Activation of HIV-1 pre-mRNA 3' processing in vitro requires both an upstream element and TAR.
about
Overlapping enhancer/promoter and transcriptional termination signals in the lentiviral long terminal repeatThe splicing factor-associated protein, p32, regulates RNA splicing by inhibiting ASF/SF2 RNA binding and phosphorylationParticipation of the nuclear cap binding complex in pre-mRNA 3' processingTat-dependent occlusion of the HIV poly(A) siteComparison of 5' and 3' long terminal repeat promoter function in human immunodeficiency virusAlternative polyadenylation of cyclooxygenase-2.REF2 encodes an RNA-binding protein directly involved in yeast mRNA 3'-end formation.Formation of mRNA 3' ends in eukaryotes: mechanism, regulation, and interrelationships with other steps in mRNA synthesisThe poly(A)-limiting element is a conserved cis-acting sequence that regulates poly(A) tail length on nuclear pre-mRNAsA downstream polyadenylation element in human papillomavirus type 16 L2 encodes multiple GGG motifs and interacts with hnRNP H.Inhibition of HIV-1 replication by eIF3f.Development of a self-inactivating lentivirus vector.Tissue-specific transcriptional targeting of a replication-competent retroviral vectorPosition-dependent inhibition of the cleavage step of pre-mRNA 3'-end processing by U1 snRNPCPSF30 and Wdr33 directly bind to AAUAAA in mammalian mRNA 3' processingForced evolution of a regulatory RNA helix in the HIV-1 genomeUpstream and downstream cis-acting elements for cleavage at the L4 polyadenylation site of adenovirus-2.RNA polymerase II kinetics in polo polyadenylation signal selection.The upstream sequence element of the C2 complement poly(A) signal activates mRNA 3' end formation by two distinct mechanisms.Juxtaposition of two distant, serine-arginine-rich protein-binding elements is required for optimal polyadenylation in Rous sarcoma virus.A common mechanism for the enhancement of mRNA 3' processing by U3 sequences in two distantly related lentiviruses.Sequences regulating poly(A) site selection within the adenovirus major late transcription unit influence the interaction of constitutive processing factors with the pre-mRNA.A conserved hairpin motif in the R-U5 region of the human immunodeficiency virus type 1 RNA genome is essential for replication3'-end-forming signals of yeast mRNA.RNA structure is a critical determinant of poly(A) site recognition by cleavage and polyadenylation specificity factor.Promoter-proximal poly(A) sites are processed efficiently, but the RNA products are unstable in the nucleus.Sequences homologous to 5' splice sites are required for the inhibitory activity of papillomavirus late 3' untranslated regions.Protein factors in pre-mRNA 3'-end processing.Poly(A) site selection in the yeast Ty retroelement requires an upstream region and sequence-specific titratable factor(s) in vitro.Assembly of the cleavage and polyadenylation apparatus requires about 10 seconds in vivo and is faster for strong than for weak poly(A) sitesU1snRNP-mediated suppression of polyadenylation in conjunction with the RNA structure controls poly (A) site selection in foamy viruses.Recruitment of a basal polyadenylation factor by the upstream sequence element of the human lamin B2 polyadenylation signal.Functionally significant secondary structure of the simian virus 40 late polyadenylation signal.A hairpin structure in the R region of the human immunodeficiency virus type 1 RNA genome is instrumental in polyadenylation site selection.The retroviruses human immunodeficiency virus type 1 and Moloney murine leukemia virus adopt radically different strategies to regulate promoter-proximal polyadenylation.Transcription and polyadenylation in a short human intergenic region.Inhibition of polyadenylation by stable RNA secondary structure.Upstream sequence elements enhance poly(A) site efficiency of the C2 complement gene and are phylogenetically conserved.Polypyrimidine tract binding protein modulates efficiency of polyadenylation.RNA motif discovery by SHAPE and mutational profiling (SHAPE-MaP).
P2860
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P2860
Activation of HIV-1 pre-mRNA 3' processing in vitro requires both an upstream element and TAR.
description
1992 nî lūn-bûn
@nan
1992 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
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1992年学术文章
@wuu
1992年学术文章
@zh-cn
1992年学术文章
@zh-hans
1992年学术文章
@zh-my
1992年学术文章
@zh-sg
1992年學術文章
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name
Activation of HIV-1 pre-mRNA 3 ...... h an upstream element and TAR.
@ast
Activation of HIV-1 pre-mRNA 3 ...... h an upstream element and TAR.
@en
type
label
Activation of HIV-1 pre-mRNA 3 ...... h an upstream element and TAR.
@ast
Activation of HIV-1 pre-mRNA 3 ...... h an upstream element and TAR.
@en
prefLabel
Activation of HIV-1 pre-mRNA 3 ...... h an upstream element and TAR.
@ast
Activation of HIV-1 pre-mRNA 3 ...... h an upstream element and TAR.
@en
P2093
P2860
P1433
P1476
Activation of HIV-1 pre-mRNA 3 ...... h an upstream element and TAR.
@en
P2093
E S Fleming
G M Gilmartin
P2860
P304
P407
P577
1992-12-01T00:00:00Z