Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
about
Alternating bipolar field stimulation identifies muscle fibers with defective excitability but maintained local Ca(2+) signals and contraction.Incidence and severity of myofiber branching with regeneration and aging.Microarchitecture is severely compromised but motor protein function is preserved in dystrophic mdx skeletal muscle.Incubating isolated mouse EDL muscles with creatine improves force production and twitch kinetics in fatigue due to reduction in ionic strength.A new function for odorant receptors: MOR23 is necessary for normal tissue repair in skeletal muscle.Altered Ca2+ kinetics associated with α-actinin-3 deficiency may explain positive selection for ACTN3 null allele in human evolutionRhoA/ROCK signaling and pleiotropic α1A-adrenergic receptor regulation of cardiac contractility.Structural and functional evaluation of branched myofibers lacking intermediate filamentsContractile properties of skinned muscle fibres from young and adult normal and dystrophic (mdx) mice.Malformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling.Human skeletal muscle plasmalemma alters its structure to change its Ca2+-handling following heavy-load resistance exercise.Disruption of action potential and calcium signaling properties in malformed myofibers from dystrophin-deficient mice.Molecular cogs in machina carnis.Mini-dystrophin restores L-type calcium currents in skeletal muscle of transgenic mdx mice.The action potential-evoked sarcoplasmic reticulum calcium release is impaired in mdx mouse muscle fibres.EDL and soleus muscles of the C57BL6J/dy2j laminin-alpha 2-deficient dystrophic mouse are not vulnerable to eccentric contractions.Properties of extensor digitorum longus muscle and skinned fibers from adult and aged male and female Actn3 knockout mice.Optical prediction of single muscle fiber force production using a combined biomechatronics and second harmonic generation imaging approach
P2860
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P2860
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
description
1990 nî lūn-bûn
@nan
1990 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի մարտին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@ast
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@en
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@nl
type
label
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@ast
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@en
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@nl
prefLabel
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@ast
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@en
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@nl
P2093
P1476
Properties of enzymatically isolated skeletal fibres from mice with muscular dystrophy.
@en
P2093
Stephenson DG
Williams DA
P304
P356
10.1113/JPHYSIOL.1990.SP017988
P407
P577
1990-03-01T00:00:00Z