Wide genetic variation in the native pial collateral circulation is a major determinant of variation in severity of stroke.
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A brief etymology of the collateral circulationGenetic dissection of the Canq1 locus governing variation in extent of the collateral circulationNatural genetic variation of integrin alpha L (Itgal) modulates ischemic brain injury in strokeOptical measurement of mouse strain differences in cerebral blood flow using indocyanine green.A BAG3 Coding Variant in Mice Determines Susceptibility to Ischemic Limb Muscle Myopathy by Directing Autophagy.Laser speckle flowmetry method for measuring spatial and temporal hemodynamic alterations throughout large microvascular networks.Skeletal muscle-specific genetic determinants contribute to the differential strain-dependent effects of hindlimb ischemia in mice.Spatial and temporal coordination of bone marrow-derived cell activity during arteriogenesis: regulation of the endogenous response and therapeutic implications.In vivo evidence for long-term vascular remodeling resulting from chronic cerebral hypoperfusion in mice.Strain-dependent variation in collateral circulatory function in mouse hindlimb.Dll4-Notch signaling determines the formation of native arterial collateral networks and arterial function in mouse ischemia models.Formation and maturation of the native cerebral collateral circulation.Endoglin deficiency impairs stroke recoveryTopological basis for the robust distribution of blood to rodent neocortex.Recanalization and reperfusion in acute ischemic stroke.Roles of individual prolyl-4-hydroxylase isoforms in the first 24 hours following transient focal cerebral ischaemia: insights from genetically modified mice.Albumin therapy enhances collateral perfusion after laser-induced middle cerebral artery branch occlusion: a laser speckle contrast flow studySurgery-related thrombosis critically affects the brain infarct volume in mice following transient middle cerebral artery occlusion.Neuroglobin over expressing mice: expression pattern and effect on brain ischemic infarct sizeAging causes collateral rarefaction and increased severity of ischemic injury in multiple tissues.TLR4 accessory molecule RP105 (CD180) regulates monocyte-driven arteriogenesis in a murine hind limb ischemia model.Acute ischemic stroke treatment, part 1: patient selection "the 50% barrier and the capillary index score".Genetic variation in retinal vascular patterning predicts variation in pial collateral extent and stroke severity.Gender differences affect blood flow recovery in a mouse model of hindlimb ischemia.Distal hypoxic stroke: a new mouse model of stroke with high throughput, low variability and a quantifiable functional deficitCollaterals: Implications in cerebral ischemic diseases and therapeutic interventionsMulti-site laser Doppler flowmetry for assessing collateral flow in experimental ischemic stroke: Validation of outcome prediction with acute MRI.Endothelial-Specific EphA4 Negatively Regulates Native Pial Collateral Formation and Re-Perfusion following Hindlimb Ischemia.Adaptation of the cerebrocortical circulation to carotid artery occlusion involves blood flow redistribution between cortical regions and is independent of eNOS.De-novo collateral formation following acute myocardial infarction: Dependence on CCR2⁺ bone marrow cells.Chloride intracellular channel 4 is required for maturation of the cerebral collateral circulation.Pharmacologically increasing collateral perfusion during acute stroke using a carboxyhemoglobin gas transfer agent (Sanguinate™) in spontaneously hypertensive rats.Transient Aortic Occlusion Augments Collateral Blood Flow and Reduces Mortality During Severe Ischemia due to Proximal Middle Cerebral Artery OcclusionAssessment of leptomeningeal collaterals using dynamic CT angiography in patients with acute ischemic stroke.Pial Collateral Reactivity During Hypertension and Aging: Understanding the Function of Collaterals for Stroke TherapyHigh-dose estrogen treatment at reperfusion reduces lesion volume and accelerates recovery of sensorimotor function after experimental ischemic stroke.Deletion or Inhibition of the Oxygen Sensor PHD1 Protects against Ischemic Stroke via Reprogramming of Neuronal Metabolism.Natural allelic variation of the IL-21 receptor modulates ischemic stroke infarct volume.A novel genetic locus modulates infarct volume independently of the extent of collateral circulation.Leptomeningeal collaterals are associated with modifiable metabolic risk factors.
P2860
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P2860
Wide genetic variation in the native pial collateral circulation is a major determinant of variation in severity of stroke.
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Wide genetic variation in the ...... riation in severity of stroke.
@ast
Wide genetic variation in the ...... riation in severity of stroke.
@en
Wide genetic variation in the ...... riation in severity of stroke.
@nl
type
label
Wide genetic variation in the ...... riation in severity of stroke.
@ast
Wide genetic variation in the ...... riation in severity of stroke.
@en
Wide genetic variation in the ...... riation in severity of stroke.
@nl
prefLabel
Wide genetic variation in the ...... riation in severity of stroke.
@ast
Wide genetic variation in the ...... riation in severity of stroke.
@en
Wide genetic variation in the ...... riation in severity of stroke.
@nl
P2093
P2860
P356
P1476
Wide genetic variation in the ...... riation in severity of stroke.
@en
P2093
James E Faber
Pranay Prabhakar
Robert Sealock
P2860
P304
P356
10.1038/JCBFM.2010.10
P577
2010-02-03T00:00:00Z