The major immediate-early gene ie3 of mouse cytomegalovirus is essential for viral growth.
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An Interferon Regulated MicroRNA Provides Broad Cell-Intrinsic Antiviral Immunity through Multihit Host-Directed Targeting of the Sterol PathwayDiscrete clusters of virus-encoded micrornas are associated with complementary strands of the genome and the 7.2-kilobase stable intron in murine cytomegalovirusElimination of ie1 significantly attenuates murine cytomegalovirus virulence but does not alter replicative capacity in cell cultureEnrichment of immediate-early 1 (m123/pp89) peptide-specific CD8 T cells in a pulmonary CD62L(lo) memory-effector cell pool during latent murine cytomegalovirus infection of the lungsRat cytomegalovirus major immediate-early enhancer switching results in altered growth characteristics.Random, asynchronous, and asymmetric transcriptional activity of enhancer-flanking major immediate-early genes ie1/3 and ie2 during murine cytomegalovirus latency in the lungs.Enhancerless cytomegalovirus is capable of establishing a low-level maintenance infection in severely immunodeficient host tissues but fails in exponential growth.Frequent coinfection of cells explains functional in vivo complementation between cytomegalovirus variants in the multiply infected hostConstruction of a rationally designed human cytomegalovirus variant encoding a temperature-sensitive immediate-early 2 protein.Combined agonist-antagonist genome-wide functional screening identifies broadly active antiviral microRNAsExpression of m157, a murine cytomegalovirus-encoded putative major histocompatibility class I (MHC-I)-like protein, is independent of viral regulation of host MHC-I.Requirement of multiple cis-acting elements in the human cytomegalovirus major immediate-early distal enhancer for viral gene expression and replication.Persisting murine cytomegalovirus can reactivate and has unique transcriptional activity in ocular tissue.Ablation of the regulatory IE1 protein of murine cytomegalovirus alters in vivo pro-inflammatory TNF-alpha production during acute infectionThe human cytomegalovirus major immediate-early enhancer determines the efficiency of immediate-early gene transcription and viral replication in permissive cells at low multiplicity of infection.An essential role of the enhancer for murine cytomegalovirus in vivo growth and pathogenesis.Absence of IE1 p72 protein function during low-multiplicity infection by human cytomegalovirus results in a broad block to viral delayed-early gene expression.CD8 T cells control cytomegalovirus latency by epitope-specific sensing of transcriptional reactivationMast cells expedite control of pulmonary murine cytomegalovirus infection by enhancing the recruitment of protective CD8 T cells to the lungs.The effect of murine cytomegalovirus IE-3 specific shRNA is dependent on intragenic target site due to multiple transcription initiation sitesA temporal gate for viral enhancers to co-opt Toll-like-receptor transcriptional activation pathways upon acute infectionReversible inhibition of murine cytomegalovirus replication by gamma interferon (IFN-γ) in primary macrophages involves a primed type I IFN-signaling subnetwork for full establishment of an immediate-early antiviral state.CD200 receptor restriction of myeloid cell responses antagonizes antiviral immunity and facilitates cytomegalovirus persistence within mucosal tissue.Mast cells as rapid innate sensors of cytomegalovirus by TLR3/TRIF signaling-dependent and -independent mechanismsA short cis-acting motif in the M112-113 promoter region is essential for IE3 to activate M112-113 gene expression and is important for murine cytomegalovirus replicationFunctional Dissection of an Alternatively Spliced Herpesvirus Gene by Splice Site MutagenesisTransplant-induced reactivation of murine cytomegalovirus immediate early gene expression is associated with recruitment of NF-κB and AP-1 to the major immediate early promoter.Cell cycle-independent expression of immediate-early gene 3 results in G1 and G2 arrest in murine cytomegalovirus-infected cells.Transactivation of cellular genes involved in nucleotide metabolism by the regulatory IE1 protein of murine cytomegalovirus is not critical for viral replicative fitness in quiescent cells and host tissuesNeutrality of the canonical NF-kappaB-dependent pathway for human and murine cytomegalovirus transcription and replication in vitro.Epigenetic control of cytomegalovirus latency and reactivation.A new reporter mouse cytomegalovirus reveals maintained immediate-early gene expression but poor virus replication in cycling liver sinusoidal endothelial cells.Liver sinusoidal endothelial cells are a site of murine cytomegalovirus latency and reactivationThe p36 isoform of murine cytomegalovirus m152 protein suffices for mediating innate and adaptive immune evasion.Role for tumor necrosis factor alpha in murine cytomegalovirus transcriptional reactivation in latently infected lungs.Mouse cytomegalovirus early M112/113 proteins control the repressive effect of IE3 on the major immediate-early promoter.Viral latency drives 'memory inflation': a unifying hypothesis linking two hallmarks of cytomegalovirus infection.Changes to lipid droplet configuration in mCMV-infected fibroblasts: live cell imaging with simultaneous CARS and two-photon fluorescence microscopy.Deletion of the rat cytomegalovirus immediate-early 1 gene results in a virus capable of establishing latency, but with lower levels of acute virus replication and latency that compromise reactivation efficiency.Stepwise adaptation of murine cytomegalovirus to cells of a foreign host for identification of host range determinants.
P2860
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P2860
The major immediate-early gene ie3 of mouse cytomegalovirus is essential for viral growth.
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2000 nî lūn-bûn
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The major immediate-early gene ...... is essential for viral growth.
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The major immediate-early gene ...... is essential for viral growth.
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The major immediate-early gene ...... is essential for viral growth.
@ast
The major immediate-early gene ...... is essential for viral growth.
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The major immediate-early gene ...... is essential for viral growth.
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The major immediate-early gene ...... is essential for viral growth.
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P2860
P1433
P1476
The major immediate-early gene ...... is essential for viral growth.
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P2093
P2860
P304
11129-11136
P356
10.1128/JVI.74.23.11129-11136.2000
P577
2000-12-01T00:00:00Z