Differential regulation of immune responses by highly and weakly virulent Cryptococcus neoformans isolates
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Immunoregulation in Fungal DiseasesInvolvement of CD14, toll-like receptors 2 and 4, and MyD88 in the host response to the fungal pathogen Cryptococcus neoformans in vivoInsights into the mechanisms of protective immunity against Cryptococcus neoformans infection using a mouse model of pulmonary cryptococcosisEnhanced gamma interferon production through activation of Valpha14(+) natural killer T cells by alpha-galactosylceramide in interleukin-18-deficient mice with systemic cryptococcosis.Regulation of cytokine expression in mice immunized with cryptococcal polysaccharide, a glucuronoxylomannan (GXM), associated with peritoneal antigen-presenting cells (APC): requirements for GXM, APC activation, and interleukin-12Phenotypic switching of Cryptococcus neoformans can produce variants that elicit increased intracranial pressure in a rat model of cryptococcal meningoencephalitisCTLA-4 down-regulates the protective anticryptococcal cell-mediated immune responseInterdependency of interleukin-10 and interleukin-12 in regulation of T-cell differentiation and effector function of monocytes in response to stimulation with Cryptococcus neoformans.Both Th1 and Th2 cytokines affect the ability of monoclonal antibodies to protect mice against Cryptococcus neoformans.Dectin-2 deficiency promotes Th2 response and mucin production in the lungs after pulmonary infection with Cryptococcus neoformans.In pulmonary paracoccidioidomycosis IL-10 deficiency leads to increased immunity and regressive infection without enhancing tissue pathology.Urease expression by Cryptococcus neoformans promotes microvascular sequestration, thereby enhancing central nervous system invasion.A flucytosine-responsive Mbp1/Swi4-like protein, Mbs1, plays pleiotropic roles in antifungal drug resistance, stress response, and virulence of Cryptococcus neoformans.Phenotypic switching of Cryptococcus neoformans occurs in vivo and influences the outcome of infectionPhenotypic switching in Cryptococcus neoformans contributes to virulence by changing the immunological host responseThe phenotype of the Cryptococcus-specific CD4+ memory T-cell response is associated with disease severity and outcome in HIV-associated cryptococcal meningitis.Loss of allergen 1 confers a hypervirulent phenotype that resembles mucoid switch variants of Cryptococcus neoformansCryptococcal urease promotes the accumulation of immature dendritic cells and a non-protective T2 immune response within the lung.The capsule of the fungal pathogen Cryptococcus neoformans.Robust Th1 and Th17 immunity supports pulmonary clearance but cannot prevent systemic dissemination of highly virulent Cryptococcus neoformans H99Age-related resistance of C57BL/6 mice to Cryptococcus neoformans is dependent on maturation of NKT cells.Defect of CARD9 leads to impaired accumulation of gamma interferon-producing memory phenotype T cells in lungs and increased susceptibility to pulmonary infection with Cryptococcus neoformans.Cryptococcal therapies and drug targets: the old, the new and the promising.Activation of Valpha14(+) natural killer T cells by alpha-galactosylceramide results in development of Th1 response and local host resistance in mice infected with Cryptococcus neoformans.Regulatory effects of macrophage inflammatory protein 1alpha/CCL3 on the development of immunity to Cryptococcus neoformans depend on expression of early inflammatory cytokines.Antibody-mediated protection in murine Cryptococcus neoformans infection is associated with pleotrophic effects on cytokine and leukocyte responses.Analogous cytokine responses to Burkholderia pseudomallei strains contrasting in virulence correlate with partial cross-protection in immunized mice.Klebsiella pneumoniae increases the levels of Toll-like receptors 2 and 4 in human airway epithelial cells.Early or late IL-10 blockade enhances Th1 and Th17 effector responses and promotes fungal clearance in mice with cryptococcal lung infectionLower expression of Th1-related cytokines and inducible nitric oxide synthase in mice with streptozotocin-induced diabetes mellitus infected with Mycobacterium tuberculosis.Fungal Strategies to Evade the Host Immune Recognition.CpG oligodeoxynucleotides promote the host protective response against infection with Cryptococcus neoformans through induction of interferon-gamma production by CD4+ T cells.
P2860
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P2860
Differential regulation of immune responses by highly and weakly virulent Cryptococcus neoformans isolates
description
1999 nî lūn-bûn
@nan
1999 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
name
Differential regulation of imm ...... yptococcus neoformans isolates
@ast
Differential regulation of imm ...... yptococcus neoformans isolates
@en
type
label
Differential regulation of imm ...... yptococcus neoformans isolates
@ast
Differential regulation of imm ...... yptococcus neoformans isolates
@en
prefLabel
Differential regulation of imm ...... yptococcus neoformans isolates
@ast
Differential regulation of imm ...... yptococcus neoformans isolates
@en
P2093
P2860
P1476
Differential regulation of imm ...... yptococcus neoformans isolates
@en
P2093
A M Adesina
J W Murphy
K L Buchanan
R Blackstock
P2860
P304
P407
P577
1999-07-01T00:00:00Z