c-myc and c-fos expression in differentiating mouse primary keratinocytes.
about
Generalized substitution of isoencoding codons shortens the duration of papillomavirus L1 protein expression in transiently gene-transfected keratinocytes due to cell differentiationModulation of c-myc, max, and mad gene expression during neural differentiation of embryonic stem cells by all-trans-retinoic acid.Phorbol ester-induced terminal differentiation is inhibited in human U-937 monoblastic cells expressing a v-myc oncogene.Malignant transformation of mouse primary keratinocytes by Harvey sarcoma virus and its modulation by surrounding normal cells.The ras and myc oncogenes cooperate in tumor induction in many tissues when introduced into midgestation mouse embryos by retroviral vectors.Response modification in carcinogenesisDevelopmentally regulated alternative splicing in the Xenopus laevis c-Myc gene creates an intron-1 containing c-Myc RNA present only in post-midblastula embryos.Coordinate changes in gene expression which mark the spinous to granular cell transition in epidermis are regulated by protein kinase C.The human cut homeodomain protein represses transcription from the c-myc promoter.Cell cycle regulation of the c-Myc transcriptional activation domain.Tyrosine phosphorylation is an early and specific event involved in primary keratinocyte differentiationRetroviral transduction of the human c-Ha-ras-1 oncogene into midgestation mouse embryos promotes rapid epithelial hyperplasia.Role of PI3K-AKT-mTOR and Wnt Signaling Pathways in Transition of G1-S Phase of Cell Cycle in Cancer Cells.The calcium signal for BALB/MK keratinocyte terminal differentiation counteracts epidermal growth factor (EGF) very early in the EGF-induced proliferative pathway.An oligomer complementary to c-myc mRNA inhibits proliferation of HL-60 promyelocytic cells and induces differentiation.Microinjection of fos-specific antibodies blocks DNA synthesis in fibroblast cells.Selective inhibition of growth-related gene expression in murine keratinocytes by transforming growth factor betaConstitutive expression of growth-related mRNAs in proliferating and nonproliferating lung epithelial cells in primary culture: evidence for growth-dependent translational control.Two oncogenes, v-fos and v-ras, cooperate to convert normal keratinocytes to squamous cell carcinoma.The tumor promoter 12-O-tetradecanoylphorbol-13-acetate and the ras oncogene modulate expression and phosphorylation of gap junction proteins.Neither macromolecular synthesis nor myc is required for cell death via the mechanism that can be controlled by Bcl-2.Transformation of murine melanocytes by basic fibroblast growth factor cDNA and oncogenes and selective suppression of the transformed phenotype in a reconstituted cutaneous environmentExpression of the carcinoma-associated keratin K6 and the role of AP-1 proto-oncoproteins
P2860
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P2860
c-myc and c-fos expression in differentiating mouse primary keratinocytes.
description
1986 nî lūn-bûn
@nan
1986 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1986 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
name
c-myc and c-fos expression in differentiating mouse primary keratinocytes.
@ast
c-myc and c-fos expression in differentiating mouse primary keratinocytes.
@en
type
label
c-myc and c-fos expression in differentiating mouse primary keratinocytes.
@ast
c-myc and c-fos expression in differentiating mouse primary keratinocytes.
@en
prefLabel
c-myc and c-fos expression in differentiating mouse primary keratinocytes.
@ast
c-myc and c-fos expression in differentiating mouse primary keratinocytes.
@en
P2093
P2860
P1433
P1476
c-myc and c-fos expression in differentiating mouse primary keratinocytes
@en
P2093
M Maruyama
M Z Gilman
R A Weinberg
P2860
P304
P356
10.1002/J.1460-2075.1986.TB04579.X
P407
P577
1986-11-01T00:00:00Z