Alpha2A-adrenoceptors strengthen working memory networks by inhibiting cAMP-HCN channel signaling in prefrontal cortex. - sprookjes - yvandenbroek - TriplyDB

Alpha2A-adrenoceptors strengthen working memory networks by inhibiting cAMP-HCN channel signaling in prefrontal cortex.

Alpha2A-adrenoceptors strengthen working memory networks by inhibiting cAMP-HCN channel signaling in prefrontal cortex.

http://www.wikidata.org/entity/Q34621253

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Cognitive enhancers for the treatment of ADHD Psychostimulants as cognitive enhancers: the prefrontal cortex, catecholamines, and attention-deficit/hyperactivity disorder The α2B-adrenergic receptor is mutant in cortical myoclonus and epilepsy Dynamic Network Connectivity: A new form of neuroplasticity The neurobiological basis of ADHD Stress signalling pathways that impair prefrontal cortex structure and function Neuromodulation of thought: flexibilities and vulnerabilities in prefrontal cortical network synapses The effect of moderate acute psychological stress on working memory-related neural activity is modulated by a genetic variation in catecholaminergic function in humans The neuropsychopharmacology of fronto-executive function: monoaminergic modulation The use of α-2A adrenergic agonists for the treatment of attention-deficit/hyperactivity disorder Alpha-2 adrenergic challenge with guanfacine one month after mild traumatic brain injury: altered working memory and BOLD response Psychosocial stress reversibly disrupts prefrontal processing and attentional control HCN1 channel subunits are a molecular substrate for hypnotic actions of ketamine.Mapping the regulator of G protein signaling 4 (RGS4): presynaptic and postsynaptic substrates for neuroregulation in prefrontal cortex Inhibition of protein kinase C signaling protects prefrontal cortex dendritic spines and cognition from the effects of chronic stress.Adrenergic pharmacology and cognition: focus on the prefrontal cortex Catecholamines and cognition after traumatic brain injury Physiological approaches to understanding molecular actions on dorsolateral prefrontal cortical neurons underlying higher cognitive processing Stress weakens prefrontal networks: molecular insults to higher cognition Lactate transport and signaling in the brain: potential therapeutic targets and roles in body-brain interaction Molecular aspects of age-related cognitive decline: the role of GABA signaling Role of signal transduction crosstalk between adenylyl cyclase and MAP kinase in hippocampus-dependent memory Cortical HCN channels: function, trafficking and plasticity Synergy and Antagonism of Active Constituents of ADAPT-232 on Transcriptional Level of Metabolic Regulation of Isolated Neuroglial Cells Profile of guanfacine extended release and its potential in the treatment of attention-deficit hyperactivity disorder Targeting Prefrontal Cortical Systems for Drug Development: Potential Therapies for Cognitive Disorders Waking State: Rapid Variations Modulate Neural and Behavioral Responses Clinical utility of guanfacine extended release in the treatment of ADHD in children and adolescents A computational psychiatry approach identifies how alpha-2A noradrenergic agonist Guanfacine affects feature-based reinforcement learning in the macaque.Enhancement of dorsal hippocampal activity by knockdown of HCN1 channels leads to anxiolytic- and antidepressant-like behaviors.The human frontal lobes and frontal network systems: an evolutionary, clinical, and treatment perspective Differential cognitive actions of norepinephrine a2 and a1 receptor signaling in the prefrontal cortex Noradrenergic Modulation of Cognition in Health and Disease.HCN1 channels in cerebellar Purkinje cells promote late stages of learning and constrain synaptic inhibition Fear signaling in the prelimbic-amygdala circuit: a computational modeling and recording study.Homeostatic regulation of synaptic excitability: tonic GABA(A) receptor currents replace I(h) in cortical pyramidal neurons of HCN1 knock-out mice.Cocaine sensitization increases I h current channel subunit 2 (HCN₂) protein expression in structures of the mesocorticolimbic system.Persistent activity in a cortical-to-subcortical circuit: bridging the temporal gap in trace eyelid conditioning Corticotropin-releasing factor in the norepinephrine nucleus, locus coeruleus, facilitates behavioral flexibility.Loss of dendritic HCN1 subunits enhances cortical excitability and epileptogenesis.

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Alpha2A-adrenoceptors strengthen working memory networks by inhibiting cAMP-HCN channel signaling in prefrontal cortex.

http://www.wikidata.org/entity/Q34621253

description

2007 nî lūn-bûn

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2007 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2007 թվականի ապրիլին հրատարակված գիտական հոդված

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2007年の論文

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Alpha2A-adrenoceptors strength ...... ignaling in prefrontal cortex.

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Alpha2A-adrenoceptors strength ...... ignaling in prefrontal cortex.

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Alpha2A-adrenoceptors strength ...... ignaling in prefrontal cortex.

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Alpha2A-adrenoceptors strength ...... ignaling in prefrontal cortex.

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Alvaro Duque

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