S-nitrosohemoglobin deficiency: a mechanism for loss of physiological activity in banked blood. - sprookjes - yvandenbroek - TriplyDB

S-nitrosohemoglobin deficiency: a mechanism for loss of physiological activity in banked blood.

S-nitrosohemoglobin deficiency: a mechanism for loss of physiological activity in banked blood.

http://www.wikidata.org/entity/Q34007606

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Hypoxic Vasodilation by Red Blood Cells: Evidence for an S-Nitrosothiol-Based Signal Decreased erythrocyte deformability after transfusion and the effects of erythrocyte storage duration Endogenous S-nitrosothiols protect against myocardial injury Relationship between red cell storage duration and outcomes in adults receiving red cell transfusions: a systematic review Treatment and prevention of gastrointestinal bleeding in patients receiving antiplatelet therapy Association between red cell transfusions and necrotizing enterocolitis Red cell physiology and signaling relevant to the critical care setting Research opportunities in optimizing storage of red blood cell products Packed red blood cells are an abundant and proximate potential source of nitric oxide synthase inhibition Parallel assay of oxygen equilibria of hemoglobin.Among very-low-birth-weight neonates is red blood cell transfusion an independent risk factor for subsequently developing a severe intraventricular hemorrhage?Feeding practices and other risk factors for developing transfusion-associated necrotizing enterocolitis Anemia in critical illness: insights into etiology, consequences, and management Microcirculatory effects of the transfusion of leukodepleted or non-leukodepleted red blood cells in patients with sepsis: a pilot study.Red blood cell storage: the story so far.An association between vasomotion and oxygen extraction Association between length of storage of red blood cell units and outcome of critically ill children: a prospective observational study.Inclusion of a nitric oxide congener in the insufflation gas repletes S-nitrosohemoglobin and stabilizes physiologic status during prolonged carbon dioxide pneumoperitoneum.Haemoglobin-based oxygen carriers: research and reality towards an alternative to blood transfusions.Evolution of adverse changes in stored RBCs Vascular effects of the red blood cell storage lesion NO Metabolites Levels in Human Red Blood Cells are Affected by Palytoxin, an Inhibitor of Na(+)/K(+)-ATPase Pump.Transfusion related morbidity in premature babies: Possible mechanisms and implications for practice.Anaerobic storage of red blood cells.Randomized study of washing 40- to 42-day-stored red blood cells.Storage of Blood Components Does Not Decrease Haemostatic Potential: In vitro Assessment of Fresh versus Stored Blood Components Using Thromboelastography Non-leukodepleted red blood cell transfusion in sepsis patients: beyond oxygenation, is there a risk of inflammation?Restoration of intracellular ATP production in banked red blood cells improves inducible ATP export and suppresses RBC-endothelial adhesion.Red blood cell age and potentiation of transfusion-related pathology in trauma patients.Protein S-nitrosylation in health and disease: a current perspective.Impaired adenosine-5'-triphosphate release from red blood cells promotes their adhesion to endothelial cells: a mechanism of hypoxemia after transfusion.Packed red cell transfusions alter mesenteric arterial reactivity and nitric oxide pathway in preterm lambs Plasma free hemoglobin and microcirculatory response to fresh or old blood transfusions in sepsis.Effects of storage-aged red blood cell transfusions on endothelial function in hospitalized patients Transfusion of human volunteers with older, stored red blood cells produces extravascular hemolysis and circulating non-transferrin-bound iron.Hemoglobin βCys93 is essential for cardiovascular function and integrated response to hypoxia.Pulmonary hypertension in lambs transfused with stored blood is prevented by breathing nitric oxide Red blood cell washing, nitrite therapy, and antiheme therapies prevent stored red blood cell toxicity after trauma-hemorrhage.Fresh red blood cell transfusion and short-term pulmonary, immunologic, and coagulation status: a randomized clinical trial.Enzymatic mechanisms regulating protein S-nitrosylation: implications in health and disease.

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P2860

S-nitrosohemoglobin deficiency: a mechanism for loss of physiological activity in banked blood.

http://www.wikidata.org/entity/Q34007606

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2007 nî lūn-bûn

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2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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S-nitrosohemoglobin deficiency ...... ical activity in banked blood.

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S-nitrosohemoglobin deficiency ...... ical activity in banked blood.

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S-nitrosohemoglobin deficiency ...... ical activity in banked blood.

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S-nitrosohemoglobin deficiency ...... ical activity in banked blood.

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S-nitrosohemoglobin deficiency ...... ical activity in banked blood.

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S-nitrosohemoglobin deficiency ...... ical activity in banked blood.

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Proceedings of the National Academy of Sciences of the United States of America

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S-nitrosohemoglobin deficiency ...... ical activity in banked blood.

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Fred Cobb

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Gregory S Ahearn

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James D Reynolds

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Jian Zhang

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Jonathan S Stamler

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Michael Angelo

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P2860

Red blood cells express a functional endothelial nitric oxide synthase

Essential roles of S-nitrosothiols in vascular homeostasis and endotoxic shock

Exercising skeletal muscle blood flow in humans responds to reduction in arterial oxyhaemoglobin, but not to altered free oxygen

Hemoglobin conformation couples erythrocyte S-nitrosothiol content to O2 gradients.

Extrapulmonary effects of inhaled nitric oxide: role of reversible S-nitrosylation of erythrocytic hemoglobin.

Export by red blood cells of nitric oxide bioactivity.

Oxygen regulation of tumor perfusion by S-nitrosohemoglobin reveals a pressor activity of nitric oxide.

Blood bank conditions and RBCs: the progressive loss of metabolic modulation.

Impaired vasodilation by red blood cells in sickle cell disease.

Evolution of adverse changes in stored RBCs

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104

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