Transgenic mouse model of ventricular preexcitation and atrioventricular reentrant tachycardia induced by an AMP-activated protein kinase loss-of-function mutation responsible for Wolff-Parkinson-White syndrome.
about
AMP-activated protein kinase, stress responses and cardiovascular diseasesMechanisms of sudden cardiac death: oxidants and metabolismActivation of AMPK alpha- and gamma-isoform complexes in the intact ischemic rat heartNotch signaling regulates murine atrioventricular conduction and the formation of accessory pathways.Aberrant activation of AMP-activated protein kinase remodels metabolic network in favor of cardiac glycogen storage.Defective Tbx2-dependent patterning of the atrioventricular canal myocardium causes accessory pathway formation in miceCharacterization of the role of gamma2 R531G mutation in AMP-activated protein kinase in cardiac hypertrophy and Wolff-Parkinson-White syndrome.A novel PRKAG2 mutation in a Chinese family with cardiac hypertrophy and ventricular pre-excitationSGLT1, a novel cardiac glucose transporter, mediates increased glucose uptake in PRKAG2 cardiomyopathyRegulation of the muscle-specific AMP-activated protein kinase alpha2beta2gamma3 complexes by AMP and implications of the mutations in the gamma3-subunit for the AMP dependence of the enzyme.Regulation of AMPK by the ubiquitin proteasome systemIdentification of a novel de novo mutation associated with PRKAG2 cardiac syndrome and early onset of heart failureAMPK in cardiovascular health and disease.Transgenic knockdown of cardiac sodium/glucose cotransporter 1 (SGLT1) attenuates PRKAG2 cardiomyopathy, whereas transgenic overexpression of cardiac SGLT1 causes pathologic hypertrophy and dysfunction in mice.A pivotal role for endogenous TGF-beta-activated kinase-1 in the LKB1/AMP-activated protein kinase energy-sensor pathway.5'-AMP Activated Protein Kinase is Involved in the Regulation of Myocardial β-Oxidative Capacity in Mice.Tomoregulin-1 prevents cardiac hypertrophy after pressure overload in mice by inhibiting TAK1-JNK pathways.Regulation of NAD(P)H oxidases by AMPK in cardiovascular systemsElectrocardiographic Characterization of Cardiac Hypertrophy in Mice that Overexpress the ErbB2 Receptor Tyrosine Kinase.Is Wolff-Parkinson-White syndrome a genetic disease?Mechanisms of disease: Genetic mechanisms of atrial fibrillation.Familial Wolff-Parkinson-White Syndrome: a disease of glycogen storage or ion channel dysfunction?AMPK alterations in cardiac physiology and pathology: enemy or ally?Role of AMP-activated protein kinase in healthy and diseased hearts.Arrhythmias in patients receiving enzyme replacement therapy for infantile Pompe disease.Genomics in cardiovascular disease.Alterations in energy metabolism in cardiomyopathies.Chronic AMPK activity dysregulation produces myocardial insulin resistance in the human Arg302Gln-PRKAG2 glycogen storage disease mouse model.AMP activated protein kinase: a next generation target for total metabolic control.AMPK: Lessons from transgenic and knockout animalsRemodeling of energy metabolism and absence of electrophysiological changes in the heart of obese hyperleptinemic mice. New insights into the pleiotropic role of leptin.AMP-activated protein kinase pathway: a potential therapeutic target in cardiometabolic disease.Integrative Analysis of PRKAG2 Cardiomyopathy iPS and Microtissue Models Identifies AMPK as a Regulator of Metabolism, Survival, and Fibrosis.Cardiac metabolism and arrhythmias.Origin and development of the atrioventricular myocardial lineage: insight into the development of accessory pathways.Recent success in the discovery of coronary artery disease genes.AMP-activated protein kinase regulation and biological actions in the heart.AMP-activated protein kinase: a key regulator of energy balance with many roles in human disease.Misinterpretation of the mouse ECG: 'musing the waves of Mus musculus'.Clinical Spectrum of PRKAG2 Syndrome.
P2860
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P2860
Transgenic mouse model of ventricular preexcitation and atrioventricular reentrant tachycardia induced by an AMP-activated protein kinase loss-of-function mutation responsible for Wolff-Parkinson-White syndrome.
description
2004 nî lūn-bûn
@nan
2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@ast
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@en
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@nl
type
label
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@ast
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@en
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@nl
prefLabel
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@ast
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@en
Transgenic mouse model of vent ...... olff-Parkinson-White syndrome.
@nl
P2093
P2860
P50
P1433
P1476
Transgenic mouse model of vent ...... Wolff-Parkinson-White syndrome
@en
P2093
A J Marian
Dirar S Khoury
Donald Weilbacher
George E Taffet
Jasvinder S Sidhu
Joanna K Davies
Robert Roberts
Ruiyong Yuan
Tapan G Rami
Yadavendra S Rajawat
P2860
P356
10.1161/01.CIR.0000151291.32974.D5
P407
P577
2004-12-20T00:00:00Z