Calcium and ER stress mediate hepatic apoptosis after burn injury. - sprookjes - yvandenbroek - TriplyDB

Calcium and ER stress mediate hepatic apoptosis after burn injury.

Calcium and ER stress mediate hepatic apoptosis after burn injury.

http://www.wikidata.org/entity/Q33793882

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14-Deoxyandrographolide desensitizes hepatocytes to tumour necrosis factor-alpha-induced apoptosis through calcium-dependent tumour necrosis factor receptor superfamily member 1A release via the NO/cGMP pathway The dual role of calcium as messenger and stressor in cell damage, death, and survival.Simvastatin protects hepatocytes from apoptosis by suppressing the TNF-α/caspase-3 signaling pathway in mice with burn injury Intensive insulin therapy in severely burned pediatric patients: a prospective randomized trial.Hepatic autophagy after severe burn in response to endoplasmic reticulum stress.Simvastatin potently induces calcium-dependent apoptosis of human leiomyoma cells.Insulin protects against hepatic damage postburn.Endoplasmic reticulum stress and insulin resistance post-trauma: similarities to type 2 diabetes.Novel mitochondria-targeted antioxidant peptide ameliorates burn-induced apoptosis and endoplasmic reticulum stress in the skeletal muscle of mice Physiologic and molecular changes in the tracheal epithelium of rats following burn injury.Measurement of hepatic protein fractional synthetic rate with stable isotope labeling technique in thapsigargin stressed HepG2 cells.Amplified RLR signaling activation through an interferon-stimulated gene-endoplasmic reticulum stress-mitochondrial calcium uniporter protein loop.Propranolol improves impaired hepatic phosphatidylinositol 3-kinase/akt signaling after burn injury.Severe injury is associated with insulin resistance, endoplasmic reticulum stress response, and unfolded protein response.Burn plus lipopolysaccharide augments endoplasmic reticulum stress and NLRP3 inflammasome activation and reduces PGC-1α in liver Differential acute and chronic effects of burn trauma on murine skeletal muscle bioenergetics Hepatic apoptosis postburn is mediated by c-Jun N-terminal kinase 2.Caspase 3 cleavage of the inositol 1,4,5-trisphosphate receptor does not contribute to apoptotic calcium release.Thermal injury activates the eEF2K-dependent eEF2 pathway in pediatric patients.XBP-1s is linked to suppressed gluconeogenesis in the Ebb phase of burn injury.Burned Adults Develop Profound Glucose Intolerance.Occurrence of multiorgan dysfunction in pediatric burn patients: incidence and clinical outcome.Severe burn-induced endoplasmic reticulum stress and hepatic damage in mice The hepatic response to thermal injury: is the liver important for postburn outcomes?Fenofibrate does not affect burn-induced hepatic endoplasmic reticulum stress Glucose control in severely thermally injured pediatric patients: what glucose range should be the target?Post-burn hepatic insulin resistance is associated with endoplasmic reticulum (ER) stress Endoplasmic Reticulum Stress and Related Pathological Processes.Endoplasmic-reticulum calcium depletion and disease.White Adipose Tissue Browning: A Double-edged Sword.Autophagy mediated by endoplasmic reticulum stress enhances the caffeine-induced apoptosis of hepatic stellate cells.Modeling Acute ER Stress in Vivo and in Vitro.Xanthine oxidase contributes to sustained airway epithelial oxidative stress after scald burn.Alcohol Modulation of the Postburn Hepatic Response.

P2860

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P2860

Calcium and ER stress mediate hepatic apoptosis after burn injury.

http://www.wikidata.org/entity/Q33793882

description

2009 nî lūn-bûn

@nan

2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Calcium and ER stress mediate hepatic apoptosis after burn injury.

@ast

Calcium and ER stress mediate hepatic apoptosis after burn injury.

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type

label

Calcium and ER stress mediate hepatic apoptosis after burn injury.

@ast

Calcium and ER stress mediate hepatic apoptosis after burn injury.

@en

prefLabel

Calcium and ER stress mediate hepatic apoptosis after burn injury.

@ast

Calcium and ER stress mediate hepatic apoptosis after burn injury.

@en

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J.1582-4934.2008.00644.X

J.1582-4934.2009.00644.X

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Journal of Cellular and Molecular Medicine

P1476

Calcium and ER stress mediate hepatic apoptosis after burn injury

@en

P2093

Celeste C Finnerty

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David N Herndon

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Gabriela A Kulp

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Gerd G Gauglitz

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José M Barral

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Juquan Song

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Marc G Jeschke

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Robert A Cox

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P2860

Signal integration in the endoplasmic reticulum unfolded protein response

Chemical chaperones reduce ER stress and restore glucose homeostasis in a mouse model of type 2 diabetes

ER stress triggers apoptosis by activating BH3-only protein Bim

Intensive insulin therapy in critically ill patients

Requirement of biphasic calcium release from the endoplasmic reticulum for Fas-mediated apoptosis.

Bcl-X(L) affects Ca(2+) homeostasis by altering expression of inositol 1,4,5-trisphosphate receptors

Control of apoptosis by IP(3) and ryanodine receptor driven calcium signals.

Anti-cachectin/TNF monoclonal antibodies prevent septic shock during lethal bacteraemia.

Cytochrome c binds to inositol (1,4,5) trisphosphate receptors, amplifying calcium-dependent apoptosis.

Apoptosis-based therapies.

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1857-1865

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10.1111/J.1582-4934.2008.00644.X

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8B

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Darren F. Boehning

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2009-08-01T00:00:00Z

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41416057

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20141609

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