Neuronal death in amyotrophic lateral sclerosis is apoptosis: possible contribution of a programmed cell death mechanism.
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Olesoxime, a cholesterol-like neuroprotectant for the potential treatment of amyotrophic lateral sclerosisInhibitory synaptic regulation of motoneurons: a new target of disease mechanisms in amyotrophic lateral sclerosis.Bax and Bcl-2 interaction in a transgenic mouse model of familial amyotrophic lateral sclerosisAberrant regulation of DNA methylation in amyotrophic lateral sclerosis: a new target of disease mechanisms.Mitochondrial permeability transition pore regulates Parkinson's disease development in mutant α-synuclein transgenic mice.Motor neurons rapidly accumulate DNA single-strand breaks after in vitro exposure to nitric oxide and peroxynitrite and in vivo axotomy.Mitochondrial proteomic analysis of a cell line model of familial amyotrophic lateral sclerosis.Sporadic amyotrophic lateral sclerosis: a hypothesis of persistent (non-lytic) enteroviral infection.Neuropathology of Amyotrophic Lateral Sclerosis and Its Variants.Vascular endothelial growth factor prevents G93A-SOD1-induced motor neuron degeneration.Skeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic miceOne universal common endpoint in mouse models of amyotrophic lateral sclerosis.Angiogenin inhibits nuclear translocation of apoptosis inducing factor in a Bcl-2-dependent manner.The crucial role of caspase-9 in the disease progression of a transgenic ALS mouse model.Preparation of the caspase-3/7 substrate Ac-DEVD-pNA by solution-phase peptide synthesisAngiogenin prevents serum withdrawal-induced apoptosis of P19 embryonal carcinoma cellsNeuroprotection by caspase inhibitors.Apoptosis in amyotrophic lateral sclerosis: a review of the evidence.Compensatory Motor Neuron Response to Chromatolysis in the Murine hSOD1(G93A) Model of Amyotrophic Lateral SclerosisMitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases.Cell death in the peripheral nervous system: potential rescue strategies.Emerging strategies for drug development in motor neuron disease.Melatonin inhibits the caspase-1/cytochrome c/caspase-3 cell death pathway, inhibits MT1 receptor loss and delays disease progression in a mouse model of amyotrophic lateral sclerosis.Review: transactive response DNA-binding protein 43 (TDP-43): mechanisms of neurodegeneration.GNX-4728, a novel small molecule drug inhibitor of mitochondrial permeability transition, is therapeutic in a mouse model of amyotrophic lateral sclerosis.The mitochondrial permeability transition pore regulates nitric oxide-mediated apoptosis of neurons induced by target deprivationProgrammed cell death in amyotrophic lateral sclerosisp53 and Cell Cycle Proteins Participate in Spinal Motor Neuron Cell Death in ALSTherapeutic developments in the treatment of amyotrophic lateral sclerosis.Peripheral neuropathy in the Twitcher mouse involves the activation of axonal caspase 3.Molecular and cellular pathways of neurodegeneration in motor neurone diseaseProtective and Toxic Neuroinflammation in Amyotrophic Lateral Sclerosis.Epigenetic regulation of motor neuron cell death through DNA methylation.Alterations in G(1) to S phase cell-cycle regulators during amyotrophic lateral sclerosis.Amyotrophic lateral sclerosis: a consensus viewpoint on designing and implementing a clinical trial.Amyotrophic lateral sclerosis: contemporary concepts in etiopathogenesis and pharmacotherapy.Wnt Signaling Alteration in the Spinal Cord of Amyotrophic Lateral Sclerosis Transgenic Mice: Special Focus on Frizzled-5 Cellular Expression PatternNuclear localization of human SOD1 and mutant SOD1-specific disruption of survival motor neuron protein complex in transgenic amyotrophic lateral sclerosis miceTruncation of the TAR DNA-binding protein 43 is not a prerequisite for cytoplasmic relocalization, and is suppressed by caspase inhibition and by introduction of the A90V sequence variantThe Many Virtues of tRNA-derived Stress-induced RNAs (tiRNAs): Discovering Novel Mechanisms of Stress Response and Effect on Human Health
P2860
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P2860
Neuronal death in amyotrophic lateral sclerosis is apoptosis: possible contribution of a programmed cell death mechanism.
description
1999 nî lūn-bûn
@nan
1999 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
name
Neuronal death in amyotrophic ...... ogrammed cell death mechanism.
@ast
Neuronal death in amyotrophic ...... ogrammed cell death mechanism.
@en
type
label
Neuronal death in amyotrophic ...... ogrammed cell death mechanism.
@ast
Neuronal death in amyotrophic ...... ogrammed cell death mechanism.
@en
prefLabel
Neuronal death in amyotrophic ...... ogrammed cell death mechanism.
@ast
Neuronal death in amyotrophic ...... ogrammed cell death mechanism.
@en
P1476
Neuronal death in amyotrophic ...... ogrammed cell death mechanism.
@en
P2093
P304
P356
10.1097/00005072-199905000-00005
P577
1999-05-01T00:00:00Z