Epistasis between mouse Klra and major histocompatibility complex class I loci is associated with a new mechanism of natural killer cell-mediated innate resistance to cytomegalovirus infection.
about
Regulation of TLR7/9 responses in plasmacytoid dendritic cells by BST2 and ILT7 receptor interactionComparative genomics of natural killer cell receptor gene clusters.Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experienceGenomic Modifiers of Natural Killer Cells, Immune Responsiveness and Lymphoid Tissue Remodeling Together Increase Host Resistance to Viral InfectionNK cell receptor/H2-Dk-dependent host resistance to viral infection is quantitatively modulated by H2q inhibitory signalsInteraction of NK inhibitory receptor genes with HLA-C and MHC class II alleles in Hepatitis C virus infection outcomeMolecular Architecture of the Major Histocompatibility Complex Class I-binding Site of Ly49 Natural Killer Cell ReceptorsGenome-Wide Exome Analysis of Cmv5-Disparate Mouse Strains that Differ in Host Resistance to Murine Cytomegalovirus Infection.Multiparametric analysis of host response to murine cytomegalovirus in MHC class I-disparate mice reveals primacy of Dk-licensed Ly49G2+ NK cells in viral control.Know Thyself: NK-Cell Inhibitory Receptors Prompt Self-Tolerance, Education, and Viral Control.Rapid discrimination of MHC class I and killer cell lectin-like receptor allele variants by high-resolution melt analysis.Self MHC class I-licensed NK cells enhance adaptive CD8 T-cell viral immunity.NK gene complex and chromosome 19 loci enhance MHC resistance to murine cytomegalovirus infectionMHC class I D(k) expression in hematopoietic and nonhematopoietic cells confers natural killer cell resistance to murine cytomegalovirusMHC class I Dk locus and Ly49G2+ NK cells confer H-2k resistance to murine cytomegalovirusNK gene complex dynamics and selection for NK cell receptors.Deficient major histocompatibility complex-linked innate murine cytomegalovirus immunity in MA/My.L-H2b mice and viral downregulation of H-2k class I proteins.Association of killer cell immunoglobulin-like receptor genes with Hodgkin's lymphoma in a familial study.Genetic control of variegated KIR gene expression: polymorphisms of the bi-directional KIR3DL1 promoter are associated with distinct frequencies of gene expression.Recognition of peptide-MHC class I complexes by activating killer immunoglobulin-like receptorsSignaling and ligand interaction of ILT7: receptor-mediated regulatory mechanisms for plasmacytoid dendritic cellsThe protozoan Neospora caninum directly triggers bovine NK cells to produce gamma interferon and to kill infected fibroblastsCytomegalovirus immunoevasin reveals the physiological role of "missing self" recognition in natural killer cell dependent virus control in vivo.A novel primary immunodeficiency with specific natural-killer cell deficiency maps to the centromeric region of chromosome 8.NK cells and their ability to modulate T cells during virus infectionsThe genetics of infectious disease susceptibility: has the evidence for epistasis been overestimated?Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains.How the virus outsmarts the host: function and structure of cytomegalovirus MHC-I-like molecules in the evasion of natural killer cell surveillanceMultigenic control and sex bias in host susceptibility to spore-induced pulmonary anthrax in miceIdentification of Mouse Cytomegalovirus Resistance Loci by ENU Mutagenesis.KIR-HLA intercourse in HIV diseaseStructural basis for recognition of MHC and MHC-like ligands by natural killer cell receptors.Natural killer cells in viral infection: more than just killers.Characterization of murine cytomegalovirus m157 from infected cells and identification of critical residues mediating recognition by the NK cell receptor Ly49H.Evolutionary struggles between NK cells and viruses.Positive regulation of plasmacytoid dendritic cell function via Ly49Q recognition of class I MHC.HLA/KIR restraint of HIV: surviving the fittestImmune evasion of natural killer cells by viruses.Antiviral NK cell responses in HIV infection: I. NK cell receptor genes as determinants of HIV resistance and progression to AIDS.Natural killer cells in immunodefense against infective agents.
P2860
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P2860
Epistasis between mouse Klra and major histocompatibility complex class I loci is associated with a new mechanism of natural killer cell-mediated innate resistance to cytomegalovirus infection.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Epistasis between mouse Klra a ...... to cytomegalovirus infection.
@ast
Epistasis between mouse Klra a ...... to cytomegalovirus infection.
@en
type
label
Epistasis between mouse Klra a ...... to cytomegalovirus infection.
@ast
Epistasis between mouse Klra a ...... to cytomegalovirus infection.
@en
prefLabel
Epistasis between mouse Klra a ...... to cytomegalovirus infection.
@ast
Epistasis between mouse Klra a ...... to cytomegalovirus infection.
@en
P2093
P2860
P356
P1433
P1476
Epistasis between mouse Klra a ...... to cytomegalovirus infection.
@en
P2093
Agnieszka Kielczewska
Andrew P Makrigiannis
J-C Loredo-Osti
Lewis L Lanier
Marie-Pierre Desrosiers
Melissa B Lodoen
Silvia M Vidal
Sonia Girard Adam
Suzanne Lemieux
Trung Pham
P2860
P2888
P304
P356
10.1038/NG1564
P407
P577
2005-05-15T00:00:00Z