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BPD Following Preterm Birth: A Model for Chronic Lung Disease and a Substrate for ARDS in ChildhoodCommon respiratory conditions of the newbornOxygen Supplementation to Stabilize Preterm Infants in the Fetal to Neonatal Transition: No Satisfactory AnswerResolvin D1 and lipoxin A4 improve alveolarization and normalize septal wall thickness in a neonatal murine model of hyperoxia-induced lung injuryEffects of hyperoxia on cytoplasmic thioredoxin system in alveolar type epithelial cells of premature ratsPostnatal inflammation in the pathogenesis of bronchopulmonary dysplasiaSustained hyperoxia-induced NF-κB activation improves survival and preserves lung development in neonatal mice.A potential role of the JNK pathway in hyperoxia-induced cell death, myofibroblast transdifferentiation and TGF-β1-mediated injury in the developing murine lung.Increased susceptibility to hyperoxic lung injury and alveolar simplification in newborn rats by prenatal administration of benzo[a]pyrene.Mice deficient in the gene for cytochrome P450 (CYP)1A1 are more susceptible than wild-type to hyperoxic lung injury: evidence for protective role of CYP1A1 against oxidative stress.Heme oxygenase in neonatal lung injury and repairAnimal models of bronchopulmonary dysplasia. The term mouse modelsVascular mediators in chronic lung disease of infancy: role of endothelial monocyte activating polypeptide II (EMAP II).Conditional overexpression of TGFβ1 promotes pulmonary inflammation, apoptosis and mortality via TGFβR2 in the developing mouse lung.Perinatal oxygen in the developing lung.Targeting inflammation to prevent bronchopulmonary dysplasia: can new insights be translated into therapies?Need for supplemental oxygen at discharge in infants with bronchopulmonary dysplasia is not associated with worse neurodevelopmental outcomes at 3 years corrected age.Prenatal administration of the cytochrome P4501A inducer, Β-naphthoflavone (BNF), attenuates hyperoxic lung injury in newborn mice: implications for bronchopulmonary dysplasia (BPD) in premature infants.Biomarkers in neonatology: the new "omics" of bronchopulmonary dysplasia.Effect of Hyperoxia on Retinoid Metabolism and Retinoid Receptor Expression in the Lungs of Newborn Mice.Vasculoprotective effects of heme oxygenase-1 in a murine model of hyperoxia-induced bronchopulmonary dysplasiaIncreased hyperoxia-induced lung injury in nitric oxide synthase 2 null mice is mediated via angiopoietin 2.Caveolin-1 regulates the expression of tight junction proteins during hyperoxia-induced pulmonary epithelial barrier breakdown.Synergistic protection against hyperoxia-induced lung injury by neutrophils blockade and EC-SOD overexpression.Omeprazole Attenuates Pulmonary Aryl Hydrocarbon Receptor Activation and Potentiates Hyperoxia-Induced Developmental Lung Injury in Newborn MiceFunctional deficiency of aryl hydrocarbon receptor augments oxygen toxicity-induced alveolar simplification in newborn miceSmall molecular modulation of macrophage migration inhibitory factor in the hyperoxia-induced mouse model of bronchopulmonary dysplasia.Mechanisms of bronchopulmonary dysplasia.Quantifying hyperoxia-mediated damage to mammalian respiratory cilia-driven fluid flow using particle tracking velocimetry optical coherence tomographyAbsence of TNF-α enhances inflammatory response in the newborn lung undergoing mechanical ventilation.Hyperoxia and interferon-γ-induced injury in developing lungs occur via cyclooxygenase-2 and the endoplasmic reticulum stress-dependent pathway.Phenotypic assessment of pulmonary hypertension using high-resolution echocardiography is feasible in neonatal mice with experimental bronchopulmonary dysplasia and pulmonary hypertension: a step toward preventing chronic obstructive pulmonary diseaSphingosine kinase 1 deficiency confers protection against hyperoxia-induced bronchopulmonary dysplasia in a murine model: role of S1P signaling and Nox proteinsHyperoxia exacerbates postnatal inflammation-induced lung injury in neonatal BRP-39 null mutant mice promoting the M1 macrophage phenotype.Human metapneumovirus in the preterm neonate: current perspectivesModerate hyperoxia induces extracellular matrix remodeling by human fetal airway smooth muscle cells.Adult Lysophosphatidic Acid Receptor 1-Deficient Rats with Hyperoxia-Induced Neonatal Chronic Lung Disease Are Protected against Lipopolysaccharide-Induced Acute Lung Injury.Hyperoxia in the term newborn: more evidence is still needed for optimal oxygen therapy.Impact of preterm birth and bronchopulmonary dysplasia on the developing lung: long-term consequences for respiratory health.Acute lung injury in preterm fetuses and neonates: mechanisms and molecular pathways.
P2860
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P2860
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Hyperoxia-derived lung damage in preterm infants.
@ast
Hyperoxia-derived lung damage in preterm infants.
@en
Hyperoxia-derived lung damage in preterm infants.
@nl
type
label
Hyperoxia-derived lung damage in preterm infants.
@ast
Hyperoxia-derived lung damage in preterm infants.
@en
Hyperoxia-derived lung damage in preterm infants.
@nl
prefLabel
Hyperoxia-derived lung damage in preterm infants.
@ast
Hyperoxia-derived lung damage in preterm infants.
@en
Hyperoxia-derived lung damage in preterm infants.
@nl
P2860
P1476
Hyperoxia-derived lung damage in preterm infants.
@en
P2093
Vineet Bhandari
P2860
P304
P356
10.1016/J.SINY.2010.03.009
P577
2010-04-28T00:00:00Z