Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner - sprookjes - yvandenbroek - TriplyDB

Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner

Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner

http://www.wikidata.org/entity/Q33822643

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Harnessing Mechanistic Knowledge on Beneficial Versus Deleterious IFN-I Effects to Design Innovative Immunotherapies Targeting Cytokine Activity to Specific Cell Types Natural killer cells in hepatitis C: Current progress Inflammatory status in human hepatic cirrhosis NK cell function and receptor diversity in the context of HCV infection Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experience Immune responses to HCV and other hepatitis viruses STAT proteins - key regulators of anti-viral responses, inflammation, and tumorigenesis in the liver Natural killer cells in liver disease Insufficient natural killer cell responses against retroviruses: how to improve NK cell killing of retrovirus-infected cells Treatment of Hepatitis C in Patients Undergoing Immunosuppressive Drug Therapy Disease-promoting effects of type I interferons in viral, bacterial, and coinfections IL28B, HLA-C, and KIR variants additively predict response to therapy in chronic hepatitis C virus infection in a European Cohort: a cross-sectional study Interferon-α improves phosphoantigen-induced Vγ9Vδ2 T-cells interferon-γ production during chronic HCV infection Hepatitis C virus impairs natural killer cell activity via viral serine protease NS3.Long-term effect on natural killer cells by interferon-α therapy on the outcomes of HCV infection.NK cell terminal differentiation: correlated stepwise decrease of NKG2A and acquisition of KIRs Dynamic Changes of the Frequency of Classic and Inflammatory Monocytes Subsets and Natural Killer Cells in Chronic Hepatitis C Patients Treated by Direct-Acting Antiviral Agents.Hepatitis C virus (HCV) evades NKG2D-dependent NK cell responses through NS5A-mediated imbalance of inflammatory cytokines.Protective immunity against hepatitis C: many shades of gray.Blockade of immunosuppressive cytokines restores NK cell antiviral function in chronic hepatitis B virus infection Changes in inflammatory biomarkers in HCV-infected patients undergoing direct acting antiviral-containing regimens with or without interferon Innate immune function in placenta and cord blood of hepatitis C--seropositive mother-infant dyads.Hepatocyte death: a clear and present danger.Increased degranulation of natural killer cells during acute HCV correlates with the magnitude of virus-specific T cell responses.Activation of natural killer cells during acute infection with hepatitis C virus.Effect of recombinant cytokines on the expression of natural killer cell receptors from patients with TB or/and HIV infection.A look behind closed doors: interaction of persistent viruses with dendritic cells.Innate immune cell networking in hepatitis C virus infection Consistent beneficial effects of killer cell immunoglobulin-like receptor 2DL3 and group 1 human leukocyte antigen-C following exposure to hepatitis C virus.Natural killer cells are characterized by the concomitantly increased interferon-γ and cytotoxicity in acute resolved hepatitis B patients.Natural Killer Cells in Viral Hepatitis.A humanized mouse model to study hepatitis C virus infection, immune response, and liver disease.Variation in both IL28B and KIR2DS3 genes influence pegylated interferon and ribavirin hepatitis C treatment outcome in HIV-1 co-infection.Innate immune genes synergize to predict increased risk of chronic disease in hepatitis C virus infection.Molecular mechanisms of alcoholic liver disease: innate immunity and cytokines.KIR/HLA interactions and pathogen immunity In vivo consequences of liver-specific interleukin-22 expression in mice: Implications for human liver disease progression.Functions of liver natural killer cells are dependent on the severity of liver inflammation and fibrosis in chronic hepatitis C.Role of TRAIL and the pro-apoptotic Bcl-2 homolog Bim in acetaminophen-induced liver damage.Cell-to-cell contact with hepatitis C virus-infected cells reduces functional capacity of natural killer cells.

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P2860

Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner

http://www.wikidata.org/entity/Q33822643

description

2009 nî lūn-bûn

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2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

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Natural killer cells are polar ...... terferon-alfa-dependent manner

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Natural killer cells are polar ...... terferon-alfa-dependent manner

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Natural killer cells are polar ...... terferon-alfa-dependent manner

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Natural killer cells are polar ...... terferon-alfa-dependent manner

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Natural killer cells are polar ...... terferon-alfa-dependent manner

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Natural killer cells are polar ...... terferon-alfa-dependent manner

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J.GASTRO.2009.08.066

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Gastroenterology

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Natural killer cells are polar ...... terferon-alfa-dependent manner

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Barbara Rehermann

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Birgit Edlich

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Christopher Koh

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Theo Heller

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P2860

Enhanced expression of interferon-regulated genes in the liver of patients with chronic hepatitis C virus infection: detection by suppression-subtractive hybridization

DNA microarray analysis of chimpanzee liver during acute resolving hepatitis C virus infection.

Viral and immunological determinants of hepatitis C virus clearance, persistence, and disease.

Nonlinear partial differential equations and applications: Genomic analysis of the host response to hepatitis C virus infection

Virus-induced type I IFN stimulates generation of immunoproteasomes at the site of infection

Liver-Infiltrating Lymphocytes in Chronic Human Hepatitis C Virus Infection Display an Exhausted Phenotype with High Levels of PD-1 and Low Levels of CD127 Expression

Inhibition of Natural Killer Cells through Engagement of CD81 by the Major Hepatitis C Virus Envelope Protein

Binding of the Hepatitis C Virus Envelope Protein E2 to CD81 Inhibits Natural Killer Cell Functions

Heterologous T cell immunity in severe hepatitis C virus infection

Natural killer cells suppress full cycle HCV infection of human hepatocytes

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10.1053/J.GASTRO.2009.08.066

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1

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Golo Ahlenstiel

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26806153

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