Green tea epigallocatechin-3-gallate mediates T cellular NF-kappa B inhibition and exerts neuroprotection in autoimmune encephalomyelitis.
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Luteolin as a therapeutic option for multiple sclerosisNeurodegeneration in multiple sclerosis: novel treatment strategiesNatural health products, modulation of immune function and prevention of chronic diseases.Potential neuroprotective properties of epigallocatechin-3-gallate (EGCG)Redox regulation of T-cell function: from molecular mechanisms to significance in human health and diseaseInduction of cellular and molecular immunomodulatory pathways by vitamin A and flavonoidsRole of "Western diet" in inflammatory autoimmune diseasesNew insights into the mechanisms of polyphenols beyond antioxidant properties; lessons from the green tea polyphenol, epigallocatechin 3-gallateMultiple sclerosis: molecular mechanisms and therapeutic opportunitiesTracking CNS and systemic sources of oxidative stress during the course of chronic neuroinflammationNT-020 treatment reduces inflammation and augments Nrf-2 and Wnt signaling in aged ratsAdaptive cellular stress pathways as therapeutic targets of dietary phytochemicals: focus on the nervous systemMechanisms of oxidative damage in multiple sclerosis and neurodegenerative diseases: therapeutic modulation via fumaric acid estersA multi-targeted approach to suppress tumor-promoting inflammationComplementary and alternative medicine for the treatment of multiple sclerosisPolyphenon E, non-futile at neuroprotection in multiple sclerosis but unpredictably hepatotoxic: Phase I single group and phase II randomized placebo-controlled studies.Iron in chronic brain disorders: imaging and neurotherapeutic implications.Epigallocatechin gallate inhibits endothelial exocytosisCognitive deficit associated with cholinergic and nerve growth factor down-regulation in experimental allergic encephalomyelitis in ratsNeuroprotective effect of combination therapy of glatiramer acetate and epigallocatechin-3-gallate in neuroinflammation.The therapeutic effects of MSc1 nanocomplex, synthesized by nanochelating technology, on experimental autoimmune encephalomyelitic C57/BL6 miceAnalysis of the effect of the active compound of green tea (EGCG) on the proliferation of peripheral blood mononuclear cells.MS14, a Marine Herbal Medicine, an Immunosuppressive Drug in Experimental Autoimmune Encephalomyelitis.EGCG functions through estrogen receptor-mediated activation of ADAM10 in the promotion of non-amyloidogenic processing of APPStorage and allogeneic transplantation of peripheral nerve using a green tea polyphenol solution in a canine modelMS14 down-regulates lipocalin2 expression in spinal cord tissue in an animal model of multiple sclerosis in female C57BL/6.Fumaric acid esters exert neuroprotective effects in neuroinflammation via activation of the Nrf2 antioxidant pathway.The influence of nutritional factors on the prognosis of multiple sclerosis.K(ATP) channel opener diazoxide prevents neurodegeneration: a new mechanism of action via antioxidative pathway activationEGCG attenuates autoimmune arthritis by inhibition of STAT3 and HIF-1α with Th17/Treg control.Epigallocatechin gallate delays the onset of type 1 diabetes in spontaneous non-obese diabetic mice.Green tea polyphenol epigallocatechin-3-gallate: inflammation and arthritis. [corrected].Rationale for the use of multifunctional drugs as neuroprotective agents for glaucoma.Impact of tea drinking upon tuberculosis: a neglected issue.Treatment of Chronic Experimental Autoimmune Encephalomyelitis with Epigallocatechin-3-Gallate and Glatiramer Acetate Alters Expression of Heme-Oxygenase-1.EGCG-mediated autophagy flux has a neuroprotection effect via a class III histone deacetylase in primary neuron cells.Health-promoting effects of green teaDietary polyphenols as modulators of brain functions: biological actions and molecular mechanisms underpinning their beneficial effectsTherapeutic Advances and Future Prospects in Progressive Forms of Multiple SclerosisAntioxidants in multiple sclerosis: do they have a role in therapy?
P2860
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P2860
Green tea epigallocatechin-3-gallate mediates T cellular NF-kappa B inhibition and exerts neuroprotection in autoimmune encephalomyelitis.
description
2004 nî lūn-bûn
@nan
2004 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@ast
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@en
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@nl
type
label
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@ast
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@en
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@nl
prefLabel
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@ast
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@en
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@nl
P2093
P50
P1476
Green tea epigallocatechin-3-g ...... autoimmune encephalomyelitis.
@en
P2093
Alina Smorodchenko
Nicolai E Savaskan
Orhan Aktas
Roland Lauster
Stefan Brocke
Timour Prozorovski
P304
P356
10.4049/JIMMUNOL.173.9.5794
P407
P577
2004-11-01T00:00:00Z