STAT3 activation of miR-21 and miR-181b-1 via PTEN and CYLD are part of the epigenetic switch linking inflammation to cancer. - sprookjes - yvandenbroek - TriplyDB

STAT3 activation of miR-21 and miR-181b-1 via PTEN and CYLD are part of the epigenetic switch linking inflammation to cancer.

STAT3 activation of miR-21 and miR-181b-1 via PTEN and CYLD are part of the epigenetic switch linking inflammation to cancer.

http://www.wikidata.org/entity/Q34088885

about

Pre-microRNA and mature microRNA in human mitochondria Telomerase reverse transcriptase regulates microRNAs Identification and functional characterization of FMN2, a regulator of the cyclin-dependent kinase inhibitor p21 MicroRNAs in NF-kappaB signaling Interferon-γ links ultraviolet radiation to melanomagenesis in mice Retinoblastoma tumor suppressor functions shared by stem cell and cancer cell strategies Establishment of Chronic Infection: Brucella's Stealth Strategy MicroRNAs in colorectal carcinoma--from pathogenesis to therapy Non-Coding RNAs in Castration-Resistant Prostate Cancer: Regulation of Androgen Receptor Signaling and Cancer Metabolism Association of Fusobacterium nucleatum with immunity and molecular alterations in colorectal cancer MicroRNAs as growth regulators, their function and biomarker status in colorectal cancer STATs: An Old Story, Yet Mesmerizing Focus on PTEN Regulation MicroRNAs and the resolution phase of inflammation in macrophages Targeting the tumor microenvironment: JAK-STAT3 signaling NF-κB and STAT3 in glioblastoma: therapeutic targets coming of age MicroRNA regulation in cancer-associated fibroblasts Circulating microRNAs in cancer: origin, function and application MicroRNA dysregulation in the spinal cord following traumatic injury The tumor microenvironment shapes hallmarks of mature B-cell malignancies.Caught in the cross fire: p53 in inflammation.Enzastaurin has anti-tumour effects in lung cancers with overexpressed JAK pathway molecules.MicroRNAs in fibrosis: opportunities and challenges Involvement of inflammation and its related microRNAs in hepatocellular carcinoma Turning 21: Induction of miR-21 as a Key Switch in the Inflammatory Response The Emerging Role of microRNAs in Schizophrenia and Autism Spectrum Disorders STAT3 upregulates miR-92a to inhibit RECK expression and to promote invasiveness of lung cancer cells MicroRNAs in B-cell lymphomas: how a complex biology gets more complex Functional screen analysis reveals miR-26b and miR-128 as central regulators of pituitary somatomammotrophic tumor growth through activation of the PTEN-AKT pathway The prognostic value of microRNAs varies with patient race/ethnicity and stage of colorectal cancer HCV infection-associated hepatocellular carcinoma in humanized mice JNK-dependent Stat3 phosphorylation contributes to Akt activation in response to arsenic exposure A multi-targeted approach to suppress tumor-promoting inflammation JNK and STAT3 signaling pathways converge on Akt-mediated phosphorylation of EZH2 in bronchial epithelial cells induced by arsenic Oncogenic transformation of human lung bronchial epithelial cells induced by arsenic involves ROS-dependent activation of STAT3-miR-21-PDCD4 mechanism SNAI1-mediated epithelial-mesenchymal transition confers chemoresistance and cellular plasticity by regulating genes involved in cell death and stem cell maintenance Bioinformatics of Cancer ncRNA in High Throughput Sequencing: Present State and Challenges MicroRNA-21 promotes Th17 differentiation and mediates experimental autoimmune encephalomyelitis TGFβ-stimulated microRNA-21 utilizes PTEN to orchestrate AKT/mTORC1 signaling for mesangial cell hypertrophy and matrix expansion A model for the epigenetic switch linking inflammation to cell transformation: deterministic and stochastic approaches

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STAT3 activation of miR-21 and miR-181b-1 via PTEN and CYLD are part of the epigenetic switch linking inflammation to cancer.

http://www.wikidata.org/entity/Q34088885

description

2010 nî lūn-bûn

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2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

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J.MOLCEL.2010.07.023

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STAT3 activation of miR-21 and ...... inking inflammation to cancer.

@en

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Dimitrios Iliopoulos

http://www.w3.org/2001/XMLSchema#string

Heather A Hirsch

http://www.w3.org/2001/XMLSchema#string

Kevin Struhl

http://www.w3.org/2001/XMLSchema#string

Martha L Bulyk

http://www.w3.org/2001/XMLSchema#string

Savina A Jaeger

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P2860

MicroRNAs: target recognition and regulatory functions

Selective blockade of microRNA processing by Lin28

Widespread microRNA repression by Myc contributes to tumorigenesis

An epigenetic switch involving NF-kappaB, Lin28, Let-7 MicroRNA, and IL6 links inflammation to cell transformation

MicroRNA expression profiles associated with prognosis and therapeutic outcome in colon adenocarcinoma

Lin28 recruits the TUTase Zcchc11 to inhibit let-7 maturation in mouse embryonic stem cells

Disrupting the pairing between let-7 and Hmga2 enhances oncogenic transformation

RAS is regulated by the let-7 microRNA family

c-Myc-regulated microRNAs modulate E2F1 expression

CYLD is a deubiquitinating enzyme that negatively regulates NF-kappaB activation by TNFR family members

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493-506

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scholarly article

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10.1016/J.MOLCEL.2010.07.023

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4

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39

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2010-08-01T00:00:00Z

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46008859

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20797623

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2929389

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