about
Protease-activated receptor-1 activation of endothelial cells induces protein kinase Calpha-dependent phosphorylation of syntaxin 4 and Munc18c: role in signaling p-selectin expressionTumor necrosis factor disrupts claudin-5 endothelial tight junction barriers in two distinct NF-κB-dependent phasesInflammation and its role in age-related macular degenerationTemporal and spatial patterns of ovarian gene transcription following an ovulatory dose of gonadotropin in the ratAtomic force microscope studies of the fusion of floating lipid bilayers.Relevance of neutrophils in the murine model of haemolytic uraemic syndrome: mechanisms involved in Shiga toxin type 2-induced neutrophilia.The functional state of neutrophils correlates with the severity of renal dysfunction in children with hemolytic uremic syndrome.Investigation of SNARE-Mediated Membrane Fusion Mechanism Using Atomic Force Microscopy.Identification of pathway-selective estrogen receptor ligands that inhibit NF-kappaB transcriptional activity.Bluetongue virus infection activates bovine monocyte-derived macrophages and pulmonary artery endothelial cellsPTX3 in small-vessel vasculitides: an independent indicator of disease activity produced at sites of inflammation.The unique characteristics of tumor vasculature and preclinical evidence for its selective disruption by Tumor-Vascular Disrupting Agents.Non-diabetic microalbuminuria, endothelial dysfunction and cardiovascular disease.Chemical modulators of sphingosine-1-phosphate receptors as barrier-oriented therapeutic moleculesNeutrophil response to dental plaque by gender and raceVasculitis: mechanisms involved and clinical manifestationsMarrow-derived cells regulate the development of early diabetic retinopathy and tactile allodynia in miceMicrobial induction of vascular pathology in the CNS.Neutrophil influx and chemokine production during the early phases of the antitumor response to the vascular disrupting agent DMXAA (ASA404)Nuclear factor-kappaB regulates estrogen receptor-alpha transcription in the human heart.Inhibition of VEGF or TGF-{beta} signaling activates endothelium and increases leukocyte rolling.The enhanced permeability retention effect: a new paradigm for drug targeting in infection.Dynamic intraoperative MRI in transsphenoidal resection of pituitary macroadenomas: a quantitative analysis.Pathological Characterization Of IFNAR(-/-) Mice Infected With Bluetongue Virus Serotype 4Syndecan 1 shedding contributes to Pseudomonas aeruginosa sepsis.Role of endothelial nitric oxide synthase-derived nitric oxide in activation and dysfunction of cerebrovascular endothelial cells during early onsets of sepsis.Transcriptome analysis of inflammation-related gene expression in endothelial cells activated by complement MASP-1.Adeno-Associated Viral Vector-Mediated mTOR Inhibition by Short Hairpin RNA Suppresses Laser-Induced Choroidal Neovascularization.Pre-conditioning cryosurgery: cellular and molecular mechanisms and dynamics of TNF-α enhanced cryotherapy in an in vivo prostate cancer model system.Toll-like receptor 4-dependent responses to lung injury in a murine model of pulmonary contusion.Relationship between oxidative burst activity and CD11b expression in neutrophils and monocytes from healthy individuals: effects of race and gender.Pulmonary contusion primes systemic innate immunity responses.Mono(2-ethylhexyl) phthalate induces both pro- and anti-inflammatory responses in rat alveolar macrophages through crosstalk between p38, the lipoxygenase pathway and PPARalpha.Vasopressin vs norepinephrine in endotoxic shock: systemic, renal, and splanchnic hemodynamic and oxygen transport effects.Exercise-induced myofibrillar disruption with sarcolemmal integrity prior to simulated diving has no effect on vascular bubble formation in rats.Potential role of proinflammatory cytokines in the pathogenetic mechanisms of vascular lesions in goats naturally infected with bluetongue virus serotype 1.Cardiovascular and hematologic effects produced by chronic treatment with etoricoxib in normotensive rats.Effects of hydroxyethyl starch on hepatic production of cytokines and activation of transcription factors in lipopolysaccharide-administered rats.Hydroxyethyl starch inhibits intestinal production of cytokines and activation of transcription factors in endotoxaemic rats.Concentration of acute phase factors in vitreous fluid in diabetic macular edema.
P2860
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P2860
description
2000 nî lūn-bûn
@nan
2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Regulation of inflammatory vascular damage.
@ast
Regulation of inflammatory vascular damage.
@en
type
label
Regulation of inflammatory vascular damage.
@ast
Regulation of inflammatory vascular damage.
@en
prefLabel
Regulation of inflammatory vascular damage.
@ast
Regulation of inflammatory vascular damage.
@en
P1476
Regulation of inflammatory vascular damage.
@en
P2093
Lentsch AB
P2860
P304
P356
10.1002/(SICI)1096-9896(200002)190:3<343::AID-PATH522>3.0.CO;2-M
P577
2000-02-01T00:00:00Z