Rapid in vivo reactivation of herpes simplex virus in latently infected murine ganglionic neurons after transient hyperthermia.
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The probability of in vivo reactivation of herpes simplex virus type 1 increases with the number of latently infected neurons in the ganglia.Spontaneous reactivation of herpes simplex virus type 1 in latently infected murine sensory gangliaEvidence that spontaneous reactivation of herpes virus does not occur in miceA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyA cultured affair: HSV latency and reactivation in neuronsOcular herpes simplex virus: how are latency, reactivation, recurrent disease and therapy interrelated?The challenges and opportunities for the development of a T-cell epitope-based herpes simplex vaccineVisualization of mouse neuronal ganglia infected by Herpes Simplex Virus 1 (HSV-1) using multimodal non-linear optical microscopyHerpes simplex virus and the lexicon of latency and reactivation: a call for defining terms and building an integrated collective frameworkHerpesviruses: latency and reactivation - viral strategies and host responseThe effects of dexamethasone and acyclovir on a cell culture model of delayed facial palsy.Neuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation.Valacyclovir for the prevention of recurrent herpes simplex virus eye disease after excimer laser photokeratectomy.De novo synthesis of VP16 coordinates the exit from HSV latency in vivo.Role of nuclear factor Y in stress-induced activation of the herpes simplex virus type 1 ICP0 promoterFrequent release of low amounts of herpes simplex virus from neurons: results of a mathematical model.Role for gamma interferon in control of herpes simplex virus type 1 reactivation.Reactivation of herpes simplex virus type 1 in the mouse trigeminal ganglion: an in vivo study of virus antigen and cytokines.Interferon-beta suppresses herpes simplex virus type 1 replication in trigeminal ganglion cells through an RNase L-dependent pathwayThe latent herpes simplex virus type 1 genome copy number in individual neurons is virus strain specific and correlates with reactivationReplication of herpes simplex virus type 1 within trigeminal ganglia is required for high frequency but not high viral genome copy number latency.Optimized viral dose and transient immunosuppression enable herpes simplex virus ICP0-null mutants To establish wild-type levels of latency in vivo.Lack of interleukin-6 (IL-6) enhances susceptibility to infection but does not alter latency or reactivation of herpes simplex virus type 1 in IL-6 knockout miceICP0, ICP4, or VP16 expressed from adenovirus vectors induces reactivation of latent herpes simplex virus type 1 in primary cultures of latently infected trigeminal ganglion cells.Modeling HSV-1 Latency in Human Embryonic Stem Cell-Derived Neurons.Tracking the spread of a lacZ-tagged herpes simplex virus type 1 between the eye and the nervous system of the mouse: comparison of primary and recurrent infectionGender influences herpes simplex virus type 1 infection in normal and gamma interferon-mutant miceRegulation of herpes simplex virus type 1 thymidine kinase gene expression by thyroid hormone receptor in cultured neuronal cells.Spontaneous molecular reactivation of herpes simplex virus type 1 latency in mice.Gamma interferon can block herpes simplex virus type 1 reactivation from latency, even in the presence of late gene expressionHerpes simplex virus type 1 latency-associated transcription unit promotes anatomical site-dependent establishment and reactivation from latency.Persistence of infectious herpes simplex virus type 2 in the nervous system in mice after antiviral chemotherapyDelaying the expression of herpes simplex virus type 1 glycoprotein B (gB) to a true late gene alters neurovirulence and inhibits the gB-CD8+ T-cell response in the trigeminal ganglionUse of differential display reverse transcription-PCR to reveal cellular changes during stimuli that result in herpes simplex virus type 1 reactivation from latency: upregulation of immediate-early cellular response genes TIS7, interferon, and interHerpes simplex virus DNA synthesis is not a decisive regulatory event in the initiation of lytic viral protein expression in neurons in vivo during primary infection or reactivation from latency.In vivo reactivation of latent herpes simplex virus 1 in mice can occur in the brain before occurring in the trigeminal ganglionHistone deacetylase inhibitors induce reactivation of herpes simplex virus type 1 in a latency-associated transcript-independent manner in neuronal cellsHematogenous vertical transmission of herpes simplex virus type 1 in miceICP0 is not required for efficient stress-induced reactivation of herpes simplex virus type 1 from cultured quiescently infected neuronal cells.
P2860
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P2860
Rapid in vivo reactivation of herpes simplex virus in latently infected murine ganglionic neurons after transient hyperthermia.
description
1992 nî lūn-bûn
@nan
1992 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Rapid in vivo reactivation of ...... after transient hyperthermia.
@ast
Rapid in vivo reactivation of ...... after transient hyperthermia.
@en
type
label
Rapid in vivo reactivation of ...... after transient hyperthermia.
@ast
Rapid in vivo reactivation of ...... after transient hyperthermia.
@en
prefLabel
Rapid in vivo reactivation of ...... after transient hyperthermia.
@ast
Rapid in vivo reactivation of ...... after transient hyperthermia.
@en
P2860
P1433
P1476
Rapid in vivo reactivation of ...... after transient hyperthermia.
@en
P2093
N M Sawtell
R L Thompson
P2860
P304
P407
P577
1992-04-01T00:00:00Z