Mamld1 knockdown reduces testosterone production and Cyp17a1 expression in mouse Leydig tumor cells - sprookjes - yvandenbroek - TriplyDB

Mamld1 knockdown reduces testosterone production and Cyp17a1 expression in mouse Leydig tumor cells

Mamld1 knockdown reduces testosterone production and Cyp17a1 expression in mouse Leydig tumor cells

http://www.wikidata.org/entity/Q33895164

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Mamld1 deficiency significantly reduces mRNA expression levels of multiple genes expressed in mouse fetal Leydig cells but permits normal genital and reproductive development Knockout of Murine Mamld1 Impairs Testicular Growth and Daily Sperm Production but Permits Normal Postnatal Androgen Production and Fertility.Disorders of sex development: new genes, new concepts.Human MAMLD1 Gene Variations Seem Not Sufficient to Explain a 46,XY DSD Phenotype.Parturition failure in mice lacking Mamld1.The Genetic and Environmental Factors Underlying Hypospadias.Notch Signaling Regulates Differentiation and Steroidogenesis in Female Mouse Ovarian Granulosa Cells.

P2860

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Mamld1 knockdown reduces testosterone production and Cyp17a1 expression in mouse Leydig tumor cells

http://www.wikidata.org/entity/Q33895164

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2011 nî lūn-bûn

@nan

2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年学术文章

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2011年学术文章

@zh-cn

2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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Mamld1 knockdown reduces testo ...... on in mouse Leydig tumor cells

@ast

Mamld1 knockdown reduces testo ...... on in mouse Leydig tumor cells

@en

type

label

Mamld1 knockdown reduces testo ...... on in mouse Leydig tumor cells

@ast

Mamld1 knockdown reduces testo ...... on in mouse Leydig tumor cells

@en

prefLabel

Mamld1 knockdown reduces testo ...... on in mouse Leydig tumor cells

@ast

Mamld1 knockdown reduces testo ...... on in mouse Leydig tumor cells

@en

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JOURNAL.PONE.0019123

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Mamld1 knockdown reduces testo ...... on in mouse Leydig tumor cells

@en

P2093

Fumihiro Sugawa

http://www.w3.org/2001/XMLSchema#string

Katsuya Nonomura

http://www.w3.org/2001/XMLSchema#string

Maki Fukami

http://www.w3.org/2001/XMLSchema#string

Mami Miyado

http://www.w3.org/2001/XMLSchema#string

Michiko Nakamura

http://www.w3.org/2001/XMLSchema#string

P2860

Integrative genomic analyses on HES/HEY family: Notch-independent HES1, HES3 transcription in undifferentiated ES cells, and Notch-dependent HES1, HES5, HEY1, HEY2, HEYL transcription in fetal tissues, adult tissues, or cancer

Cryptorchidism in mice mutant for Insl3

The Hes gene family: repressors and oscillators that orchestrate embryogenesis

Mastermind-like domain-containing 1 (MAMLD1 or CXorf6) transactivates the Hes3 promoter, augments testosterone production, and contains the SF1 target sequence.

Gonadotropin binding and stimulation of steroidogenesis in Leydig tumor cells.

Notch signaling in development and cancer.

Use of an aqueous soluble tetrazolium/formazan assay for cell growth assays in culture.

Establishment of gonadotropin-responsive murine leydig tumor cell line.

Notch signaling maintains Leydig progenitor cells in the mouse testis

Steroidogenic factor-1 (SF-1, Ad4BP, NR5A1) and disorders of testis development.

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e19123

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scholarly article

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4

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51114533

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21559465

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3084764

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