Nicotinamide phosphoribosyl transferase (Nampt) is required for de novo lipogenesis in tumor cells.
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New strategies to maximize therapeutic opportunities for NAMPT inhibitors in oncologyRedox control of glutamine utilization in cancerMetabolomics analysis of metabolic effects of nicotinamide phosphoribosyltransferase (NAMPT) inhibition on human cancer cellsMetabolic Effects of Known and Novel HDAC and SIRT Inhibitors in Glioblastomas Independently or Combined with TemozolomideEIF2A-dependent translational arrest protects leukemia cells from the energetic stress induced by NAMPT inhibitionDepletion of the central metabolite NAD leads to oncosis-mediated cell death.Mice deficient in Sfrp1 exhibit increased adiposity, dysregulated glucose metabolism, and enhanced macrophage infiltrationNicotinamide phosphorybosiltransferase overexpression in thyroid malignancies and its correlation with tumor stage and with survivin/survivin DEx3 expressionSirtuins Function as the Modulators in Aging-related Diseases in Common or RespectivelyExtensive regulation of nicotinate phosphoribosyltransferase (NAPRT) expression in human tissues and tumors.Dependence of tumor cell lines and patient-derived tumors on the NAD salvage pathway renders them sensitive to NAMPT inhibition with GNE-618Targeting of NAD metabolism in pancreatic cancer cells: potential novel therapy for pancreatic tumors.Inhibition of Nicotinamide Phosphoribosyltransferase Induces Apoptosis in Estrogen Receptor-Positive MCF-7 Breast Cancer Cells.Pregnane xenobiotic receptor in cancer pathogenesis and therapeutic response.Physiological and pathophysiological roles of NAMPT and NAD metabolism.Targeting Nicotinamide Phosphoribosyltransferase as a Potential Therapeutic Strategy to Restore Adult Neurogenesis.Hepatic NAD(+) deficiency as a therapeutic target for non-alcoholic fatty liver disease in ageing.Oleate ameliorates palmitate-induced reduction of NAMPT activity and NAD levels in primary human hepatocytes and hepatocarcinoma cells.Association between Nicotinamide Phosphoribosyltransferase and de novo Lipogenesis in Nonalcoholic Fatty Liver Disease.Intracellular NAD(H) levels control motility and invasion of glioma cells.Endostatin inhibits androgen-independent prostate cancer growth by suppressing nuclear receptor-mediated oxidative stress.CD38 Inhibits Prostate Cancer Metabolism and Proliferation by Reducing Cellular NAD Pools
P2860
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P2860
Nicotinamide phosphoribosyl transferase (Nampt) is required for de novo lipogenesis in tumor cells.
description
2012 nî lūn-bûn
@nan
2012 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Nicotinamide phosphoribosyl tr ...... vo lipogenesis in tumor cells.
@ast
Nicotinamide phosphoribosyl tr ...... vo lipogenesis in tumor cells.
@en
Nicotinamide phosphoribosyl transferase
@nl
type
label
Nicotinamide phosphoribosyl tr ...... vo lipogenesis in tumor cells.
@ast
Nicotinamide phosphoribosyl tr ...... vo lipogenesis in tumor cells.
@en
Nicotinamide phosphoribosyl transferase
@nl
prefLabel
Nicotinamide phosphoribosyl tr ...... vo lipogenesis in tumor cells.
@ast
Nicotinamide phosphoribosyl tr ...... vo lipogenesis in tumor cells.
@en
Nicotinamide phosphoribosyl transferase
@nl
P2093
P2860
P1433
P1476
Nicotinamide phosphoribosyl tr ...... vo lipogenesis in tumor cells.
@en
P2093
Michael J Thomas
Ralph B D'Agostino
Sarah C Bowlby
Steven J Kridel
P2860
P304
P356
10.1371/JOURNAL.PONE.0040195
P407
P577
2012-06-29T00:00:00Z