Unbiased analysis of potential targets of breast cancer susceptibility loci by Capture Hi-C.
about
4D nucleomes in single cells: what can computational modeling reveal about spatial chromatin conformation?Chromatin organization in pluripotent cells: emerging approaches to study and disrupt functionAn Overview of Genome Organization and How We Got There: from FISH to Hi-CCapture Hi-C identifies a novel causal gene, IL20RA, in the pan-autoimmune genetic susceptibility region 6q23C-ing the Genome: A Compendium of Chromosome Conformation Capture Methods to Study Higher-Order Chromatin OrganizationIDH-mutant glioma specific association of rs55705857 located at 8q24.21 involves MYC deregulationLineage-Specific Genome Architecture Links Enhancers and Non-coding Disease Variants to Target Gene PromotersCHiCAGO: robust detection of DNA looping interactions in Capture Hi-C dataThree new pancreatic cancer susceptibility signals identified on chromosomes 1q32.1, 5p15.33 and 8q24.21Fine mapping of type 1 diabetes susceptibility loci and evidence for colocalization of causal variants with lymphoid gene enhancers.HiCUP: pipeline for mapping and processing Hi-C data.Integrating epigenomic data and 3D genomic structure with a new measure of chromatin assortativityRNA-Seq based genome-wide analysis reveals loss of inter-chromosomal regulation in breast cancer.Single-cell Hi-C for genome-wide detection of chromatin interactions that occur simultaneously in a single cell.Genome-wide mapping and analysis of chromosome architecture.The pluripotent regulatory circuitry connecting promoters to their long-range interacting elements.Comparison of Hi-C results using in-solution versus in-nucleus ligationGenome-wide mapping of promoter-anchored interactions with close to single-enhancer resolution.4C-ker: A Method to Reproducibly Identify Genome-Wide Interactions Captured by 4C-Seq Experiments.An intergenic risk locus containing an enhancer deletion in 2q35 modulates breast cancer risk by deregulating IGFBP5 expressionChromatin interaction analysis reveals changes in small chromosome and telomere clustering between epithelial and breast cancer cellsCapture Hi-C reveals novel candidate genes and complex long-range interactions with related autoimmune risk lociCCSI: a database providing chromatin-chromatin spatial interaction information.Chromatin interactions and candidate genes at ten prostate cancer risk loci.Effects on the transcriptome upon deletion of a distal element cannot be predicted by the size of the H3K27Ac peak in human cellsFrom GWAS to function: lessons from blood cells.A pathway-centric view of spatial proximity in the 3D nucleome across cell lines.A functional variant rs4442975 modulating FOXA1-binding affinity does not influence the risk or progression of breast cancer in Chinese Han population.Strategies for fine-mapping complex traits.Genetics of coronary heart disease: towards causal mechanisms, novel drug targets and more personalized prevention.Chromosome conformation capture technologies and their impact in understanding genome function.Genetic susceptibility to rheumatoid arthritis and its implications for novel drug discovery.Global transcription network incorporating distal regulator binding reveals selective cooperation of cancer drivers and risk genesCapturing genomic relationships that matterChallenges and progress in interpretation of non-coding genetic variants associated with human disease.Activation of the alpha-globin gene expression correlates with dramatic upregulation of nearby non-globin genes and changes in local and large-scale chromatin spatial structure.Active and Inactive Enhancers Cooperate to Exert Localized and Long-Range Control of Gene Regulation.Polycomb repressive complex PRC1 spatially constrains the mouse embryonic stem cell genome.A Compendium of Chromatin Contact Maps Reveals Spatially Active Regions in the Human Genome.Chromosome contacts in activated T cells identify autoimmune disease candidate genes.
P2860
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P2860
Unbiased analysis of potential targets of breast cancer susceptibility loci by Capture Hi-C.
description
2014 nî lūn-bûn
@nan
2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@ast
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@en
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@nl
type
label
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@ast
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@en
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@nl
prefLabel
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@ast
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@en
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@nl
P2093
P2860
P50
P356
P1433
P1476
Unbiased analysis of potential ...... tibility loci by Capture Hi-C.
@en
P2093
Eleni Perrakis
Ioannis Assiotis
Iwanka Kozarewa
James Campbell
Kerry Fenwick
Laura R Broome
Maryou Lambros
Nicola H Dryden
Simon Andrews
Stefan Schoenfelder
P2860
P304
P356
10.1101/GR.175034.114
P50
P577
2014-08-13T00:00:00Z