about
Involvement of BDNF in age-dependent alterations in the hippocampusPIKE-mediated PI3-kinase activity is required for AMPA receptor surface expressionSynaptopodin-deficient mice lack a spine apparatus and show deficits in synaptic plasticitySpecialized pathways from the primate amygdala to posterior orbitofrontal cortex.Stress duration modulates the spatiotemporal patterns of spine formation in the basolateral amygdalaAxonal varicosity density as an index of local neuronal interactionsGeneral and variable features of varicosity spacing along unmyelinated axons in the hippocampus and cerebellum.Antagonism of brain insulin-like growth factor-1 receptors blocks estradiol effects on memory and levels of hippocampal synaptic proteins in ovariectomized ratsEstradiol and cognitive function: past, present and future.Role of perinatal long-chain omega-3 fatty acids in cortical circuit maturation: Mechanisms and implications for psychopathologyCellular consequences of stress and depression.Enhancement of learning and memory after activation of cerebral Rho GTPasesThe evolving role of dendritic spines and memory: Interaction(s) with estradiolNeuritin reverses deficits in murine novel object associative recognition memory caused by exposure to extremely low-frequency (50 Hz) electromagnetic fields.Estrogens facilitate memory processing through membrane mediated mechanisms and alterations in spine density.Estradiol acts via estrogen receptors alpha and beta on pathways important for synaptic plasticity in the mouse hippocampal formationActivity-induced protocadherin arcadlin regulates dendritic spine number by triggering N-cadherin endocytosis via TAO2beta and p38 MAP kinasesThe fragile X mental retardation protein in circadian rhythmicity and memory consolidation.The occurrence of aging-dependent reticulon 3 immunoreactive dystrophic neurites decreases cognitive function.Expression of long-term potentiation in aged rats involves perforated synapses but dendritic spine branching results from high-frequency stimulation alone.Fragile X protein FMRP is required for homeostatic plasticity and regulation of synaptic strength by retinoic acid.Impaired extinction of cued fear memory and abnormal dendritic morphology in the prelimbic and infralimbic cortices in VPAC2 receptor (VIPR2)-deficient mice.
P2860
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P2860
description
2000 nî lūn-bûn
@nan
2000 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Spine changes associated with long-term potentiation.
@ast
Spine changes associated with long-term potentiation.
@en
Spine changes associated with long-term potentiation.
@nl
type
label
Spine changes associated with long-term potentiation.
@ast
Spine changes associated with long-term potentiation.
@en
Spine changes associated with long-term potentiation.
@nl
prefLabel
Spine changes associated with long-term potentiation.
@ast
Spine changes associated with long-term potentiation.
@en
Spine changes associated with long-term potentiation.
@nl
P2093
P1433
P1476
Spine changes associated with long-term potentiation.
@en
P2093
P304
P356
10.1002/1098-1063(2000)10:5<596::AID-HIPO10>3.3.CO;2-P
P577
2000-01-01T00:00:00Z