An activated c-Ha-ras allele blocks the induction of muscle-specific genes whose expression is contingent on mitogen withdrawal.
about
Human carcinoembryonic antigen, an intercellular adhesion molecule, blocks fusion and differentiation of rat myoblastsHistone deacetylase 4 associates with extracellular signal-regulated kinases 1 and 2, and its cellular localization is regulated by oncogenic RasPositive regulation of skeletal myogenesis by R-RasRetinoic acid induces myogenin synthesis and myogenic differentiation in the rat rhabdomyosarcoma cell line BA-Han-1CDifferential regulation of skeletal alpha-actin transcription in cardiac muscle by two fibroblast growth factors.Peptide growth factors can provoke "fetal" contractile protein gene expression in rat cardiac myocytes.Dihydropyridine receptor gene expression is regulated by inhibitors of myogenesis and is relatively insensitive to denervation.Dexamethasone-dependent inhibition of differentiation of C2 myoblasts bearing steroid-inducible N-ras oncogenes.Differentiation of cardiac myocytes after mitogen withdrawal exhibits three sequential states of the ventricular growth responseGrowth factors, signaling pathways, and the regulation of proliferation and differentiation in BC3H1 muscle cells. II. Two signaling pathways distinguished by pertussis toxin and a potential role for the ras oncogene.Growth factors, signaling pathways, and the regulation of proliferation and differentiation in BC3H1 muscle cells. I. A pertussis toxin-sensitive pathway is involved.Aberrant regulation of MyoD1 contributes to the partially defective myogenic phenotype of BC3H1 cells.Inhibition of myogenesis by the H-ras oncogene: implication of a role for protein kinase C.c-myc inhibition of MyoD and myogenin-initiated myogenic differentiation.c-Ha-ras down regulates the alpha-fetoprotein gene but not the albumin gene in human hepatoma cells.Fibroblast growth factor and transforming growth factor beta repress transcription of the myogenic regulatory gene MyoD1A ras-dependent pathway abolishes activity of a muscle-specific enhancer upstream from the muscle creatine kinase gene.Effect of cellular determination on oncogenic transformation by chemicals and oncogenes.Expression of the Gs protein alpha-subunit disrupts the normal program of differentiation in cultured murine myogenic cells.Amplification of MDM2 inhibits MyoD-mediated myogenesis.Transcription of muscle-specific genes is repressed by reactivation of pp60v-src in postmitotic quail myotubesExpression of two new protein isoforms of the neurofibromatosis type 1 gene product, neurofibromin, in muscle tissues.
P2860
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P2860
An activated c-Ha-ras allele blocks the induction of muscle-specific genes whose expression is contingent on mitogen withdrawal.
description
1987 nî lūn-bûn
@nan
1987 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1987 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
name
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@ast
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@en
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@nl
type
label
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@ast
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@en
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@nl
prefLabel
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@ast
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@en
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@nl
P2093
P2860
P356
P1476
An activated c-Ha-ras allele b ...... tingent on mitogen withdrawal.
@en
P2093
P2860
P304
P356
10.1073/PNAS.84.24.8956
P407
P577
1987-12-01T00:00:00Z