Stages of restricted HIV-1 infection in astrocyte cultures derived from human fetal brain tissue.
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Role of microglia in central nervous system infectionsHIV-1, methamphetamine and astrocytes at neuroinflammatory CrossroadsMultiple Protein Kinases via Activation of Transcription Factors NF-κB, AP-1 and C/EBP-δ Regulate the IL-6/IL-8 Production by HIV-1 Vpr in AstrocytesSTAT3 and its phosphorylation are involved in HIV-1 Tat-induced transactivation of glial fibrillary acidic proteinGenetic composition of human immunodeficiency virus type 1 in cerebrospinal fluid and blood without treatment and during failing antiretroviral therapy.Genetic variation and HIV-associated neurologic disease.In vitro activation of feline immunodeficiency virus in ramified microglial cells from asymptomatically infected cats.Molecular programming of endothelin-1 in HIV-infected brain: role of Tat in up-regulation of ET-1 and its inhibition by statinsSterile alpha motif and histidine/aspartic acid domain-containing protein 1 (SAMHD1)-facilitated HIV restriction in astrocytes is regulated by miRNA-181a.Interactions of HIV and drugs of abuse: the importance of glia, neural progenitors, and host genetic factors.HIV-1 entry and trans-infection of astrocytes involves CD81 vesicles.MEK1/2 inhibitors block basal and transforming growth factor 1beta1-stimulated JC virus multiplication.Fluorescently-labeled RNA packaging into HIV-1 particles: Direct examination of infectivity across central nervous system cell types.Cell-cell contact viral transfer contributes to HIV infection and persistence in astrocytesDNA-binding transcription factor NF-1A negatively regulates JC virus multiplication.HIV-1 Tat-shortened neurite outgrowth through regulation of microRNA-132 and its target gene expression.Astrocytic expression of HIV-1 viral protein R in the hippocampus causes chromatolysis, synaptic loss and memory impairment.Chronic low-level expression of HIV-1 Tat promotes a neurodegenerative phenotype with aging.HIV-1 Tat Induces Unfolded Protein Response and Endoplasmic Reticulum Stress in Astrocytes and Causes Neurotoxicity through Glial Fibrillary Acidic Protein (GFAP) Activation and Aggregation.Astrocytic expression of HIV-1 Nef impairs spatial and recognition memory.HIV-1 infection of microglial cells in a reconstituted humanized mouse model and identification of compounds that selectively reverse HIV latency.
P2860
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P2860
Stages of restricted HIV-1 infection in astrocyte cultures derived from human fetal brain tissue.
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2000 nî lūn-bûn
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2000 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2000 թվականի մայիսին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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Stages of restricted HIV-1 inf ...... from human fetal brain tissue.
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Stages of restricted HIV-1 inf ...... from human fetal brain tissue.
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type
label
Stages of restricted HIV-1 inf ...... from human fetal brain tissue.
@ast
Stages of restricted HIV-1 inf ...... from human fetal brain tissue.
@en
prefLabel
Stages of restricted HIV-1 inf ...... from human fetal brain tissue.
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Stages of restricted HIV-1 inf ...... from human fetal brain tissue.
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P1476
Stages of restricted HIV-1 inf ...... from human fetal brain tissue.
@en
P2093
P478
P577
2000-05-01T00:00:00Z