Suppression of genome instability by redundant S-phase checkpoint pathways in Saccharomyces cerevisiae.
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An overview of Cdk1-controlled targets and processesDap1p, a heme-binding protein that regulates the cytochrome P450 protein Erg11p/Cyp51p in Saccharomyces cerevisiaeThe BRCA2-interacting protein BCCIP functions in RAD51 and BRCA2 focus formation and homologous recombinational repairSpectroscopic and biochemical characterization of heme binding to yeast Dap1p and mouse PGRMC1pEsc4/Rtt107 and the control of recombination during replication.Proteasome nuclear activity affects chromosome stability by controlling the turnover of Mms22, a protein important for DNA repair.An N-terminal acidic region of Sgs1 interacts with Rpa70 and recruits Rad53 kinase to stalled forks.Esc2 and Sgs1 act in functionally distinct branches of the homologous recombination repair pathway in Saccharomyces cerevisiae.Phosphatase complex Pph3/Psy2 is involved in regulation of efficient non-homologous end-joining pathway in the yeast Saccharomyces cerevisiae.RMI1/NCE4, a suppressor of genome instability, encodes a member of the RecQ helicase/Topo III complexMutation in Rpa1 results in defective DNA double-strand break repair, chromosomal instability and cancer in miceReplisome instability, fork collapse, and gross chromosomal rearrangements arise synergistically from Mec1 kinase and RecQ helicase mutations.Increased genome instability and telomere length in the elg1-deficient Saccharomyces cerevisiae mutant are regulated by S-phase checkpoints.PARP3 is a promoter of chromosomal rearrangements and limits G4 DNA.Formation of complex and unstable chromosomal translocations in yeastThe extent of error-prone replication restart by homologous recombination is controlled by Exo1 and checkpoint proteins.Pathways and Mechanisms that Prevent Genome Instability in Saccharomyces cerevisiaeThe CHEK2 1100delC mutation identifies families with a hereditary breast and colorectal cancer phenotype.Saccharomyces cerevisiae as a model system to define the chromosomal instability phenotype.Multiple regulatory mechanisms to inhibit untimely initiation of DNA replication are important for stable genome maintenance.Delineation of WRN helicase function with EXO1 in the replicational stress responseRecQ helicases: at the heart of genetic stability.Multiple genetic pathways involving the Caenorhabditis elegans Bloom's syndrome genes him-6, rad-51, and top-3 are needed to maintain genome stability in the germ line.Regulation of gross chromosomal rearrangements by ubiquitin and SUMO ligases in Saccharomyces cerevisiae.Checkpoint genes and Exo1 regulate nearby inverted repeat fusions that form dicentric chromosomes in Saccharomyces cerevisiaeThe dissolution of double Holliday junctions.Alternate pathways involving Sgs1/Top3, Mus81/ Mms4, and Srs2 prevent formation of toxic recombination intermediates from single-stranded gaps created by DNA replication.Suppression of gross chromosomal rearrangements by yKu70-yKu80 heterodimer through DNA damage checkpoints.The Rad1-Rad10 complex promotes the production of gross chromosomal rearrangements from spontaneous DNA damage in Saccharomyces cerevisiaeRole of the nuclease activity of Saccharomyces cerevisiae Mre11 in repair of DNA double-strand breaks in mitotic cells.Replication fork stalling by bulky DNA damage: localization at active origins and checkpoint modulationMutator genes for suppression of gross chromosomal rearrangements identified by a genome-wide screening in Saccharomyces cerevisiae.The yeast Sgs1 helicase is differentially required for genomic and ribosomal DNA replication.A Rad53 independent function of Rad9 becomes crucial for genome maintenance in the absence of the Recq helicase Sgs1The contribution of the S-phase checkpoint genes MEC1 and SGS1 to genome stability maintenance in Candida albicansSaccharomyces cerevisiae chromatin-assembly factors that act during DNA replication function in the maintenance of genome stability.DNA polymerase stabilization at stalled replication forks requires Mec1 and the RecQ helicase Sgs1.Causes of oncogenic chromosomal translocation.Differential genetic interactions between Sgs1, DNA-damage checkpoint components and DNA repair factors in the maintenance of chromosome stability.Directed Evolution of RecA Variants with Enhanced Capacity for Conjugational Recombination
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P2860
Suppression of genome instability by redundant S-phase checkpoint pathways in Saccharomyces cerevisiae.
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2002 nî lūn-bûn
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2002 թուականի Մարտին հրատարակուած գիտական յօդուած
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2002 թվականի մարտին հրատարակված գիտական հոդված
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2002年の論文
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2002年論文
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2002年論文
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2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
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2002年论文
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name
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@ast
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@en
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@nl
type
label
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@ast
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@en
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@nl
prefLabel
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@ast
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@en
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@nl
P2860
P356
P1476
Suppression of genome instabil ...... s in Saccharomyces cerevisiae.
@en
P2093
Kyungjae Myung
Richard D Kolodner
P2860
P304
P356
10.1073/PNAS.062702199
P407
P577
2002-03-26T00:00:00Z