Lipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function. - sprookjes - yvandenbroek - TriplyDB

Lipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.

Lipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.

http://www.wikidata.org/entity/Q34142439

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Mapping of phospholipids by MALDI imaging (MALDI-MSI): realities and expectations Cytokine and Growth Factor Activation In Vivo and In Vitro after Spinal Cord Injury Mitochondria as a source and target of lipid peroxidation products in healthy and diseased heart Imaging mass spectrometry reveals loss of polyunsaturated cardiolipins in the cortical contusion, hippocampus, and thalamus after traumatic brain injury Antioxidant therapies in traumatic brain and spinal cord injury Green tea polyphenols protect spinal cord neurons against hydrogen peroxide-induced oxidative stress.Pharmacological inhibition of lipid peroxidation attenuates calpain-mediated cytoskeletal degradation after traumatic brain injury.Antioxidant therapies for acute spinal cord injury.Neuroprotection by acetyl-L-carnitine after traumatic injury to the immature rat brain.Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice.N-acetylcysteine treatment following spinal cord trauma reduces neural tissue damage and improves locomotor function in mice.Lipid peroxidation in brain or spinal cord mitochondria after injury Acetyl-L-carnitine treatment following spinal cord injury improves mitochondrial function correlated with remarkable tissue sparing and functional recovery.Acrolein-mediated injury in nervous system trauma and diseases Cellular and subcellular oxidative stress parameters following severe spinal cord injury.Administration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivo.Interplay between exercise and dietary fat modulates myelinogenesis in the central nervous system.Phenelzine mitochondrial functional preservation and neuroprotection after traumatic brain injury related to scavenging of the lipid peroxidation-derived aldehyde 4-hydroxy-2-nonenal.Unilateral microinjection of acrolein into thoracic spinal cord produces acute and chronic injury and functional deficits.Neuroprotective effect of tea polyphenols on oxyhemoglobin induced subarachnoid hemorrhage in mice.New insights in the pathogenesis of multiple sclerosis--role of acrolein in neuronal and myelin damage.Diet and cognition: interplay between cell metabolism and neuronal plasticity.Oxidative stress and antioxidative parameters in patients with spinal cord injury: implications in the pathogenesis of disease.Effect of chondroitinase ABC on inflammatory and oxidative response following spinal cord injury.Treating SCA1 Mice with Water-Soluble Compounds to Non-Specifically Boost Mitochondrial Function.Alda-1 reduces cerebral ischemia/reperfusion injury in rat through clearance of reactive aldehydes.CONTINUOUS INFUSION OF PHENELZINE, CYCLOSPORINE A OR THE COMBINATION: EVALUATION OF MITOCHONDRIAL BIOENERGETICS, OXIDATIVE DAMAGE AND CYTOSKELETAL DEGRADATION FOLLOWING SEVERE CONTROLLED CORTICAL IMPACT TRAUMATIC BRAIN INJURY IN RATS.Chronic traumatic encephalopathy-integration of canonical traumatic brain injury secondary injury mechanisms with tau pathology.Phenelzine Protects Brain Mitochondrial Function In Vitro and In Vivo following Traumatic Brain Injury by Scavenging the Reactive Carbonyls 4-Hydroxynonenal and Acrolein Leading to Cortical Histological Neuroprotection.The antioxidant effect of astaxanthin is higher in young mice than aged: a region specific study on brain.Synaptic Mitochondria Sustain More Damage than Non-Synaptic Mitochondria after Traumatic Brain Injury and Are Protected by Cyclosporine A.Synergistic Effect of Quercetin and α-Lipoic Acid on Aluminium Chloride Induced Neurotoxicity in Rats.

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P2860

Lipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.

http://www.wikidata.org/entity/Q34142439

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Lipid peroxidation-derived rea ...... brain mitochondrial function.

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Lipid peroxidation-derived rea ...... brain mitochondrial function.

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Lipid peroxidation-derived rea ...... brain mitochondrial function.

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Lipid peroxidation-derived rea ...... brain mitochondrial function.

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Lipid peroxidation-derived rea ...... brain mitochondrial function.

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Journal of Neurotrauma

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Lipid peroxidation-derived rea ...... brain mitochondrial function.

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Darren M Miller

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Edward D Hall

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Indrapal N Singh

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Radhika A Vaishnav

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P2860

The reaction of no with superoxide

Peroxynitrite: biochemistry, pathophysiology and development of therapeutics

The mitochondrial generation of hydrogen peroxide. General properties and effect of hyperbaric oxygen

Acrolein is increased in Alzheimer's disease brain and is toxic to primary hippocampal cultures.

Antioxidant therapies for traumatic brain injury.

Temporal and spatial dynamics of peroxynitrite-induced oxidative damage after spinal cord contusion injury.

Brain protein oxidation in age-related neurodegenerative disorders that are associated with aggregated proteins.

Pharmacological treatment of acute spinal cord injury: how do we build on past success?

Role of free radicals in the neurodegenerative diseases: therapeutic implications for antioxidant treatment.

Aldehyde-sequestering drugs: tools for studying protein damage by lipid peroxidation products.

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1311-1320

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10.1089/NEU.2009.1172

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