Increased infarct wall thickness by a bio-inert material is insufficient to prevent negative left ventricular remodeling after myocardial infarction. - sprookjes - yvandenbroek - TriplyDB

Increased infarct wall thickness by a bio-inert material is insufficient to prevent negative left ventricular remodeling after myocardial infarction.

Increased infarct wall thickness by a bio-inert material is insufficient to prevent negative left ventricular remodeling after myocardial infarction.

http://www.wikidata.org/entity/Q33952558

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Physiological Implications of Myocardial Scar Structure Why Is Infarct Expansion Such an Elusive Therapeutic Target?Intramyocardial injection of a synthetic hydrogel with delivery of bFGF and IGF1 in a rat model of ischemic cardiomyopathy Discrete microstructural cues for the attenuation of fibrosis following myocardial infarction.Targeted injection of a biocomposite material alters macrophage and fibroblast phenotype and function following myocardial infarction: relation to left ventricular remodeling Intramyocardial delivery of mesenchymal stem cell-seeded hydrogel preserves cardiac function and attenuates ventricular remodeling after myocardial infarction Tailoring the degradation rates of thermally responsive hydrogels designed for soft tissue injection by varying the autocatalytic potential.Epicardial placement of mesenchymal stromal cell-sheets for the treatment of ischemic cardiomyopathy; in vivo proof-of-concept study.Catheter-deliverable hydrogel derived from decellularized ventricular extracellular matrix increases endogenous cardiomyocytes and preserves cardiac function post-myocardial infarction.Postinfarction Functional Recovery Driven by a Three-Dimensional Engineered Fibrin Patch Composed of Human Umbilical Cord Blood-Derived Mesenchymal Stem Cells Post-infarct biomaterials, left ventricular remodeling, and heart failure: is good good enough?Dual delivery of hepatocyte and vascular endothelial growth factors via a protease-degradable hydrogel improves cardiac function in rats.Timing effect of intramyocardial hydrogel injection for positively impacting left ventricular remodeling after myocardial infarction Intramyocardial injection of hydrogel with high interstitial spread does not impact action potential propagation Localized targeting of biomaterials following myocardial infarction: a foundation to build on Modulating In Vivo Degradation Rate of Injectable Extracellular Matrix Hydrogels Oxime cross-linked injectable hydrogels for catheter delivery.Injectable hydrogel therapies and their delivery strategies for treating myocardial infarction.3D-culture system for heart regeneration and cardiac medicine.Biomaterials in myocardial tissue engineering.Developing injectable nanomaterials to repair the heart Ventricular wall biomaterial injection therapy after myocardial infarction: Advances in material design, mechanistic insight and early clinical experiences.Biomaterial-Enhanced Cell and Drug Delivery: Lessons Learned in the Cardiac Field and Future Perspectives.Effect of bone marrow-derived extracellular matrix on cardiac function after ischemic injury Novel functionalization of discrete polymeric biomaterial microstructures for applications in imaging and three-dimensional manipulation.Intramyocardial injection of heart tissue-derived extracellular matrix improves postinfarction cardiac function in rats.Designing Acellular Injectable Biomaterial Therapeutics for Treating Myocardial Infarction and Peripheral Artery Disease.Immunohistochemical detection of differentially localized up-regulation of lysyl oxidase and down-regulation of matrix metalloproteinase-1 in rhesus monkey model of chronic myocardial infarction.An in vitro investigation to assess procedure parameters for injecting therapeutic hydrogels into the myocardium.Gelatin Microspheres as Vehicle for Cardiac Progenitor Cells Delivery to the Myocardium.Cardiac Regeneration with Human Pluripotent Stem Cell-Derived Cardiomyocytes A Thin Layer of Decellularized Porcine Myocardium for Cell Delivery

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P2860

Increased infarct wall thickness by a bio-inert material is insufficient to prevent negative left ventricular remodeling after myocardial infarction.

http://www.wikidata.org/entity/Q33952558

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2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011年の論文

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Increased infarct wall thickne ...... g after myocardial infarction.

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Increased infarct wall thickne ...... g after myocardial infarction.

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Aboli A Rane

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Amul Shah

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Diane P Hu

http://www.w3.org/2001/XMLSchema#string

Jeffrey H Omens

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Joyce S Chuang

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Karen L Christman

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Kirk L Peterson

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Nancy D Dalton

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Yusu Gu

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P2860

Biomaterials for the Treatment of Myocardial Infarction

Injectable self-assembling peptide nanofibers create intramyocardial microenvironments for endothelial cells

Morphologic study of small intestinal submucosa as a body wall repair device

Regulation of collagen degradation in the rat myocardium after infarction

Synthesis, characterization and therapeutic efficacy of a biodegradable, thermoresponsive hydrogel designed for application in chronic infarcted myocardium

Targeted in vivo extracellular matrix formation promotes neovascularization in a rodent model of myocardial infarction

Left ventricular remodeling after myocardial infarction: pathophysiology and therapy.

Injectable hydrogel properties influence infarct expansion and extent of postinfarction left ventricular remodeling in an ovine model

Identification of the alphavbeta3 integrin-interacting motif of betaig-h3 and its anti-angiogenic effect.

Controlled delivery of PDGF-BB for myocardial protection using injectable self-assembling peptide nanofibers.

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e21571

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10.1371/JOURNAL.PONE.0021571

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6

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21731777

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3121880

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