MMPs initiate Schwann cell-mediated MBP degradation and mechanical nociception after nerve damage.
about
Frequency-dependent effects of vibration on peripheral nerves and sensory nerve function in a rat model of hand-arm vibration syndrome. (Erratum: Characterization of frequency-dependent responses of the vascular systemGlial cells and chronic painProtease regulation: the Yin and Yang of neural development and diseaseNOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9Efficacy of a metalloproteinase inhibitor in spinal cord injured dogs.Matrix metalloproteinase inhibition enhances the rate of nerve regeneration in vivo by promoting dedifferentiation and mitosis of supporting schwann cellsThe MMP-9/TIMP-1 axis controls the status of differentiation and function of myelin-forming Schwann cells in nerve regeneration.Schwann cell-derived Apolipoprotein D controls the dynamics of post-injury myelin recognition and degradationRole of matrix metalloproteinases and therapeutic benefits of their inhibition in spinal cord injury.Matrix metalloproteinase-14 both sheds cell surface neuronal glial antigen 2 (NG2) proteoglycan on macrophages and governs the response to peripheral nerve injuryActive immunization with myelin-derived altered peptide ligand reduces mechanical pain hypersensitivity following peripheral nerve injuryDistribution and tumor necrosis factor-alpha isoform binding specificity of locally administered etanercept into injured and uninjured rat sciatic nerve.The alternatively spliced fibronectin CS1 isoform regulates IL-17A levels and mechanical allodynia after peripheral nerve injury.Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.Matrix metalloproteinases in neuropathic pain and migraine: friends, enemies, and therapeutic targets.miR-182 inhibits Schwann cell proliferation and migration by targeting FGF9 and NTM, respectively at an early stage following sciatic nerve injury.Discovering cytokines as targets for chemotherapy-induced painful peripheral neuropathy.Regulation of cytokine expression by Schwann cells in response to α2-macroglobulin binding to LRP1.Berberine ameliorates cold and mechanical allodynia in a rat model of diabetic neuropathyDivergent role for MMP-2 in myelin breakdown and oligodendrocyte death following transient global ischemia.MMP-9 controls Schwann cell proliferation and phenotypic remodeling via IGF-1 and ErbB receptor-mediated activation of MEK/ERK pathway.Heterogeneity of serum gelatinases MMP-2 and MMP-9 isoforms and charge variants.Therapeutic potential of matrix metalloprotease inhibitors in neuropathic pain.N-acetyl-cysteine attenuates remifentanil-induced postoperative hyperalgesia via inhibiting matrix metalloproteinase-9 in dorsal root ganglia.Matrix metalloproteinases and neurotrauma: evolving roles in injury and reparative processes.Matrix metalloproteinases: potential therapeutic target for diabetic neuropathic pain.Spinal cord injury induced neuropathic pain: Molecular targets and therapeutic approaches.Reciprocal relationship between membrane type 1 matrix metalloproteinase and the algesic peptides of myelin basic protein contributes to chronic neuropathic pain.Inhibiting HMGB1 with Glycyrrhizic Acid Protects Brain Injury after DAI via Its Anti-Inflammatory Effect.Spinal activity of interleukin 6 mediates myelin basic protein-induced allodynia.Neuron-Derived ADAM10 Production Stimulates Peripheral Nerve Injury-Induced Neuropathic Pain by Cleavage of E-Cadherin in Satellite Glial Cells.Activation and expression of μ-calpain in dorsal root contributes to RTX-induced mechanical allodynia.Autotaxin and lysophosphatidic acid1 receptor-mediated demyelination of dorsal root fibers by sciatic nerve injury and intrathecal lysophosphatidylcholine.Protective reactivity and alteration of the brain tissue in alcoholics evidenced by SOD1, MMP9 immunohistochemistry, and electron microscopy.Valproic acid attenuates blood-spinal cord barrier disruption by inhibiting matrix metalloprotease-9 activity and improves functional recovery after spinal cord injury.A sensitive and selective ELISA methodology quantifies a demyelination marker in experimental and clinical samples.Acute- and late-phase matrix metalloproteinase (MMP)-9 activity is comparable in female and male rats after peripheral nerve injury.Levetiracetam exhibits protective properties on rat Schwann cells in vitro.
P2860
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P2860
MMPs initiate Schwann cell-mediated MBP degradation and mechanical nociception after nerve damage.
description
2008 nî lūn-bûn
@nan
2008 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
MMPs initiate Schwann cell-med ...... ociception after nerve damage.
@ast
MMPs initiate Schwann cell-med ...... ociception after nerve damage.
@en
type
label
MMPs initiate Schwann cell-med ...... ociception after nerve damage.
@ast
MMPs initiate Schwann cell-med ...... ociception after nerve damage.
@en
prefLabel
MMPs initiate Schwann cell-med ...... ociception after nerve damage.
@ast
MMPs initiate Schwann cell-med ...... ociception after nerve damage.
@en
P2093
P2860
P1476
MMPs initiate Schwann cell-med ...... ociception after nerve damage.
@en
P2093
Hideo Kobayashi
Jennifer Dolkas
Kinshi Kato
Robert R Myers
Sharmila Chattopadhyay
Shin-Ichi Kikuchi
Veronica I Shubayev
P2860
P304
P356
10.1016/J.MCN.2008.08.008
P577
2008-09-05T00:00:00Z