p38 mitogen-activated protein kinase activates eNOS in endothelial cells by an estrogen receptor alpha-dependent pathway in response to black tea polyphenols.
about
Increased responsiveness of human coronary artery endothelial cells in inflammation and coagulationPolyphenol fraction of extra virgin olive oil protects against endothelial dysfunction induced by high glucose and free fatty acids through modulation of nitric oxide and endothelin-1.Tea and cardiovascular disease.Activation of insulin-like growth factor I receptor-mediated pathway by ginsenoside Rg1.Species Comparison of the Role of p38 MAP Kinase in the Female Reproductive System.Interleukin-17A Promotes Aortic Endothelial Cell Activation via Transcriptionally and Post-translationally Activating p38 Mitogen-activated Protein Kinase (MAPK) Pathway.Acute erythropoietin cardioprotection is mediated by endothelial response.Effects of flavonoid-containing beverages and EGCG on endothelial functionp38 MAPK activity is stimulated by vascular endothelial growth factor receptor 2 activation and is essential for shear stress-induced angiogenesisBenefit of Monascus-fermented products for hypertension prevention: a review.Antidiabetic Effects of Tea.A novel vasculo-angiogenic effect of cilostazol mediated by cross-talk between multiple signalling pathways including the ERK/p38 MAPK signalling transduction cascade.p38 MAPK-dependent eNOS upregulation is critical for 17beta-estradiol-mediated cardioprotection following trauma-hemorrhage.Identification of a PKCepsilon-dependent regulation of myocardial contraction by epicatechin-3-gallate.The efficacy of black tea in ameliorating endothelial function is equivalent to that of green tea.Venthamarai chooranam, a polyherbal Siddha medicine, alleviates hypertension via AT₁R and eNOS signaling pathway in 2K1C hypertensive rats.Epigallocatechin gallate, a green tea polyphenol, mediates NO-dependent vasodilation using signaling pathways in vascular endothelium requiring reactive oxygen species and Fyn.EGCG, a green tea polyphenol, improves endothelial function and insulin sensitivity, reduces blood pressure, and protects against myocardial I/R injury in SHR.Enhanced estradiol-induced vasorelaxation in aortas from type 2 diabetic mice may reflect a compensatory role of p38 MAPK-mediated eNOS activation.Nitric oxide and p38 MAP kinase mediate shear stress-dependent inhibition of MMP-2 production in microvascular endothelial cells.
P2860
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P2860
p38 mitogen-activated protein kinase activates eNOS in endothelial cells by an estrogen receptor alpha-dependent pathway in response to black tea polyphenols.
description
2005 nî lūn-bûn
@nan
2005 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
p38 mitogen-activated protein ...... onse to black tea polyphenols.
@ast
p38 mitogen-activated protein ...... onse to black tea polyphenols.
@en
type
label
p38 mitogen-activated protein ...... onse to black tea polyphenols.
@ast
p38 mitogen-activated protein ...... onse to black tea polyphenols.
@en
prefLabel
p38 mitogen-activated protein ...... onse to black tea polyphenols.
@ast
p38 mitogen-activated protein ...... onse to black tea polyphenols.
@en
P2093
P2860
P1433
P1476
p38 mitogen-activated protein ...... ponse to black tea polyphenols
@en
P2093
Elad Anter
Oz M Shapira
Richard H Karas
P2860
P304
P356
10.1161/01.RES.0000168807.63013.56
P50
P577
2005-05-05T00:00:00Z