Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
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The crystal structures of theSalmonellatype III secretion system tip protein SipD in complex with deoxycholate and chenodeoxycholateCrystal Structure of PrgI-SipD: Insight into a Secretion Competent State of the Type Three Secretion System Needle Tip and its Interaction with Host LigandsIdentification of the bile salt binding site on IpaD from Shigella flexneri and the influence of ligand binding on IpaD structure.The Structure and Function of Type III Secretion SystemsBacterial genome instabilityBile salt receptor complex activates a pathogenic type III secretion systemRespiratory pathogens adopt a chronic lifestyle in response to bile.The Escherichia coli BarA-UvrY two-component system is a virulence determinant in the urinary tract.Genome-wide screen of Salmonella genes expressed during infection in pigs, using in vivo expression technologyBile acid-induced virulence gene expression of Vibrio parahaemolyticus reveals a novel therapeutic potential for bile acid sequestrants.Contribution of three bile-associated loci, bsh, pva, and btlB, to gastrointestinal persistence and bile tolerance of Listeria monocytogenes.Signal transduction pathway mediated by the novel regulator LoiA for low oxygen tension induced Salmonella Typhimurium invasion.vttRA and vttRB Encode ToxR family proteins that mediate bile-induced expression of type three secretion system genes in a non-O1/non-O139 Vibrio cholerae strain.Bile acids and bicarbonate inversely regulate intracellular cyclic di-GMP in Vibrio cholerae.Fur activates the expression of Salmonella enterica pathogenicity island 1 by directly interacting with the hilD operator in vivo and in vitroComplex regulatory network encompassing the Csr, c-di-GMP and motility systems of Salmonella Typhimurium.Genetic and functional characterization of the Escherichia coli BarA-UvrY two-component system: point mutations in the HAMP linker of the BarA sensor give a dominant-negative phenotype.Adaptation and preadaptation of Salmonella enterica to Bile.BarA-UvrY two-component system regulates virulence of uropathogenic E. coli CFT073Identification of immunogenic Salmonella enterica serotype Typhi antigens expressed in chronic biliary carriers of S. Typhi in Kathmandu, Nepal.Bile-induced DNA damage in Salmonella enterica.Functional genomic analysis of bile salt resistance in Enterococcus faeciumConversion of commensal Escherichia coli K-12 to an invasive form via expression of a mutant histone-like protein.Chronic and acute infection of the gall bladder by Salmonella Typhi: understanding the carrier stateMlc regulation of Salmonella pathogenicity island I gene expression via hilE repression.Analysis of global transcriptional profiles of enterotoxigenic Escherichia coli isolate E24377A.Repression of Salmonella enterica phoP expression by small molecules from physiological bile.Intestinal GPS: bile and bicarbonate control cyclic di-GMP to provide Vibrio cholerae spatial cues within the small intestine.Salmonella stress management and its relevance to behaviour during intestinal colonisation and infection.The interaction between bacteria and bile.Formate acts as a diffusible signal to induce Salmonella invasionModulation of the enterohemorrhagic E. coli virulence program through the human gastrointestinal tract.Structure and biophysics of type III secretion in bacteriaGallbladder epithelium as a niche for chronic Salmonella carriage.Salmonella enterica serovar typhimurium strains with regulated delayed attenuation in vivo.Energy Taxis toward Host-Derived Nitrate Supports a Salmonella Pathogenicity Island 1-Independent Mechanism of Invasion.New technologies in using recombinant attenuated Salmonella vaccine vectors.The role of type III secretion system 2 in Vibrio parahaemolyticus pathogenicity.A sophisticated multi-step secretion mechanism: how the type 3 secretion system is regulated.Fine tuning of virulence regulatory pathways in enteric bacteria in response to varying bile and oxygen concentrations in the gastrointestinal tract.
P2860
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P2860
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
description
2000 nî lūn-bûn
@nan
2000 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@ast
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@en
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@nl
type
label
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@ast
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@en
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@nl
prefLabel
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@ast
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@en
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@nl
P2860
P1476
Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.
@en
P2093
P2860
P304
P356
10.1128/IAI.68.12.6763-6769.2000
P407
P577
2000-12-01T00:00:00Z