SOCS1 is a negative regulator of metabolic reprogramming during sepsis
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Glycine Relieves Intestinal Injury by Maintaining mTOR Signaling and Suppressing AMPK, TLR4, and NOD Signaling in Weaned Piglets after Lipopolysaccharide ChallengeInterplay between Cellular Metabolism and Cytokine Responses during Viral InfectionGlutamate alleviates intestinal injury, maintains mTOR and suppresses TLR4 and NOD signaling pathways in weanling pigs challenged with lipopolysaccharideMacrophage-derived LTB4 promotes abscess formation and clearance of Staphylococcus aureus skin infection in miceNetwork analysis of inflammatory responses to sepsis by neutrophils and peripheral blood mononuclear cells
P2860
SOCS1 is a negative regulator of metabolic reprogramming during sepsis
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2017 nî lūn-bûn
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2017 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2017 թվականի հուլիսին հրատարակված գիտական հոդված
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2017年の論文
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2017年論文
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2017年論文
@zh-hant
2017年論文
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2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
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SOCS1 is a negative regulator of metabolic reprogramming during sepsis
@ast
SOCS1 is a negative regulator of metabolic reprogramming during sepsis
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type
label
SOCS1 is a negative regulator of metabolic reprogramming during sepsis
@ast
SOCS1 is a negative regulator of metabolic reprogramming during sepsis
@en
prefLabel
SOCS1 is a negative regulator of metabolic reprogramming during sepsis
@ast
SOCS1 is a negative regulator of metabolic reprogramming during sepsis
@en
P2093
P2860
P1433
P1476
SOCS1 is a negative regulator of metabolic reprogramming during sepsis
@en
P2093
Annie Rocio Piñeros Alvarez
Brian Paul McCarthy
C Henrique Serezani
Jose Carlos Alves-Filho
Nicole Glosson-Byers
Paul R Territo
Sarah Sturgeon
Soujuan Wang
Stephanie Brandt
P2860
P356
10.1172/JCI.INSIGHT.92530
P577
2017-07-06T00:00:00Z