Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion.
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Expression of complement components differs between kidney allografts from living and deceased donorsAlternative complement pathway deficiency ameliorates chronic smoke-induced functional and morphological ocular injuryPericytes and immune cells contribute to complement activation in tubulointerstitial fibrosis.Natural IgM anti-leukocyte autoantibodies attenuate excess inflammation mediated by innate and adaptive immune mechanisms involving Th-17Pathogenesis of acute kidney injury: foundation for clinical practice.Immune mechanisms and novel pharmacological therapies of acute kidney injury.Soluble thrombomodulin protects ischemic kidneysComplement regulation in renal disease modelsReview: Complement and its regulatory proteins in kidney diseases.Smoke exposure causes endoplasmic reticulum stress and lipid accumulation in retinal pigment epithelium through oxidative stress and complement activation.Activation of complement factor B contributes to murine and human myocardial ischemia/reperfusion injury.Amelioration of acute kidney injury in lipopolysaccharide-induced systemic inflammatory response syndrome by an aldose reductase inhibitor, fidarestat.Tissue-specific deletion of Crry from mouse proximal tubular epithelial cells increases susceptibility to renal ischemia-reperfusion injury.A comparative transcriptome analysis identifying FGF23 regulated genes in the kidney of a mouse CKD model.Defective complement inhibitory function predisposes to renal disease.Both IL-1β and TNF-α regulate NGAL expression in polymorphonuclear granulocytes of chronic hemodialysis patients.An allelic variant of Crry in the murine Sle1c lupus susceptibility interval is not impaired in its ability to regulate complement activation.B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion.Binding of factor H to tubular epithelial cells limits interstitial complement activation in ischemic injuryThe complement inhibitors Crry and factor H are critical for preventing autologous complement activation on renal tubular epithelial cells.Loss of CD11b exacerbates murine complement-mediated tubulointerstitial nephritis.Cellular pathophysiology of ischemic acute kidney injuryComplement alternative pathway activation in the autologous phase of nephrotoxic serum nephritis.Complement activation and toll-like receptor-2 signaling contribute to cytokine production after renal ischemia/reperfusion.Mammalian DNA is an endogenous danger signal that stimulates local synthesis and release of complement factor B.C3a and C5a promote renal ischemia-reperfusion injury.New concepts of complement in allorecognition and graft rejectionNew Insights on Complement Inhibitor CD59 in Mouse Laser-Induced Choroidal Neovascularization: Mislocalization After Injury and Targeted Delivery for Protein Replacement.Annexin A2 Enhances Complement Activation by Inhibiting Factor HTriggers of inflammation after renal ischemia/reperfusion.The immunomodulatory role of carbon monoxide during transplantation.Blocking properdin, the alternative pathway, and anaphylatoxin receptors ameliorates renal ischemia-reperfusion injury in decay-accelerating factor and CD59 double-knockout miceAloperine protects mice against ischemia reperfusion (IR)-induced renal injury by regulating PI3K/AKT/mTOR signaling and AP-1 activity.The role of complement and Toll-like receptors in organ transplantation.Collectin-11 detects stress-induced L-fucose pattern to trigger renal epithelial injuryMembrane protein Crry maintains homeostasis of the complement system.Distinct roles for the complement regulators factor H and Crry in protection of the kidney from injury.The innate immune response in ischemic acute kidney injury.The alternative complement pathway revisited.Discrimination between host and pathogens by the complement system.
P2860
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P2860
Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@ast
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@en
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@nl
type
label
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@ast
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@en
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@nl
prefLabel
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@ast
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@en
Altered renal tubular expressi ...... on after ischemia/reperfusion.
@nl
P2093
P2860
P356
P1476
Altered renal tubular expressi ...... ion after ischemia/reperfusion
@en
P2093
Damian M Kraus
Danica Ljubanović
Gregory Proctor
Hector Molina
Joshua M Thurman
Nicholas P Barry
V Michael Holers
P2860
P304
P356
10.1172/JCI24521
P407
P577
2006-01-26T00:00:00Z