EZH1 and EZH2 promote skeletal growth by repressing inhibitors of chondrocyte proliferation and hypertrophy - sprookjes - yvandenbroek - TriplyDB

EZH1 and EZH2 promote skeletal growth by repressing inhibitors of chondrocyte proliferation and hypertrophy

EZH1 and EZH2 promote skeletal growth by repressing inhibitors of chondrocyte proliferation and hypertrophy

http://www.wikidata.org/entity/Q33815652

about

Genetic Dissection of a QTL Affecting Bone Geometry.Anti-inflammatory and chondroprotective effects of the S-adenosylhomocysteine hydrolase inhibitor 3-Deazaneplanocin A, in human articular chondrocytes.Programmed cell senescence in skeleton during late puberty.Growth and growth disorders in 2017: Genetic and epigenetic regulation of childhood growth.PRC2 Is Dispensable in Vivo for β-Catenin-Mediated Repression of Chondrogenesis in the Mouse Embryonic Cranial Mesenchyme.Hypophosphatemia Regulates Molecular Mechanisms of Circadian Rhythm MicroRNA-128a represses chondrocyte autophagy and exacerbates knee osteoarthritis by disrupting Atg12

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EZH1 and EZH2 promote skeletal growth by repressing inhibitors of chondrocyte proliferation and hypertrophy

http://www.wikidata.org/entity/Q33815652

description

2016 nî lūn-bûn

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2016 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2016 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

EZH1 and EZH2 promote skeletal ...... proliferation and hypertrophy

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EZH1 and EZH2 promote skeletal ...... proliferation and hypertrophy

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type

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EZH1 and EZH2 promote skeletal ...... proliferation and hypertrophy

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EZH1 and EZH2 promote skeletal ...... proliferation and hypertrophy

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EZH1 and EZH2 promote skeletal ...... proliferation and hypertrophy

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EZH1 and EZH2 promote skeletal ...... proliferation and hypertrophy

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Nature Communications

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EZH1 and EZH2 promote skeletal ...... proliferation and hypertrophy

@en

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Chithra Keembiyehetty

http://www.w3.org/2001/XMLSchema#string

Jeffrey Baron

http://www.w3.org/2001/XMLSchema#string

Julian C Lui

http://www.w3.org/2001/XMLSchema#string

Kevin M Barnes

http://www.w3.org/2001/XMLSchema#string

Michal Ad

http://www.w3.org/2001/XMLSchema#string

Presley Garrison

http://www.w3.org/2001/XMLSchema#string

Quang Nguyen

http://www.w3.org/2001/XMLSchema#string

Weiping Chen

http://www.w3.org/2001/XMLSchema#string

Youn Hee Jee

http://www.w3.org/2001/XMLSchema#string

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EZH1 and EZH2 cogovern histone H3K27 trimethylation and are essential for hair follicle homeostasis and wound repair

NSD1 mutations are the major cause of Sotos syndrome and occur in some cases of Weaver syndrome but are rare in other overgrowth phenotypes

EZH1 mediates methylation on histone H3 lysine 27 and complements EZH2 in maintaining stem cell identity and executing pluripotency

Role of histone H3 lysine 27 methylation in Polycomb-group silencing

A novel mutation in EED associated with overgrowth

The methyltransferases enhancer of zeste homolog (EZH) 1 and EZH2 control hepatocyte homeostasis and regeneration

Mice Deficient in AKAP13 (BRX) Are Osteoporotic and Have Impaired Osteogenesis

Weaver Syndrome-Associated EZH2 Protein Variants Show Impaired Histone Methyltransferase Function In Vitro

Dietary omega-3 polyunsaturated fatty acids suppress expression of EZH2 in breast cancer cells.

Coordinated postnatal down-regulation of multiple growth-promoting genes: evidence for a genetic program limiting organ growth.

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13685

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10.1038/NCOMMS13685

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7

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2016-11-29T00:00:00Z

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27897169

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