Suppression of cancer progression by MGAT1 shRNA knockdown. - sprookjes - yvandenbroek - TriplyDB

Suppression of cancer progression by MGAT1 shRNA knockdown.

Suppression of cancer progression by MGAT1 shRNA knockdown.

http://www.wikidata.org/entity/Q34407965

about

Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis.Golgi N-glycan branching N-acetylglucosaminyltransferases I, V and VI promote nutrient uptake and metabolism.Glycan structures contain information for the spatial arrangement of glycoproteins in the plasma membrane.Galectins as new prognostic markers and potential therapeutic targets for advanced prostate cancers.Biosynthetic Machinery Involved in Aberrant Glycosylation: Promising Targets for Developing of Drugs Against Cancer What Have We Learned from Glycosyltransferase Knockouts in Mice?Serum tri- and tetra-antennary N-glycan is a potential predictive biomarker for castration-resistant prostate cancer.The directed migration of gonadal distal tip cells in Caenorhabditis elegans requires NGAT-1, a ß1,4-N-acetylgalactosaminyltransferase enzyme."Stealth dissemination" of macrophage-tumor cell fusions cultured from blood of patients with pancreatic ductal adenocarcinoma.A mechanism for bistability in glycosylation

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Suppression of cancer progression by MGAT1 shRNA knockdown.

http://www.wikidata.org/entity/Q34407965

description

2012 nî lūn-bûn

@nan

2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

@wuu

name

Suppression of cancer progression by MGAT1 shRNA knockdown.

@ast

Suppression of cancer progression by MGAT1 shRNA knockdown.

@en

Suppression of cancer progression by MGAT1 shRNA knockdown.

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type

label

Suppression of cancer progression by MGAT1 shRNA knockdown.

@ast

Suppression of cancer progression by MGAT1 shRNA knockdown.

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Suppression of cancer progression by MGAT1 shRNA knockdown.

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prefLabel

Suppression of cancer progression by MGAT1 shRNA knockdown.

@ast

Suppression of cancer progression by MGAT1 shRNA knockdown.

@en

Suppression of cancer progression by MGAT1 shRNA knockdown.

@nl

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Suppression of cancer progression by MGAT1 shRNA knockdown

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Carol J Swallow

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Craig D Simpson

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David R Rose

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Francis Zih

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Harry Schachter

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James W Dennis

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Mahadeo A Sukhai

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Marcela Gronda

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Neil Maclean

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Reza Beheshti Zavareh

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P2860

Metabolomic profiles delineate potential role for sarcosine in prostate cancer progression

A high-content chemical screen identifies ellipticine as a modulator of p53 nuclear localization

Physiological and glycomic characterization of N-acetylglucosaminyltransferase-IVa and -IVb double deficient mice

Mice lacking N-acetylglucosaminyltransferase I activity die at mid-gestation, revealing an essential role for complex or hybrid N-linked carbohydrates

A lentiviral RNAi library for human and mouse genes applied to an arrayed viral high-content screen

Beyond aerobic glycolysis: transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis

Integrin-ligand binding properties govern cell migration speed through cell-substratum adhesiveness

Plasma membrane domain organization regulates EGFR signaling in tumor cells

Inhibition of the sodium/potassium ATPase impairs N-glycan expression and function.

Suppression of tumor growth and metastasis in Mgat5-deficient mice.

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e43721

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