Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion - sprookjes - yvandenbroek - TriplyDB

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

http://www.wikidata.org/entity/Q34606869

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ROS and Brain Gliomas: An Overview of Potential and Innovative Therapeutic Strategies Neutralizing the EGF receptor in glioblastoma cells stimulates cell migration by activating uPAR-initiated cell signaling.Mechanisms of glioma formation: iterative perivascular glioma growth and invasion leads to tumor progression, VEGF-independent vascularization, and resistance to antiangiogenic therapy.Bevacizumab treatment induces metabolic adaptation toward anaerobic metabolism in glioblastomas Phosphorylated SATB1 is associated with the progression and prognosis of glioma.Phase 2 study of bosutinib, a Src inhibitor, in adults with recurrent glioblastoma.Pro-Tumorigenic Phosphorylation of p120 Catenin in Renal and Breast Cancer.Src family kinases differentially influence glioma growth and motility Phosphorylation and isoform use in p120-catenin during development and tumorigenesis Establishment of human iPSC-based models for the study and targeting of glioma initiating cells The Effect of Chemoradiotherapy with SRC Tyrosine Kinase Inhibitor, PP2 and Temozolomide on Malignant Glioma Cells In Vitro and In Vivo p120catenin alteration in cancer and its role in tumour invasion.SDF-1 Blockade Enhances Anti-VEGF Therapy of Glioblastoma and Can Be Monitored by MRI.Genomic analyses reveal broad impact of miR-137 on genes associated with malignant transformation and neuronal differentiation in glioblastoma cells.VEGF-VEGFR Signals in Health and Disease.Molecular targets in glioblastoma.Src Family Kinases in Brain Edema After Acute Brain Injury.EGFRvIII mutations can emerge as late and heterogenous events in glioblastoma development and promote angiogenesis through Src activation.Reprogramming strategies for the establishment of novel human cancer models.PTPIP51 levels in glioblastoma cells depend on inhibition of the EGF-receptor.EGFR and EGFRvIII Promote Angiogenesis and Cell Invasion in Glioblastoma: Combination Therapies for an Effective Treatment.Orphan Nuclear Receptor TR3/Nur77 is a Specific Therapeutic Target for Hepatic Cancers.

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Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

http://www.wikidata.org/entity/Q34606869

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2013 nî lūn-bûn

@nan

2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2013年の論文

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2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@ast

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@en

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@nl

type

label

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@ast

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@en

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@nl

prefLabel

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@ast

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@en

Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

@nl

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JOURNAL.PONE.0056505

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Targeting Src family kinases inhibits bevacizumab-induced glioma cell invasion

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Brett L Carlson

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Caterina Giannini

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Deborah Huveldt

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Fausto Rodriguez

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Jann N Sarkaria

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Laura J Lewis-Tuffin

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Mark A Schroeder

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Panos Z Anastasiadis

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P2860

Activation of Rac1 by a Crk SH3-binding protein, DOCK180

Yes and PI3K bind CD95 to signal invasion of glioblastoma

VEGF inhibits tumor cell invasion and mesenchymal transition through a MET/VEGFR2 complex

Integrin-regulated FAK-Src signaling in normal and cancer cells

Newest findings on the oldest oncogene; how activated src does it

Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis

SKI-606 (bosutinib), a novel Src kinase inhibitor, suppresses migration and invasion of human breast cancer cells.

Increased fibroblast growth factor-inducible 14 expression levels promote glioma cell invasion via Rac1 and nuclear factor-kappaB and correlate with poor patient outcome

Endosomal NADPH oxidase regulates c-Src activation following hypoxia/reoxygenation injury

Accelerated metastasis after short-term treatment with a potent inhibitor of tumor angiogenesis

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e56505

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scholarly article

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10.1371/JOURNAL.PONE.0056505

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2

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Evanthia Galanis

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2013-02-14T00:00:00Z

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235778505

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