00Z