Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
about
Cancer of the Pancreas: Molecular Pathways and Current Advancement in TreatmentPeroxisome proliferator activated receptors at the crossroad of obesity, diabetes, and pancreatic cancerManagement of Incidental Pancreatic Cystic LesionsActin cytoskeletal control during epithelial to mesenchymal transition: focus on the pancreas and intestinal tractA rapid in vivo screen for pancreatic ductal adenocarcinoma therapeutics.Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward.MicroRNAs of the mir-17~92 cluster regulate multiple aspects of pancreatic tumor development and progression.CD90 and CD24 Co-Expression Is Associated with Pancreatic Intraepithelial NeoplasiasExploring the Functional Disorder and Corresponding Key Transcription Factors in Intraductal Papillary Mucinous Neoplasms Progression.Neutrophils: Critical components in experimental animal models of cancer.Endoscopic ultrasound-guided fine-needle aspiration plus KRAS and GNAS mutation in malignant intraductal papillary mucinous neoplasm of the pancreas.Contribution of microRNA analysis to characterisation of pancreatic lesions: a review.Familial Pancreatic Cancer and the Future of Directed Screening.Targeting reactive oxygen species in development and progression of pancreatic cancer.Natural History of Pancreatic Cysts.Exclusive and Combined Use of Statins and Aspirin and the Risk of Pancreatic Cancer: a Case-Control StudyDuct- and Acinar-Derived Pancreatic Ductal Adenocarcinomas Show Distinct Tumor Progression and Marker Expression.Identification of a pyruvate-to-lactate signature in pancreatic intraductal papillary mucinous neoplasms.KRAS: The Critical Driver and Therapeutic Target for Pancreatic Cancer.Mucinous cystic neoplasms of the pancreas: high-resolution cross-sectional imaging features with clinico-pathologic correlation.Prevalence of incidental pancreatic cyst on upper endoscopic ultrasound.Cellular senescence in gastrointestinal diseases: from pathogenesis to therapeutics.[Early stage pancreatic cancer].Krüppel-like Factor 5, Increased in Pancreatic Ductal Adenocarcinoma, Promotes Proliferation, Acinar to Ductal Metaplasia, Pancreatic Intraepithelial Neoplasia, and Tumor Growth in Mice.Identification and Validation of a Diagnostic and Prognostic Multi-Gene Biomarker Panel for Pancreatic Ductal Adenocarcinoma.Evaluation of SAS1B as a target for antibody-drug conjugate therapy in the treatment of pancreatic cancer.Metabonomic alterations from pancreatic intraepithelial neoplasia to pancreatic ductal adenocarcinoma facilitate the identification of biomarkers in serum for early diagnosis of pancreatic cancer.Light scattering spectroscopy identifies the malignant potential of pancreatic cysts during endoscopy
P2860
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P2860
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
description
2014 nî lūn-bûn
@nan
2014 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մարտին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@ast
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@en
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@nl
type
label
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@ast
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@en
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@nl
prefLabel
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@ast
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@en
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@nl
P2093
P2860
P921
P356
P1476
Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.
@en
P2093
P2860
P304
P356
10.1155/2014/474905
P407
P5008
P577
2014-03-24T00:00:00Z