Monocyte chemoattractant protein-1 deficiency protects against visceral fat-induced atherosclerosis.
about
Adipose inflammation initiates recruitment of leukocytes to mouse femoral artery: role of adipo-vascular axis in chronic inflammation.Metabolic syndrome and insulin resistance: underlying causes and modification by exercise training.P-selectin glycoprotein ligand-1 regulates adhesive properties of the endothelium and leukocyte trafficking into adipose tissueAtherosclerosis and leukocyte-endothelial adhesive interactions are increased following acute myocardial infarction in apolipoprotein E deficient mice.Epicardial and perivascular adipose tissues and their influence on cardiovascular disease: basic mechanisms and clinical associations.Leptin receptor-induced STAT3-independent signaling pathways are protective against atherosclerosis in a murine model of obesity and hyperlipidemiaTransfer of intracellular HIV Nef to endothelium causes endothelial dysfunction.Effects of intravenous glucose and lipids on innate immune cell activation in healthy, obese, and type 2 diabetic subjects.Perivascular visceral adipose tissue induces atherosclerosis in apolipoprotein E deficient mice.Cytokines in atherosclerosis: Key players in all stages of disease and promising therapeutic targets.A novel anti-inflammatory natural product from Sphaeranthus indicus inhibits expression of VCAM1 and ICAM1, and slows atherosclerosis progression independent of lipid changes.Cytokines: roles in atherosclerosis disease progression and potential therapeutic targetsAllergic lung inflammation promotes atherosclerosis in apolipoprotein E-deficient mice.Aging exacerbates obesity-induced oxidative stress and inflammation in perivascular adipose tissue in mice: a paracrine mechanism contributing to vascular redox dysregulation and inflammation.H-2g, a glucose analog of blood group H antigen, mediates monocyte recruitment in vitro and in vivo via IL-8/CXCL8.High phosphate diet reduces atherosclerosis formation in apolipoprotein E-deficient mice.Membrane-Tethered Metalloproteinase Expressed by Vascular Smooth Muscle Cells Limits the Progression of Proliferative Atherosclerotic Lesions.MCP-1, KC-like and IL-8 as critical mediators of pathogenesis caused by Babesia canis.Activation of the JAK/STAT3 and PI3K/AKT pathways are crucial for IL-6 trans-signaling-mediated pro-inflammatory response in human vascular endothelial cells
P2860
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P2860
Monocyte chemoattractant protein-1 deficiency protects against visceral fat-induced atherosclerosis.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մարտին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Monocyte chemoattractant prote ...... l fat-induced atherosclerosis.
@ast
Monocyte chemoattractant prote ...... l fat-induced atherosclerosis.
@en
type
label
Monocyte chemoattractant prote ...... l fat-induced atherosclerosis.
@ast
Monocyte chemoattractant prote ...... l fat-induced atherosclerosis.
@en
prefLabel
Monocyte chemoattractant prote ...... l fat-induced atherosclerosis.
@ast
Monocyte chemoattractant prote ...... l fat-induced atherosclerosis.
@en
P2093
P2860
P1476
Monocyte chemoattractant prote ...... al fat-induced atherosclerosis
@en
P2093
Andrew P Wright
Daniel T Eitzman
Hana M Russo
Kevin J Wickenheiser
P2860
P304
P356
10.1161/ATVBAHA.110.205914
P407
P577
2010-03-18T00:00:00Z